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- 内皮型NO合酶eNOS
- 诱导型NO合酶iNOS
- 胰岛素抵抗大鼠血糖正常阶段心肌内皮素1、内皮型一氧化氮合酶表达的改变Alteration of myocardial ET-1 and eNOS gene expressions in insulin-resistant rats during normal glycemia stage
- 诱生型NO合酶iNOS
- 内皮型一氧化氮合酶基因多态性与2型糖尿病并发冠心病的相关性研究A study on relationship between the endothelial constitutive nitric oxide synthase gene polymorphism and the coronary disease in patients with type 2 diabetes
- 内皮NO合酶在血管疾病中的不同作用:NO合成减少氧解偶联和其药理学逆转作用Janus-faced role of endothelial NO synthase in vascular disease: uncoupling of oxygen reduction from NO synthesis and its pharmacological reversal
- 燃料型NOfuel-NO
- 内皮型一氧化氮合成酶Endothelial nitric oxide synthase
- 先天性心脏病肺动脉高压患者肺组织内皮型一氧化氮合成酶的表达及肺血管重建Expression of eNOS and vascular remodeling in human pulmonary vascular of pulmonary hypertension with congenital heart disease
- (4)NO合酶阻断剂L-NAME(0.5 mmol/L)对GST的效应无影响。Pretreatment with NG-nitro-L-arginine methyl ester (L-NAME, 0.5 mmol/L), a nitric oxide (NO) synthase inhibitor, did not affect the effects of GST on AV node cells.
- 内皮脂酶endothelial lipase
- 内皮型一氧化氮合成酶基因在重度妊高征胎盘绒毛组织的表达Expression of Endothelial Nitric Oxide Synthase Gene in Placental Villus of Preeclampsia
- NO供体L 精氨酸 (L Arg)与ET受体拮抗剂TAK 0 44在一定程度上可减轻肝I/R损伤 ,而NO合酶抑制剂L NAME进一步加重了损伤。NO donor L Arg and ET receptor antagonist TAK 044 could alleviate the hepatic I/R injury in some degree, whereas NO synthase inhibitor L NAME aggravated the damage.
- 血管内皮细胞型PAIPAI-1 also
- 胸苷磷酸化酶/血小板衍化内皮细胞生长因子Thymidine phosphorylase/platelet-derived endothelial cell growth factor
- 内皮细胞型一氧化氮合成酶eNOS
- 目的 探讨兔角膜内皮细胞上Na+ K+ ATP酶的分布。Objective To study the distribution of Na + K + ATPase in corneal endothelial cells of rabbit.
- 2型糖尿病并发脑血管病患者内皮功能的研究A Study on Endothelium Function in Patients with Type 2 Diabetes Mellitus Complicated with Cere-brovascular Disease
- 诱发型一氧化氮合酶在人类鼻黏膜上皮细胞中的表达Effects of hypoxia on the expression of inducible isoform of nitric oxide syntheses in nasal mucosal epithelial cells
- 预防接触型角膜内皮镜检查致感染的护理体会Nursing experience of preventing infection caused by contact keratoscopy examination