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- 內皮型NO合酶eNOS
- 誘導型NO合酶iNOS
- 胰島素抵抗大鼠血糖正常階段心肌內皮素1、內皮型一氧化氮合酶表達的改變Alteration of myocardial ET-1 and eNOS gene expressions in insulin-resistant rats during normal glycemia stage
- 誘生型NO合酶iNOS
- 內皮型一氧化氮合酶基因多態性與2型糖尿病併發冠心病的相關性研究A study on relationship between the endothelial constitutive nitric oxide synthase gene polymorphism and the coronary disease in patients with type 2 diabetes
- 內皮NO合酶在血管疾病中的不同作用:NO合成減少氧解偶聯和其藥理學逆轉作用Janus-faced role of endothelial NO synthase in vascular disease: uncoupling of oxygen reduction from NO synthesis and its pharmacological reversal
- 燃料型NOfuel-NO
- 內皮型一氧化氮合成酶Endothelial nitric oxide synthase
- 先天性心臟病肺動脈高壓患者肺組織內皮型一氧化氮合成酶的表達及肺血管重建Expression of eNOS and vascular remodeling in human pulmonary vascular of pulmonary hypertension with congenital heart disease
- （4）NO合酶阻斷劑L-NAME(0.5 mmol/L)對GST的效應無影響。Pretreatment with NG-nitro-L-arginine methyl ester (L-NAME, 0.5 mmol/L), a nitric oxide (NO) synthase inhibitor, did not affect the effects of GST on AV node cells.
- 內皮脂酶endothelial lipase
- 內皮型一氧化氮合成酶基因在重度妊高征胎盤絨毛組織的表達Expression of Endothelial Nitric Oxide Synthase Gene in Placental Villus of Preeclampsia
- NO供體L 精氨酸 (L Arg)與ET受體拮抗劑TAK 0 44在一定程度上可減輕肝I/R損傷 ,而NO合酶抑製劑L NAME進一步加重了損傷。NO donor L Arg and ET receptor antagonist TAK 044 could alleviate the hepatic I/R injury in some degree, whereas NO synthase inhibitor L NAME aggravated the damage.
- 血管內皮細胞型PAIPAI-1 also
- 胸苷磷酸化酶／血小板衍化內皮細胞生長因子Thymidine phosphorylase/platelet-derived endothelial cell growth factor
- 內皮細胞型一氧化氮合成酶eNOS
- 目的 探討兔角膜內皮細胞上Na+ K+ ATP酶的分佈。Objective To study the distribution of Na + K + ATPase in corneal endothelial cells of rabbit.
- 2型糖尿病併發腦血管病患者內皮功能的研究A Study on Endothelium Function in Patients with Type 2 Diabetes Mellitus Complicated with Cere-brovascular Disease
- 誘髮型一氧化氮合酶在人類鼻黏膜上皮細胞中的表達Effects of hypoxia on the expression of inducible isoform of nitric oxide syntheses in nasal mucosal epithelial cells
- 預防接觸型角膜內皮鏡檢查致感染的護理體會Nursing experience of preventing infection caused by contact keratoscopy examination
