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- A549细胞表面Fas受体表达相对较多; 细胞凋亡率30.45%(P<0.02)。Conclusion FNQ3 can increase the cell surface Fas receptor expression and up-regulate the sensitivity of Fas-mediated apoptosis in a time- and dose-dependent manner.
- 目的探讨2-甲基萘[2,3-b]呋喃-4,9-酮(FNQ3)对A549细胞表面Fas受体表达和细胞凋亡的调节效应。Objective To study the effect of 2-Methylnaphtho[2,3-b]furan-4,9-dione(FNQ3) on Fas receptor expression and apoptosis in A549 cells.
- Fas受体Fas
- 可溶性Fas受体soluble Fas
- Fc特异性结合 ,呈浓度剂量依赖关系。 细胞ELISA实验证实 3A8能与Jurkat细胞表面Fas受体结合。Fc in a dose-dependent manner, and the binding of the 3A8 peptide to the Fas receptor on the surface of Jurkat cells was inhibited the proliferation of Jurkat cell.
- 2-甲基萘[2,3-b]呋喃-4,9-酮(FNQ3)对肺腺癌细胞株(A549)细胞Fas受体表达和细胞凋亡的影响Effect of FNQ3 on Fas Receptor Expression and Apoptosis in Lung Adenocarcinoma Cell A549
- 受体2AT2
- SLE患者T淋巴细胞表面Fas受体分子数与SLE的活动性呈正相关,是一较好的评价SLE疾病活动性的指标。Increased expression of Fas receptor molecules on CD3+ T lymphocytes accelerates the apoptosis of lymphocytes and stimulates the production of antibodies. It is an indicator for disease activity evaluation in SLE patients.
- 受体Arecep-tor A
- Fas基因Fas gene
- A549细胞表面Fas受体表达和A549细胞FasmRNA及细胞凋亡敏感度都随着药物浓度和投药时间的增加而增加。FNQ3 increased Fas mRNA product,the expression of Fas receptor and apoptosis in a dose- and time-dependent manner.
- Fas抗原Fas antigen
- VIP受体VIP receptor
- APJ受体APJ receptor
- Fas系统Fas system
- 目的通过定量流式细胞术检测系统性红斑狼疮(SLE)患者T淋巴细胞表面Fas受体的分子数,探讨SLE的发病机制及与疾病活动指数的相关性。Objective To study the expression of surface Fas receptor molecules on CD3+ T lymphocytes, and to investigate the relationship with pathogenesis and activity of systemic lupus erythematosus (SLE).
- CSa受体C5a receptor
- Fas/FasLFas/FasL
- Fc受体1CD89
- 可溶性FasSoluble Fas