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- The biphasic decline of viral replication on treatment reflects these two modes of action. 治疗过程中病毒复制的双相下降反映了这两种作用模式。
- By doing this, our body couldn't function well due > to the massive viral replication. 如果病毒大量制自己，则我们的身体就不能正常工作了。
- By doing this, our body couldn't unction well due to the massive viral replication. 于是，我们身体当中的免疫细胞就会很快的杀掉这些感染病毒。
- Thus we use all antiviral agents to suppress viral replication and HBIG to bind down the residual virus, and this combination works very well. 所以我们使用抗病毒药物来抑制病毒的活性，然后用乙肝免疫球蛋白（HBIG）来结合残留的病毒，这种办法非常有效。
- HIV protease is the essential enzyme for viral replication,and one of important targets in looking for anti-AIDS drugs design and research. HIV蛋白酶是病毒复制的必需酶,是寻找治疗获得性免疫缺陷病药物的重要靶点之一。
- Exploiting the activation of the DNA damage pathway by viral replication for the generation of antiviral drugs needs to be examined. 利用病毒复制激活DNA修复途径而制成的抗病毒药物需要进一步检测。
- If viral replication required that the whole virus enter the cell, both P32 and S35 tracers would be located in the bacterial cytoplasm. 如果病毒的复制需要整个病毒进入细菌，那P32和S35示踪原子都会聚积在细菌的细胞质中。
- So overwhelming was the excitement that overtook us when substances were found to potently inhibit the protease enzyme, thereby blocking viral replication in the test tube. 我们发现了一种物质，有可能抑制蛋白酶，从而阻止病毒在试管中繁殖，那时我们真是兴奋得难以自已。
- They applied the concentrated protein topically on the cornea, and found that the inflammation and viral replication were significantly suppressed, too. 他们采用浓缩蛋白滴眼液对角膜发现炎症反应和病毒复制显着压抑,太。
- Hepsera slows the progression of chronic hepatitis B by interfering with viral replication and causing DNA chain termination after its incorporation into viral DNA. 通过干扰病毒的复制来减缓慢性乙肝的病变进程，和通过与病毒DNA整合来终止DNA分子链。
- So analogs, including nucleoside and nucleotide analogs are now potent drugs, which suppress complicated viral replication of HVC, I will say in 70% to 90% of the patients. 因此，现在核苷类药物是强效药物，能抑制复杂的丙肝病毒复制，我认为能抑制70%25到90%25的患者。
- Therefore, when you give ribavirin to the patients in addition to the interferon, it makes it easier because the viral replication is already inhibited. 因此，如果除了干扰素以外还加用利巴韦林，治疗会更有效，因为病毒复制已经被抑制了。
- Experiments have already established that blocking viral replication with antiretroviral medication attenuates immune activation and boosts the number of CD4+ T cells. 科学家目前已进行几个实验，包括运用抗反转录病毒药物阻断病毒复制，用以降低免疫系统的活化程度并且提高CD4+T细胞的数量。
- Therefore， prolonged， adequate suppression of viral replication should be the practical goal for the treatment of CHB disease in the Asian population. 因此，对于亚洲人群中的CHB疾病，对病毒复制进行持续的、足够的抑制应成为临床治疗目标。
- The next step would be immunosuppressive therapy, which could slow down viral replication by limiting the T cell activation and prevent the CD4+ T cells from committing suicide. 接下来，就是采用免疫抑制疗法，藉由限制T细胞的活化来减少病毒的复制，并阻止CD4+T细胞的凋亡。
- Lamivudine, and most nucleoside analogues, acts by suppression of viral replication through competitive inhibition of reverse transcriptase and DNA polymerase. 拉米还有大多数的核苷类似物，通过竞争性地阻断翻转录酶和DNA聚合酶来抑制病毒复制。
- These data suggest that proanthocyanidin isolated from blueberry leaves may have potential usefulness as an anti-HCV compound by inhibiting viral replication. 上述研究显示，蓝莓叶原花青素可抑制HCV的复制，在抗HCV方面有着潜在的用途。
- Any of a group of glycoproteins produced by cells in response to infection by a virus that act to prevent viral replication and have the ability to induce resistance to viral antigens. 干扰素细胞由于对病毒感染反应而生成的一种糖原蛋白，它能防止病毒复制并能对病毒抗原产生抵抗能力
- The IFN and inflammatory response against viral infections is mediated by cellular pattern-recognition receptors(PRR) that recognize specific molecular structures on viral particle or products of viral replication. 细胞模式识别受体(PRR)能特异性识别病毒分子或其复制产物而产生IFN与炎性细胞因子。
- Over the last half of century interferon (IFN), originally discovered as an antiviral protein, has developed from an inhibitor of viral replication to a major force in the antiviral response. 在最后世纪干扰素的一半(IFN)， 最初作为抗病毒的蛋白质发现，在抗病毒的回应里已经从病毒的复制的抑制剂到一种主要的力量发展。
