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- renal interstitial fluid 腎間質液
- OBJECTIVE: The renal interstitial fibrosis(RIF) is almost the common pathway by which all the various chronic kidney diseases (CKD) progress to end stage renal disease(ESRD). 目的：腎間質纖維化(RIF)是各種不同病因的慢性腎臟病(CKD)隨著病程進展進入終末期腎病(ESRD)的一個進行性、不可逆的共同途徑。
- Tao He Cheng Qi Tang can restain RIF, which mechanism related to restrain the growth factor of renal interstitial fibroblasts in RIF and decrease extracellular matrix. 桃核承氣湯抗腎間質纖維化的作用是通過抑制促纖維生長因子的生成和減少細胞外基質而實現的。
- Objective To investigate the relationship between renal biopsy pathology (RBP) and spiral CT dynamic contrast scanning (SCTDCS) in renal interstitial fibrosis (RIF). 目的通過不同程度腎間質纖維化(RIF)腎穿刺病理檢查(RBP)與螺旋CT薄層動態增強掃描(SCTDCS)對照,探討RIF病理與螺旋CT診斷指標之間的相關性。
- Interstitial cells; interstitial fluid. 間質細胞；間質流體
- One or more of these factors can act to increase interstitial fluid volume. 上述一個或幾個因素可使間質液容量增加。
- Renal interstitial fibrosis plays an important role in diabetic nephropathy,and epithelial-to-mesenchymal transition(EMT) may be the key step. 腎小管病變及腎間質纖維化在糖尿病腎病中的重要作用逐漸被認識,其中腎小管上皮細胞向間充質細胞轉化(EMT)可能是其發揮作用的關鍵環節,與腎間質纖維化的程度相平行。
- Conclusion tetrandrine, tetramethylpyrazine and amygdalin may play important roles in prevention and reversion of renal interstitial fibrosis. 結論漢防己甲素、川芎嗪、苦杏仁甙在預防及逆轉腎間質纖維化中起重要作用。
- CONCLUSION: Decoction of BSHXXZ could markedly improve renal function and delay the progression of glomerulosclerosis and renal interstitial fibrosis. 結論：補腎活血泄濁湯能夠明顯保護病鼠腎功能，明顯減輕病鼠細胞外基質沉積，延緩病變腎小球硬化的發生。
- The layer will finally disappear when the slope of the geotherm is too low to produce interstitial fluid at any depth. 當地熱斜率太低，以至任何深度上都不能產生空隙流體時，低速層將最後消失。
- Gao YL, Chen YP, Dong HR, et al. Experimental study on renal interstitial fibrosis of chronic aristolochic acid nephropahty in rats. Chin J Nephrol2005;21(1):31-35. 高艷麗;諶貽璞;董鴻瑞;等.;慢性馬兜鈴酸腎病腎間質纖維化發病機制的初步探討
- Abstract： Objective To study the mechanism of renal interstitial fibrosis after unilateral ureteral Obstruction and effects of enalapril on the process of the fibrosis. 摘 要： 目的 研究單側輸尿管梗阻后腎間質纖維化發生機理及血管緊張素轉換酶抑製劑依那普利對其的調節作用。
- The microcirculation is the place where the blood and interstitial fluid to carry out the exchange of substances. 微循環是血液和組織之間進行物質交換的場所。
- Now many stata showed that endothelin had powerful effect on renal of physiology or pathology,which took part in the occurrence and development of renal interstitial fibrosis. 有研究表明,內皮素作為細胞因子之一參與了腎間質纖維化的發生和發展,而內皮素受體拮抗劑通過拮抗內皮素的作用,而發揮腎保護作用。
- Objective Investigate the protective and therapeutic effect of Yishenhuoxueqingli recipe on renal interstitial fibrosis of unilateral ureteral obstruction(UUO)rats. 目的探討依據益腎活血清利法組方對進展性腎間質纖維化的防治作用。
- Objective:To study the influence of total salvianolic acid(TSA) on the renal interstitial fibrosis caused by unilateral ureteral obstruction (UUO) in rats. 目的觀察丹參總酚酸對單側輸尿管梗阻(UUO)大鼠腎間質纖維化的影響。
- A point which has been overlooked hitherto is that any build-up of interstitial fluid would produce a marked lowering of the thermal conductivity. 至今仍被忽視之點，即空隙流體的增加將使導熱率明顯降低。
- Objective To study the effect of anisotropic hydraulic conductivity coefficient on interstitial fluid flow in solid tumor. 摘要目的研究實體腫瘤組織間質各向異性傳導係數對流體流動的影響。
- To investigate the expression of matrix metalloproteinase-9/tissue inhibitor of matrix metalloproteinase-1(MMP9/TIMP1)in rats with renal interstitial fibrosis and the mechanism of renal regulation of "Fu fang bie jia ruan gan fang". 目的探討基質金屬蛋白酶9/基質金屬蛋白酶組織抑製劑(1MMP-9/TIMP-1)在腎間質纖維化中的表達情況及其復方鱉甲軟肝方的調節作用。
- Deng YY, Chen YP, Hu ZY, et al.The mechanism of renal interstitial fibrosis after unilateral ureteral obstuction in rats and effect of enalapril on this process[J].Chin J Nephrol, 2000, 16(4): 234-7. [2]鄧躍毅;陳以平;胡仲儀;等.;單側輸尿管梗阻后腎間質纖維化的產生機制及依那普利對其調節作用[J]
