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- cardiomyocyte microenviroment 心肌细胞微环境
- Regional microenviroment hypoxia is a common feature in inflammation and malignancy. 局部微环境缺氧是炎症和恶性肿瘤的共同特征。
- The abnormality of the action potential repolarization in cardiomyocyte is closely related with arrhythmia. 心肌细胞动作电位复极化异常与心律失常的发生有密切关系。
- However , MCI-154, a calcium sensitizer, has no impairment on cardiomyocyte relaxation. MCI-154是一种钙增敏剂,但不损害舒张功能。
- Objective To investigate cardiomyocyte apoptosis in viral myocarditis (VMC)mice and its relationship with VMC. 目的 研究病毒性心肌炎(VMC)小鼠心肌细胞凋亡及其意义。
- Protective effects of continuous warm blood cardioplegia on cardiomyocyte sarcolemma of canine heart during open-heart surgery. 温血心停跳液持续灌注对心内直视术中犬心肌细胞质膜的保护作用。
- Objective To study ultrastructure changes of myocardium and morphic changes of cardiomyocyte apoptosis in mice with viral myocarditis(VMC). 目的研究病毒性心肌炎(VMC)小鼠心肌超微结构改变及细胞凋亡的形态学变化。
- If the same hematopoietic stem cell gies rise to a cardiomyocyte, this is unexpected and therefore qualifies as transdifferentiation. 而如果分化为心肌细胞,那就属于意料外的分化,可定义为转分化。
- Abstract: The present work was to investigate the effects of vasonatrin peptide (VNP) on cardiomyocyte protein synthesis induced by moderate hypoxia. 低氧性心室肥厚常见于慢性缺血性心脏病,虽然初期心肌肥厚是一种有益的代偿反应,但持续性心肌肥厚最终可导致扩张性心肌病、心衰和猝死。
- OEA enhanced fatty acid oxidation in skeletal muscle strips, dissociated hepatocytes, and primary cardiomyocyte cultures. OEA在骨骼肌、游离肝细胞、和主要心肌细胞的培养菌中强化脂肪酸氧化。
- This article reviews the mechanisms, control genes and signalling pathways mediating cardiomyocyte apoptosis in myocardial ischemia/reperfusion injury. 现就心肌缺血/再灌注损伤中细胞凋亡发生的原因、参与调控的相关基因及信号途径作一综述。
- Cardiomyocyte apoptosis was detected with in situ terminal deoxynucleotidyl transferase(TdT) mediated dUTP nick end labeling(TUNEL) and DNA gel electrophoresis. 末端脱氧核糖核酸转移酶介导的dUTP切口末端标记技术(TUNEL)和DNA凝胶电泳检测心肌细胞凋亡。
- Objective To explore the effects and the mechanisms of growth hormone (GH) on cardiomyocyte apoptosis in adriamycin (ADR)-induced cardiomyopathy in rats. 目的:探讨生长激素(Growth hormone,GH)对阿霉素(Adriamycin,ADR)心肌病大鼠心肌细胞凋亡的影响及其机制。
- Objective: To evaluate influences of regular dose of adriamycin (ADR) on heart function and sarcoplasmic reticulum (SR) Ca 2+ ATPase in cardiomyocyte of rabbits. 目的 :探讨治疗剂量阿霉素 (ADR)对心脏血流动力学及心肌肌浆网 Ca2 + - ATP酶活性的影响。
- Reactive oxygen species can mediate signalling pathways in cardiac hypertrophy and cardiomyocyte apoptosis, and lead to endothelial dysfunction by inactivating nitrogen monoxide. 活性氧通过信号通路介导心肌细胞肥大和凋亡;通过灭活一氧化氮等机制导致内皮功能紊乱。
- This review describes the origin and precise location of HSC in the embryo and in AGM region, the hematopoietic microenviroment and the hemato- poietic regulatory mechanisms in AGM region. 本文综述了有关造血干细胞在胚胎和AGM区中的起源与定位 ,AGM区造血微环境以及其中的分子调控机制等问题。
- Conclusion: LN and FN in endometrium may play a critical role in construction of microenviroment for embryo implantation, and may also he an important regulator in the process of implantation. 结论:层粘连蛋白和纤维粘连蛋白在营造胚泡着床微环境和植入调节中发挥重要作用。
- In IPC group the apoptosis ratio of cardiomyocyte markedly decreased and the ultrastructural damage attenuated (P<0.05). UCP2 was induced following ischemic preconditioning(P<0.05, vs. IR). IPC组Rainio评分和心肌细胞凋亡率均低于IR组(P<0.;05);IPC组的UCP2mRNA和蛋白表达水平均较IR组明显增加(P<0
- Methods Our experiment established animal model of VMC by the way of coxsackie virus B3(CVB3) inoculation,then we studied the changes and apoptosis of cardiomyocyte by ways of microscope and electron microscope. 方法本实验在接种柯萨奇病毒B3(CVB3)建立VMC动物模型的基础上,用光学显微镜及电子显微镜观察心肌细胞的病变及心肌细胞凋亡。
- The RT-PCR studies showed that 1.6mg/kg of As2O3 enhanced the L-type calcium channel mRNA expression(compared with control groups,P<0.01). Conclusion As2O3 could prolong the QT interval of guinea pig cardiomyocyte. RT-PCR结果显示1.;6mg/kg As2O3可以使豚鼠心肌L型钙通道mRNA表达增强(与对照组相比;P<0