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- PACAP1 38 stimulated the proliferation of human pancreatic cancer cells ASPC 1 which were dose dependent. PACAP1-38促进ASPC-1的生长,促进ASPC-1细胞cAMP、Ca2+的生成,且呈剂量依赖性;
- This study investigated the death pattern of pancreatic cancer cells line Panc-1 induced by ART and its mechanisms. 本研究旨在探讨ART诱导胰腺癌细胞死亡的方式及机制,为探寻治疗胰腺癌的新策略提供思路。
- Studies have shown it can cause prostate and pancreatic cancer cells to kill themselves. It also helps prevent and heal stomach ulcers. 研究发现,辣椒素可以使前列腺癌和胰腺癌的癌细胞自我毁灭,还能够预防和治疗胃溃疡。
- The style of K-ras gene point mutation at codon 12 was GAT in human pancreatic cancer cell line. 人胰腺癌细胞株PANC-1存在K-ras基因的点突变,其突变方式为CAT。
- Blocking shh pathway can inhibit the growth of pancreatic cancer cell and induce apoptosis. 通过特异性阻断该信号通路,可以抑制胰腺癌细胞增殖,并促进细胞凋亡。
- Using a human pancreatic cancer cell line, she and her team found that adding thymoquinone killed approximately 80 percent of the cancer cells. 使用人体胰腺癌细胞株,她的小组发现加入百里香醌能够杀灭80%25的癌细胞。
- Studies have also shown it can cause prostate and pancreatic cancer cells to kill themselves.It also helps prevent and heal stomach ulcers. 研究还发现,辣椒素可以使前列腺癌和胰腺癌的癌细胞自我毁灭,还能够预防和治疗胃溃疡。
- Conclusions: there were over expressions of PTCH mRNA and Smo mRNA in human pancreatic cancer tissues and human pancreatic cancer cell lines. 小结:PTCH和Smo mRNA在人胰腺癌细胞系和组织中均存在过表达;
- METHODS The cytotoxic effects of 5-FU on AsPC-1, Capan-1, Mia-PaCa-2, Panc-1 and T3M4 pancreatic cancer cells were assessed by the sulforhodamine B protein assay (SRB). 方法 用 SRB方法检测5 - FU对胰腺癌细胞株 As PC- 1、Capan- 1、Mia- Pa Ca- 2、Panc- 1和 T3 M4的毒性作用。
- Anderson team shows in lab experiments that inhibiting the protein in pancreatic cancer cells leads to a form of programmed cell suicide called autophagy, or self-digestion. Anderson研究组成员的实验证实了:抑制胰腺癌细胞中TG2蛋白能导致细胞的程序化死亡,被称为自噬或者自我消化。
- Methods Three pancreatic cancer cell lines(Panc-1,Sw 1990,Aspc-1) were chosen to detect the mRNA expressions of hENT1,hENT2,hCNT1,hCNT2 and hCNT3 with RT-PCR. 方法选择胰腺癌细胞株Panc-1、Sw1990和Aspc-1进行实验。 RT-PCR法检测细胞株hENT1、hENT2、hCNT1、hCNT2和hCNT3的mRNA表达。
- Methods: mRNA expressions of hENT1, hENT2, hCNT1, hCNT2 and hCNT3 in Panc-1, Sw1990 and Aspc-1 pancreatic cancer cell lines were investigated by RT-PCR. 方法:逆转录-聚合酶链反应(RT-PCR)检测三种胰腺癌细胞株Panc-1,Sw1990和Aspc-1中hENT1、hENT2、hCNT1、hCNT2、hCNT3的mRNA表达。
- Molecular cytogenetic techniques hake to be used beside conventional G-band karyotyping for accurate identifying abnormal chromosomes of pancreatic cancer cell lines. 若要准确识别染色体异常,还需结合其他分子细胞遗传学技术。
- Analyze and contrast the apoptosis rate of pancreatic cancer cell groups induced by 5-Fu、tea polyphenols、5-Fu and tea polyphenols、sulfasalazine and 5-Fu and group of compare. 用流式细胞仪测定、比较单用茶多酚组、5-Fu组、5-Fu加茶多酚组、柳氮磺胺吡啶加5-Fu组及对照组细胞诱导凋亡率。
- TK-gene system (retrovirus transfer of the herpes simplex virus thymidine kinase gene) is one of the much more mature plans. We have succeeded in transmitting herpes TK gene into human pancreatic cancer cells, named SW1990 by retrovirus. 由缺陷型逆转录病毒介导的TK基因系统(RV-HSV-TK)是众多肿瘤生物治疗方法中一个技术较为成熟的方案,本实验采用该系统在体外成功地转染了人胰腺癌细胞株SW1990并能够稳定传代培养,转染了TK基因的SW1990细胞(SW±K),其生长曲线与未转染的SW1990细胞无差异。
- They also found these cells are at least 100 times more tumorigenic than cancer cells that did not hae one of three protein markers they beliee to be associated with pancreatic cancer stem cells. 他们还发现这些细胞至少比一般的癌细胞(缺乏他们认为的和胰腺癌干细胞相关的蛋白标记物的癌细胞)的致瘤能力大100倍。
- They also found these cells are at least 100 times more tumorigenic than cancer cells that did not have one of three protein markers they believe to be associated with pancreatic cancer stem cells. 他们还发现这些细胞至少比一般的癌细胞(缺乏他们认为的和胰腺癌干细胞相关的蛋白标记物的癌细胞)的致瘤能力大100倍。
- Salvicine (SAL) was used to reverse the drug resistance in a gemcitabine-resistant pancreatic cancer cell line (SW1990-GEM). RT-PCR, flow cytometry and MTT assay were employed to evaluate the effect of reversing drug resistance by salvicine. 选用沙尔威辛(SAL)进行逆转耐药实验,采用了RT-PCR法、流式细胞仪和MTT法,评估SAL逆转耐药的效果。
- METHODS:RT-PCR analysis was used to detect the expression level of KiSS-1 mRNA in human pancreatic cancer cell line AsPC-1 and tissues of 40 cases of pancreatic cancer, 27 cases of their metastastic site and 9 cases of normal pancreatic tissues. 方法:应用RT-PCR方法检测KiSS-1基因mRNA在人胰腺癌细胞株AsPC-1、40例胰腺癌及27例转移灶和9例正常胰腺组织中的表达情况。
- PACAP6 38 inhibited this effect of PACAP1 38. Conclusions: PACAP1 38 can stimulate the proliferation of human pancreatic cancer cell strain ASPC 1. The growth promoting effect of PACAP1 38 were mediated by the corresponding receptors. 结论:PACAP1-38促进ASPC-1的增殖;