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- The uptake of ~ 125I-UdR by HepG2 cells was higher than that of Na~ 125I. HepG2对12 5 I UdR的摄取明显高于Na12 5 I。
- The results indicated that CDW extracts could have obvious LPO effect on HepG2 cells in vitro. 结果表明氯化饮水有机提取物对体外培养的HepG2细胞具有明显的脂质过氧化作用。
- Furthermore, the induction of apoptosis by CSE in HepG2 cells was observed with AO/EB fluorescence stain. 0mg/mL赤参水提物处理HepG2细胞24h后,细胞荧光染色出现了明显的凋亡细胞;
- Objective To explore effects of polychlorinated biphenyl, PCB153 on DNA damage induced by benzo(a)pyrene (B[a]P) in HepG2 cells. 目的观察多氯联苯(PCBs)153和苯并[a]芘(B[a]P)单独及联合处理对人肝肿瘤细胞HepG2的DNA损伤作用。
- The proliferation caused by ALR had been obstructed by low Na~+, K~+-ATPase activity in HepG2 cell. 对于HepG2细胞，ALR促进其增殖与Na~+，K~+-ATPase的酶活密切相关，部分抑制Na~+，K~+-ATPase活性，能够减弱ALR促细胞增殖作用；
- Results The results of ELBA, IFA and Wetem-blot indicated that the HBc-Mep fusion protein, about 33kDa was expressed in transfected HepG2 cells. Western-hot结果显示表达产物约为33kDa,并能与抗preS2多抗特异性结合。
- Compounds 1 and 2 are new compounds,some compounds showed cytotoxic activities on SF-268,MCF-7 and HepG2 cell lines. 其中化合物1和2为新化合物,部分化合物显示一定的细胞毒活性。
- Conclusion Organic extracts of CDW from three raw water sources of C river, H river and D lake in Wuhan could induce DNA strand breaks in HepG2 cells. 最高浓度氯化饮水有机提取物诱导 Hep G2 DNA损伤的强度 ;H水 >D湖和 C江 (均 P<0 .;0 1)。
- PCB126 at certain concentrations showed no genotoxic effect in HepG2 cells, but it might enhance the genotoxic properties of B(a)P. PCB126在本试验条件下未显示出遗传毒性作用,但对B(a)P的遗传毒性作用具有一定的增强效应。
- Methods DNA damage was tested in HepG2 cells exposed to organic extracts from CDW by using single cell gel electrophoresis assay (SCGE). 方法 氯化饮水有机提取物染毒 Hep G2后 ,用单细胞凝胶电泳 (亦称彗星试验 )检测 DNA损伤。
- To study the effective medicine for anti-flavivirus, different concentrations of suramin were added to HepG2 cells after viral absorption. 寻找有效的抗黄病毒类药物,为临床应用提供理论依据。
- RT-PCR showed that hepG2 cells were positive for B7-1 at the messenger RNA(mRNA)level. The hepG2/Hb7-1 cell clone strongly expressing B7-1(positive rate:92.2%)was picked out. 而HepG2 /hB7 1的B7 1分子阳性表达率达到 92 6%25 ,转染B7 1基因后较转染前增大了 2 8倍多。
- It was suggested that the enhancement of DNA damage induced by B[a]P could be associated with the increases of CYP1A1 and CYP2B1 activities induced by PCB153 in HepG2 cells. PCB153在一定浓度范围内使B[a]P诱导的HepG2细胞DNA损伤作用显著增强,可能与其诱导的CYP1A1、CYP2B1酶活性升高有关。
- The resistance of HepG2 cells to 5-Fu was gradually enhanced and the apoptosis peak was delayed as the exposed time extended under the surroundings of low glucose. 在低糖环境下生长时间越长的HepG2细胞对5-Fu的抵抗越强,而且随着低糖培养时间的增加,5-Fu诱导的HepG2细胞的凋亡高峰延迟。
- Based on these points, mis study aims to clarify the significance of NHE-1 gene in regulation of the proliferation/apoptosis of HepG2 cell lines. 本文就着眼于此，探索NHE-1基因在肝癌HepG2细胞增殖与凋亡调控中的意义，以期为肝癌基因治疗提供新靶点。
- The inhibition effects of ~ 125I-UdR on HepG2 cell growth were obvious. ~ 125I-UdR may be incorporated inside the HepG2 cells and the uptake of HepG2 was dose dependent. 12 5 I UdR对HepG2细胞生长有明显抑制作用 ,HepG2细胞摄取12 5 I UdR量有浓度依赖性。
- The anti-NKG2D pAb could obviously inhibit the cytotoxicity of NK cells towards K562 and HepG2 cells and decrease their cytotoxicity to both target cells for 6.7 and 4.1 times, respectively. 抗NKG2DpAb能显著抑制NK细胞对K5 6 2、HepG2细胞的细胞毒效应 ;使NK细胞对两种靶细胞的细胞毒效应分别下降了 6 .;7和 4
- Objective To study DNA damage in HepG2 cells exposed to organic extracts from chlorinated drinking water (CDW) from three raw water sources of C river, H river and D lake in Wuhan. 目的 研究武汉市主要水源 C江、D湖、H水的氯化饮水有机提取物对 Hep G2 DNA的损伤。
- The two recombinant plasmids were transfected into the HepG2 cells , then the inhibition effect on HBV replication by the genome of the HBV defective virus was studyed by ELISA dot hybridization and semiquantitative PCR. 构建含乙肝缺陷病毒基因组的重组逆转录病毒载体和含乙肝病毒基因组的重组逆转录病毒载体，共转染HepG2细胞，ELISA、PCR方法和斑点杂交初步研究乙肝缺陷病毒基因组对HBV复制的影响。
- In conclusion, Immunocal seemed to enhance the cytotoxicity of baicalein by inducing more apoptosis;this increase in apoptotic cells may be associated with the depletion of GSH in HepG2 cells. 小结：怡妙康能增强黄岑黄素的细胞毒性效果，并促进细胞凋亡，肿瘤细胞的凋亡与细胞内谷胱甘肽降低有关。
