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- Objective: To explore whether gancyclovir (GCV) can inhibit the proliferation and induce the erythro differentiation of the K562 human myeloid leukemia cell line. 目的：探討更昔洛韋（GCV）對人白血病細胞的抗增殖和誘導分化作用。
- Methods Apoptosis induced by Ara c in human myeloid leukemia cell line HL 60 was investigated by applying light microscopy, electron microscopy combined with DNA electrophoresis and flow cytometry analysis techniques. 方法應用光鏡、電鏡，結合DNA電泳及流式細胞儀分析技術，觀察阿糖胞苷（Ara－c）誘導的急性髓系白血病細胞系HL－60凋亡模式。
- Objective: To explore whether nucleoside analog acycolovir(ACV) and gancyclovir(GCV) can inhibit the proliferation and induce the erythro differentiation of a K562 human myeloid leukemia cell line. 目的 :探討核苷類似物阿昔洛韋 ( ACV)、更昔洛韋 ( GCV)對人白血病細胞的抗增殖和誘導分化作用。
- CIK cells generated from patients could kill K562 cells and lysis myeloid leukemia cells in vitro. 從病人體內分離培養的CIK細胞，在體外有強的殺傷K562細胞和裂解髓系白血病細胞的作用；
- Methods Human myeloid leukemia cells, K562 and NB4, were transduced by MDR retrovirus and the resistant cells were selected. 方法用MDR病毒轉導人類白血病細胞K562和NB4，獲得耐葯細胞系；
- The researchers wondered what would happen if they exposed p53 in normal and myeloid leukemia cells to antioxidants such as curcumin and resveratrol. 他們想知道，如果讓正常及骨髓性白血症細胞接觸了像薑黃素及白藜蘆醇一類的抗氧化劑，對細胞里的p53有什麽影響。
- Ara c induced apoptosis in human myeloid leukemia cell line HL 60 阿糖胞苷誘導HL-60細胞凋亡的研究
- Methods:NMPs were prepared by high salt extraction and immunized rabbit to raise polyclonal antibody which was used to detect the membrance transferred from 2 D gel of nuclear matrix in normal WBC and acute myeloid leukemia cells by Western blot. 方法 :高鹽抽提法製備白血病細胞NMPs ,免疫動物製備多克隆抗體 ,以此作為免疫學探針對正常WBC和白血病細胞NMPs進行雙向電泳 ( 2DE)_免疫印跡 (Westernblot)檢測。
- Objective To analyse the effect of G-CSF (Granocyte) and GM-CSF on proliferation of myeloid leukemic cells. 目的了解粒細胞集落刺激因子(G-CSF)和粒細胞-巨噬細胞集落刺激因子(GM-CSF)對髓系白血病細胞增殖的影響。
- The results revealed that two cases of amitosis existed in acute myeloid leukemic cells. 結果發現在2例標本中存在白血病細胞的無絲分裂現象。
- Methods:ABC immunocytochemistry was used in detection of the expressions of bcl-2,p170,and CD 34 in leukemic cells from 77 cases of acute leukemia(AL)and 23 cases of chronic myeloid leukemia (CML) patients. 方法 :用ABC免疫細胞化學法檢測 77例急性白血病和 2 3例慢性粒細胞白血病bcl- 2 ,p170 ,CD34 的表達。
- Study on Expression of VEGF in Chronic Myeloid Leukemia Cells 血府逐瘀湯影響慢性粒細胞白血病VEGF表達的體外研究
- Effect of autocrine vascular endothelial growth factor on the biological activity of acute myeloid leukemia cell line HL-60 自分泌血管內皮細胞生長因子對HL-60細胞生物學活性的影響
- Mu 6-1 Myeloid leukemia of spleen. 脾臟類骨髓細胞白血病.
- Study on Induction of Dendritic Cells from Myeloid Leukemia Cell Lines and Their Antitumor Immune Function 白血病細胞系來源的樹突狀細胞誘導及其抗腫瘤免疫功能研究
- acute myeloid leukemia cell 白血病細胞
- The objective is to explore the effect and the mechanism of arsenic trioxide(As 2O 3) on different cell lines of chronic myeloid leukemia(CML). 本研究探討三氧化二砷 (As2 O3)對兩種慢性粒細胞白血病 (CML)細胞系有無作用及作用機制。
- HHV 6 DNA sequence was also present in two leukemia cell lines. 兩株白血病細胞株也擴增出HHV?6DNA序列。
- Objective:To study the variation of p73 gene expression in the process of acute myeloid leukemia(AML) cell line U937 apoptosis induced by adenosine triphosphate (ATP). 目的 :探討三磷酸腺苷 (ATP)誘導 U93 7細胞凋亡過程中 p73 m RNA的表達變化 ,以進一步研究 p73基因在 U93 7細胞凋亡中的作用。
- The histological analyses showed renal cell carcinoma in 76.3% of patients, with less frequent evidence of oncocytoma (6.3%) and acute myeloid leukemia (3.8%). 病理學檢驗分析發現，76.;3%25病患罹患腎細胞惡性腫瘤，而嗜酸細胞瘤（6
