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- 细胞株K562cell line K562
- 白血病细胞株K562malignant myeloid cell lines K562
- GP-7抑制多药耐药白血病细胞株K562/ADM的作用优于依托泊苷。Its inhibitory effect on K562/ADM cells is superior to etoposide.
- 阿霉素纳米粒对白血病多药耐药细胞株K562/ADR耐药性的逆转作用Reversal effect of adriamycin-loaded nanoparticles on human leukemia multidrug -resisitant cell line K562/ADR
- 细胞cell
- L-1时作用则相反。 结论GP-7可抑制多药耐药白血病细胞株K562/ADM的增殖,诱导细胞凋亡。CONCLUSION GP-7 may inhibit multidrug-resistant leukemia K562/ADM cells proliferation and induce apoptosis.
- 复方藤梨根制剂体外对MDR细胞株K562/ADR和K562/VCR细胞积聚和外排阿霉素(ADR)的影响研究The study of effects on accumulation and efflux of intracellular adrimycine with FFTLG for multidrug resistant cell lines K562/ADR and K562/VCR in vitro
- 细胞株cell strain
- 人淋巴因子激活的杀伤细胞对肝癌细胞株凋谢作用的观察A Observaion of Apoptoisi of Liver Cancer Cells Induced by Human LAK Cells
- T24细胞株T24 Cell lines
- NB4细胞株NB4 cell line
- 肝细胞株hepatocyte lines
- A20细胞株A20 cell line
- B9细胞株B9 cell line
- CEM细胞株CEM cell line
- CHO细胞株CHO cell
- CNE细胞株CNE
- EJ细胞株EJ cell line
- H22细胞株H22 cell line
- 人细胞株Human cell line