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- mean renal blood flow 平均腎血流
- The changes of systolic peak velocity (VS), diastolic peak velocity (VD), mean glow velocity (VM), pulsatility index (PI) and resistent index (RI) of renal blood flow were observed. 並觀測腎血流收縮期峰值流速(VS)，舒張末期峰值流速(VD)，平均流速(VM)，搏動指數(PI)及阻力指數(RI)的變化。
- The age-related reduction in renal blood flow is accompanied by a gradual loss of functioning glomeruli. 年齡相關性的腎血流量減少並伴隨著功能性腎單位的逐漸減少。
- The mechanisms of verapamil protection against ARF are: verapamil increased renal blood flow and GFR, improved renal cortical blood circulation, etc. 其機理與增加腎血流量、善腎皮質缺血區域的血液循環等因素有關。
- In fact cardiac output or renal blood flow may be above normal at the same time that capillary perfusion is very deficient. 事實上，心輸出量，或腎血流量可能就在毛細血管灌注十分不定時，反而高於正常的流量。
- There are inappropriate changes of renal hemodynamics that renal blood flow reduce 50%,and glomerular function reduce rapid at the process of ARF. ARF發生時，腎臟存在異常的腎血流動力學的改變，腎血流量減少50%25，腎小球有效濾過壓下降。
- Abstract： Objective:To study the renal blood flow perfusion and its clinical significance in hydronephrosis by Color Doppler Energy(CDE). 摘 要： 目的：利用彩色多普勒能量顯像技術(CDE)探討不同程度腎積水患者腎臟的血流灌注及其臨床意義。
- He found that dopamine increased renal blood flow but did not improe medullary PO2.This finding is most likely due to the inhibition of tubulo-glomerular feedback (TGF). 結果發現，多巴胺增加腎血流但並不增加髓質的PO2，這很可能是因為球管反饋（TGF）抑制的原因。
- He found that dopamine increased renal blood flow but did not improve medullary PO2.This finding is most likely due to the inhibition of tubulo-glomerular feedback (TGF). 結果發現，多巴胺增加腎血流但並不增加髓質的PO2，這很可能是因為球管反饋（TGF）抑制的原因。
- The earliest abnormal of renal blood flow occured in hypertensives included that the end diastolic velocity (V d) of interlobar artery was decreased and the acceleration time (AT) of segmental and interlobar artery was increased. 葉間動脈舒張期末血流速度 (Vd)的減慢、血流峰速加速時間的延長是高血壓病患者最早出現的腎血流動力學異常 ;
- The effects of 4 week treatment with tetrandrine (200mg3/d,po) and nimodipine (40mg3/d,po) on renal blood flow (RBF) and glomerular filtration rate (GFR) were studied in 30 patients with essential hypertension,in single blind,random and cross-over trial. 採用單盲、隨機和交叉的方法，觀察粉防己鹼（200mg3/d，po，4w）和尼莫地平（40mg3/d，po，4w）對30例原發性高血壓患者的腎血流動力學的影響。
- After renal ischemia reperfusion, the high expression of iNOSmRNA in tubules is an important molecular mechanism of renal ischemia reperfusion injury (2) Keep cNOS/iNOS constant may be important for mudulation of renal blood flow and GFR. 缺血再灌注后 ,皮質腎小管上皮中iNOSmRNA的高表達是導致腎缺血再灌注損傷的重要分子機制。 (2 )cNOS/iN OS比值與缺血再灌注過程中腎功能的變化密切相關 ,該比值的恆定對腎血流量和腎小球濾過率(GFR)的調節可能具有重要意義
- Reduced RFV/Co reflected the degree of renal injury.3.Many clinical factors affected the change of renal blood flow dynamics such as Co, hematocrit (HCT), and metabolic acidosis. 4. 3.;心輸出量的大小、血液粘滯度及祛斑是否合併代謝性酸中毒將對腎血流動力學改變有直接影響。
- High serum uric acid resulted in renal function letdown or descending in renal blood flow is more frequent in the patients with hypertension than those with normal blood pressure[1]. 由於高血壓病人腎功能減退或腎血流量下降，故高血壓患者中高尿酸血症較正常血壓者多[1]。
- METHODS Using one kidney one clip renal hypertensive model, we determined the arterial pres-sure, heart weight, renal blood flow (RBF) and glomerular filtration rate (GFR) at varied treatment. 方法 採用一腎一包紮型腎性高血壓模型 ;測定不同條件下動物血壓、心臟重量、腎血流量(RBF)和腎小球濾過率 (GFR) .
- METHOD: Rabbit models with hemorrhagic shock and Smao shock were used, and the influences of drug on pressure, renal blood flow, lipid peroxide and cell ultrastructure of both models were observed. 方法採用家兔失血性休克模型及腸系膜上動脈夾閉型休克模型,觀察藥物對休克模型動物的血壓、腎血流量、脂質過氧化物、細胞超微結構等的影響。
- effective renal blood flow, ERBF 腎有效血流量
- Keywords noradrenaline;renal blood flow;angiotonics; 去甲腎上腺素;腎臟血流;血管收縮劑;
- The stoppage of blood flow through a blood vessel or body part. 止血法阻止血液流過某一血管或身體某一部位
- Relationship of renal function with renal volume and renal blood flow in autosomal dominant polycystic kidney disease 常染色體顯性多囊腎病腎功能與部分參數的相關性分析
