您要查找的是不是:
- Akbar AN,Salmon M. Celluar environments and apoptosis:tissue microenvironments control activated T cell death [J]. Immunol Today, 1997,18(2):72-75. 中国神经免疫学和神经病学杂志2000年第2期第7卷综述作者:赵宏纲李一单位:白求恩医科大学免疫学教研室;长春130021关键词:T细胞;凋亡;中枢神经系统...
- HLA DR Ag was strongly expressed on activated T cells,macrophages. 巨噬细胞、T淋巴细胞高表达HLA DR抗原 (P <0 0 5 )。
- Conclusions: Macrophages from traumatized mice may suppress T cell functions via altering messenger molecule levels in activated T cells. 结论: 创伤后巨噬细胞可通过改变活化T细胞内信使分子水平,进而抑制T细胞功能。
- Objective: To study the correlation of multidrug resistance protein P170 and active T cell in acute myeloblatlic leukemia(AML). 研究目的:探讨急性髓性白血病耐药蛋白P_(170)与活化T细胞的关系。
- The B7 molecules can be expressed on APCs including monocytes, macrophages, B cells, dendritic cells,and activated T cells. 并进一步证明了是肿瘤细胞表面的B7分子协同激活了肿瘤抗原特性的CD8+CTL细胞。 不久Allison等人也报道了相似结果,肯定了前者的成果。
- IL-2 is mainly produced in activated T cells and could be released into blood from them in vivo. The half-life of IL-2 is very short, less than 10 min in peripheral blood. 在体状态下,IL-2主要由活化的T细胞产生并可释放入血,但IL-2在外周血中的半衰期很短,还不足10min。
- Results:36 cases with RPU were diagnosed and HLA DR Ag was strongly expressed on activated T cells, macrophages,epithelia and vascular endothelia in RPU tissues. 结果 :RPU组溃疡粘膜经组织病理学检查均符合 RPU的诊断 ,病变粘膜中巨噬细胞、T淋巴细胞、上皮细胞、血管内皮细胞异常表达 HLA- DR抗原。
- CLA, expressed by activated T cells, can accelerate the homing of T lymphocytes to the skin, which is important to the occurrence, development and relapse of psoriasis. 细胞在银屑病的发病中扮演重要角色,活化的T细胞表达皮肤淋巴细胞相关抗原,可加速T细胞的皮肤归巢,对银屑病的发生、发展及复发有重要作用。
- GLB 7 didn?t influence the production of IP 3 and DAG in active T cells. GLB7对活化的T细胞中IP3 和DAG未产生明显影响。
- FasL expression in transfected chondrocytes was analyzed by FACS. The apoptosis rate of Fas+ Jurkat cells and activated T cells with FasL+ chondrocytes induction were detected by JAM test. FACS检测FasL的表达,应用JAM试验测定FasL+的软骨细胞诱导Fas+的Jurkat细胞及活化的T细胞的凋亡率。
- METHODS: The gene encoding human BTLA was amplified by RT-PCR from PHA activated T cells,and then the target gene fragment was inserted into retrovirus vector pEGZ-Term after being digested with EcoR I and BamH I. 方法:通过RT-PCR法从经PHA活化的人外周血T细胞中克隆出人BTLA编码区全长基因,经EcoR I和BamHI双酶切后插入逆转录病毒载体pEGZ-Term中构建成重组载体pEGZ-Term/BTLA。
- After T cells being activated by PHA or by anti-CD3 with anti-CD28 mAbs, the GL50 and ICOS expressions on activated T cells were detected by means of FACS on Oh, 24h, 48h, 72h, and 96h. 将人外周血T细胞用PHA或联合抗CD3和抗CD28单抗激发后,分别于0h、24h、48h、72h、96h用免疫荧光标记和流式细胞术分析,检测细胞表面GL50分子和ICOS分子的动态表达,结果显示:GL50分子的表达在T细胞活化后24h即开始上调,72h时达到高峰;
- The decrease of activated T cells and the increasing Tregs suggests that T cells in the patients with PNS are over-activated, and the immunesuppression function mediated induced by Tregs is decreased. 并有T细胞过度激活及调节性T细胞所介导的免疫抑制功能的降低。
- Superantigen SEA has great ability to active T cells, B7.1 molecules can provide the second signal for T cell activation and play a very important role during the process of antigen presenting and recognizing. 超抗原SEA具有强大的T细胞激活能力,而B7.;1分子可为T细胞活化提供第二信号,在抗原递呈和识别过程中具有十分重要的作用。
- To activate T cells, one receptor responds to a specific foreign invader, or antigen, and another, CD28, signals a green light for an immune response. 一般T细胞的活化,除了要有一个特定外来物(抗原)与细胞表面受体结合,还需要另一个受体蛋白CD28发出免疫反应的绿灯信号。
- Conclusion:B7 vaccine could improve the immunogenicity of tumor cells , activate T cells and enhance in vivo the anti-tumor immunity. 结论:B7-1基因的导入并有效表达可增强肿瘤细胞免疫原性,激活T细胞,提高宿主抗瘤免疫。
- Abstract: Objective: To compare PGD210 plasmid transfected dendritic cells with untransfected dendritic cells in activating T cells to kill the K562 cells. 目的: 比较PGD210转染的DC与未经转染的DC,刺激T细胞诱发免疫应答,有效激发T细胞并杀伤K562细胞的功能。
- CD152, a ligand expressed on the surface of the activated T cells that inhibits co-stimulatory signals, is associated with negative regulation of T-cell activation in the antigen-presenting process. CD152是表达于活化T细胞表面的共刺激信号抑制配体,与抗原递呈过程中T细胞活化的负调控有关。
- nuclear factor of activated T cells 激活T细胞核因子
- Nuclear factors of activated T cells 活化T细胞核因子