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- CONCLUSION Actinomycin D can stimulate encephalitis B virus replication. 结论:放线菌素D可使持续感染的乙脑病毒复制增加。
- These results will be helpful for understanding the photodynamic processes of ADE and actinomycin D. 这些结果将为详细了解ADE以及放线菌素D的光动力作用过程提供理论依据。
- THE EFFECT OF CYCLOHEXIMroE AND ACTINOMYCIN D ON POLLEN GERMINATION AND POLLEN TUBE GROWTH OF PINUS BUNGEANA AUCC. 放线菌酮和放线菌素D对白皮松花粉萌发和花粉管生长的调控作用
- Actinomycin D can inhibit expression of C myc mRNA and intimal hyperplasia in graft. 大剂量应用放射菌素D可以抑制C mycmRNA表达 ,从而抑制移植血管内膜增殖。
- The protection for the mice sensitized by actinomycin D from endotoxin-induced lethality was investigated. 并以内毒素制备家兔发热模型,测定其肛温变化。
- Objective: To prepare actinomycin D vaginal bioadhesive tablets with sustained effect and less toxicity. 目的:研制一种持续作用时间长,毒副作用小的放线菌素D阴道生物黏附片。
- RNA synthesis in macrophages, however, was improved as the dosage of actinomycin D increased. 但随着放线菌素D浓度增高,反而促进巨噬细胞的RNA合成。
- While in denervated muscle, actinomycin D inhibited proliferation of satellite cells aswell as increase of endocytosis, but could not prevent muscle atrophy after denerva-tion. 在去神经的肌肉中,放线菌素D抑制了卫星细胞增殖的同时还抑制了胞纳的增加,但不能阻止去神经肌肉的萎缩。
- TPA and 5-Azacytidine enhanced the effects of PHA,Actinomycin D inhibited but Hydroxyurea and Colchicine had no influence on it. TPA、5-氮胞苷可增加PHA的作用,羟基脲和秋水仙素不影响,而放线菌素D可抑制pHA的效应。
- Since there is antigenicity in the polypeptide structure Iof actinomycin D itself, results obtained with two types of antibiotics are consistent. 由于放线菌素D本身的多肽结构而具有抗原性,因此,用两种抗菌素所得的实验结果是相符的。
- Prados J,Melguizo C,Marchal JA,Velez C,Alvarez L,Aranega A.Therapeutic diffeentiation in a human rhabdomyosarcoma cell line selected for resistance to actinomycin D. 张虹;韩嫒嫒;周晓冬;何彦津;宋国祥.;眼眶腺样囊性癌动物模型的建立及局部化学治疗的初步观察
- In order to find how Na2SeO3 enhancing the biosynthesis of GSH-Px, transcription inhibition Actinomycin D(AMD) and translation inhibitor cyclonheximide (CHM) were used. 麦苗生长三天后,在含0.;75mg/l的Na_2SeO_3培养液中加入不同浓度的抑制剂,根施麦苗。
- The induction of cellulase in washed mycelia was inhibited also by actidione, 5-fluouraeil and 6-azauracil greatly, but inhibited by actinomycin D only slightly. 槐糖对菌丝体纤维素酶的诱导形成能不同程度地被核酸代谢抑制剂如放线菌酮、5-氟尿嘧啶、6-氮杂尿嘧啶和放线菌素D抑制。
- The macrophages did not respond to the stimulation effect of actinomycin D in the presence of sucrose,and the complete inhibition of RNA synth esis was observed. 蔗糖的存在能使巨噬细胞丧失对放线菌素D这种刺激作用反应的能力,其RNA合成完全被抑制。
- RD/VCR cell line was cross-resistant to vincristine (VCR), adriamycin (ADM), etoposide (VP16) and actinomycin D (DAC), but not resistant to fluorouracil (5-FU) and cisplatin (DDP). RD/VCR细胞对长春新碱、阿霉素(ADM)、鬼臼乙叉甙(VP16)和放线菌素D(DAC)交叉耐药,对氟尿嘧啶(5-FU)、顺铂 DDP)无明显耐药性。
- Objective To study the effects of actinomycin D(ACTD) and VM-26 on proliferation in rat glioma cells C6.Methods The inhibition effect of actinomycin D or VM-26 was compared in treatment of rat glioma cell line by 3H-TdR method in vitro. 目的研究放线菌素D(Actinomycin D,ACTD)和替尼泊甙(Teniposide,VM-26)对大鼠胶质瘤细胞增殖的影响。 方法应用免疫细胞化学方法,观察C6细胞经不同剂量ACTD和VM-26作用48h后PCNA、CyclinD1表达的改变,FCM法分析细胞凋亡;
- Observed the effects of mRNA transcriptional inhibitor Actinomycin D, protein synthesis inhibitor Actidione and NO synthase(NOS) inhibitor N-monomethyl-L-arginine(L-NMMA) on the activity of iNOS and intracellular GSH in mouse peritoneal macrophages. 2观察 m RNA转录抑制剂、蛋白质合成抑制剂和 NOS抑制剂对巨噬细胞 i NOS活性和细胞内 GSH水平的影响 ;
- The effect was blocked effectively by the RNA transcriptional inhibitor Actinomycin D, protein synthesis inhibitor Actidione and N-monomethyl-L-arginine (L-NMA), a NO synthaase (NOS) inhibitor. 结果还表明,LTN的这一促进作用能被RNA转录抑制剂放线菌素D、蛋白质合成抑制剂放线菌酮和一氧化氮合酶(NOS)抑制剂L-NMA所抑制。
- After actinomycin D treatment and cellular DNA electrophoresis and detection of TUNEL(TdT mediated bio dUTP nick endlabeling), Sf9 35 cells were found to be resistant to apoptosis induced by actinomycin D. 经放线菌素D处理后的细胞核酸电泳和TdT介导bio?DUTP缺口末端标记(TUNEL)试剂盒检测,证实Sf9?35具有抗凋亡特性。
- By use of laser flash photolysis and steady state absorption techniques, a systematic study was carried out on the interaction of ADE, which is a kind of model compound of actinomycin D, with DNA and BSA. 利用激光光解结合稳态吸收光谱技术,对放线菌素D的模型化合物ADE与DNA和牛血清蛋白的相互作用进行了系统研究。