您要查找的是不是：
- 白血病细胞株K562malignant myeloid cell lines K562
- GP-7抑制多药耐药白血病细胞株K562/ADM的作用优于依托泊苷。Its inhibitory effect on K562/ADM cells is superior to etoposide.
- sFLT-1明显抑制白血病细胞株K562及HL-60的生长,并且随着sFLT-1浓度的增加,其抑制率增加,以用药后48小时抑制作用最明显。sFLT-1 could effectively inhibit the growth of K562 and HL60 cell lines in dose-dependent manner.
- L-1时作用则相反。 结论GP-7可抑制多药耐药白血病细胞株K562/ADM的增殖,诱导细胞凋亡。CONCLUSION GP-7 may inhibit multidrug-resistant leukemia K562/ADM cells proliferation and induce apoptosis.
- 细胞株K562cell line K562
- 阿糖胞苷体外对人白血病细胞株HL-60细胞增殖抑制和诱导凋亡的相关性研究Study on the proliferation inhibition and apoptosis induction of Ara-C on treatment HL-60 in vitro
- 重组人IL-2激活外周血干细胞移植物的抗白血病细胞毒作用Anti-leukemia cytotoxicity of peripheral blood stem cell grafts activated by rhIL-2
- 细胞株cell strain
- 白血病细胞抗原leukemia cell antigen
- 人淋巴因子激活的杀伤细胞对肝癌细胞株凋谢作用的观察A Observaion of Apoptoisi of Liver Cancer Cells Induced by Human LAK Cells
- 白血病细胞leukemia cell
- 复方藤梨根制剂体外对MDR细胞株K562/ADR和K562/VCR细胞积聚和外排阿霉素(ADR)的影响研究The study of effects on accumulation and efflux of intracellular adrimycine with FFTLG for multidrug resistant cell lines K562/ADR and K562/VCR in vitro
- 白血病细胞浸润leukemia myelosis
- T24细胞株T24 Cell lines
- 红白血病细胞K562Erythroleukemia K562 cells
- K562／A02细胞株K562/A02 cell line
- 非细胞毒性剂量的三氧化二砷和苦参碱均可部分逆转有多药耐药表型的细胞株K562/ADM对阿霉素的耐药J险，二者联合应用效果优于单独应用，具有协同作用。K562/ADM had no drug resistance to As2O3 and matrine.
- 从病人体内分离培养的CIK细胞，在体外有强的杀伤K562细胞和裂解髓系白血病细胞的作用；CIK cells generated from patients could kill K562 cells and lysis myeloid leukemia cells in vitro.
- K562／AO2细胞株K5 6 2/AO2 cell strain
- 转录因子Sp1、Sp3对K562白血病细胞增殖、凋亡及耐药性影响的研究Transcription Factors Sp1 and Sp3 Influence K562 Leukemia Cell Proliferation, Apoptosis and Drug Resistance
