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- osteosarcoma cell subline 骨肉瘤细胞株
- Objective To study the influence of CXCR4 expression on the exosomatic invasive capability of osteosarcomatous cell subline. 目的 探讨趋化因子受体CXCR4对骨肉瘤细胞株的体外侵袭能力的影响。
- Objective: To investigate the effect of cinnamic acid (CINN) on differentiation induction of a human lung adenocarcinoma cell subline (A549). 目的：研究肉桂酸对人肺腺癌细胞的诱导分化作用。
- CONCLUSIONS: Kappa-Selenocarrageenan can induce apoptosis of human osteosarcoma cell in vitro. 结论:硒酸酯多糖可在体外诱导人骨肉瘤细胞凋亡。
- Objective To study the expression of BMP(bone morphogenetic protein)-2,-3,-6,and-12 in osteosarcoma cell lines. 目的研究骨形态发生蛋白(bone morphogenetic protein,BMP)-2、-3、-6和-12在骨肉瘤细胞株中的表达,为下一步研究BMPs在骨肉瘤发生发展中的作用奠定基础。
- Objective: To study the effect of melittin on apoptsis and necrosis of osteosarcoma cell Tine U2OS in vitro. 目的：研究蜂毒素是否能够诱导骨肉瘤细胞U2OS凋亡与坏死。
- Methods RNA was isolated from the human osteosarcoma cell line U-20S by single-step method of acid guanidinium isothiocyanate phenol-chloroform extraction. 方法 采用异硫氰酸胍一步法从人骨肉瘤细胞株U-2OS 中提取总RNA，以逆转录一聚合酶链反应（Reverse Transcription coupled Polymerase Chain Reaction，RT-PCR）扩增cDNA基因片段。
- These findings suggest that K562 cell line has a capability of inducing acquired resistance to VER,which may be associated with overexpression of PgP,and then leading to reduction of intracellular accumulation of VER in K562/VER cell subline. 说明K562细胞系能产生对VER的获得性耐药性，其耐药机制可能与PgP表达过量，并由此导致细胞内VER聚集下降等因素有关。
- Methods A DDP-resistant human ovarian adenocar- cinoma cell subline (COC1 /DDP ) was developed by contineous stepwise selection in increasing concentra- tion of DDP from the parent cell line COC1 in vitro. The multiple changes of biological. 方法应用顺铂连续作用并逐步提高药量的递增压力选择法，从人卵巢腺癌细胞COC1中分离出对顺铂耐药的细胞亚株（COC1/DDP），并比较耐药细胞和亲代细胞某些生物学特性差异。
- To understand whether K562 cell was able to produce the acquired resistance to verapamil(VER),a model of K562 cell subline resistance to VER(K562/VER) was established through gradually increasing VER concentration in the media. 为探讨人白血病细胞对耐药逆转维拉帕米的耐药机制，我们通过不断提高培养液中维拉帕米浓度建立维拉帕米耐药性K562亚系（K562/VER），采用MTT法测定维拉帕米、潘生丁和环孢素A对K562、K562/VER细胞的毒性或耐药性；
- Chemically synthesized short survivin-siRNA can effectively inhibit survivin expression and cell proliferation in osteosarcoma cell line MG-63.Short survivin-siRNA may be a useful therapeutic strategy for cancer through the regulation of gene expression. 化学合成的survivin-siRNA能有效抑制survivin基因在骨肉瘤MG-63细胞中的表达,并对骨肉瘤MG-63细胞增殖有抑制作用。
- Objective To study the relationship between bone morphogenetic protein(BMP) expression and proliferating activity of osteosarcoma and detect the effect of BMP antibody on osteosarcoma cell. 目的 研究骨形成蛋白 ( bone morphogenetic protein,BMP)在骨肉瘤中的表达及其与肿瘤增殖活性的关系 ,并观察 BMP单抗体外对骨肉瘤细胞作用 ,为骨肉瘤的单抗治疗打下基础。
- MATERIALS: Human osteosarcoma cell line (OS-9901) was graciously provided by Professor Fan, Institute of Osteoscarcoma, Tangdu Hospital of Fourth Medical University of Chinese PLA. 材料：人骨肉瘤细胞系OS-9901由解放军第四军医大学唐都医院全军骨肿瘤研究所范清宇教授惠赠。
- Objective: To study the proliferation inhibition effect of docetaxel combined with cisplatin on osteosarcoma cells. 摘要目的：研究多西紫杉醇联合顺铂对人骨肉瘤细胞的增殖抑制作用。
- Objective To study the killing effect of Zijin powder extract on human osteosarcoma cells. 目的探讨中药紫金散对人骨肉瘤细胞的杀伤作用。
- drug resistant cell subline of gastric cancer 胃癌耐药细胞亚系
- Human Osteosarcoma Cells Alter Cytoskeletal and Adhesion Proteins in Response to Physiological Levels of Hydrostatic Pressure. 人类骨肉瘤细胞可改变细胞骨架、及粘连蛋白质对静水压力的反应。
- Conclusion: Combination of docetaxel and cisplatin have more significant effect of the proliferation inhibition on osteosarcoma cells than either of the drugs alone has. 结论：多西紫杉醇和顺铂联合用药比单独用药对骨肉瘤细胞株增殖抑制作用更强。
- Objective:To study the ultrastructure changes of osteosarcoma cells(OS 732) inactivated by alcohol of two different concentrations and clinical application value. 目的:探讨两种不同浓度的酒精灭活骨肉瘤细胞(OS-732)后细胞内部结构变化和临床应用价值。
- Flow cytometry was used to detect theexpression of HLA-l, HLA-2, B7-l, B7-2 in parent cell lines (COC1 andSKOV3 ) and multidrug resistant cell sublines (COCI/DDP andSKOV3/Adr). 运用流式细胞仪技术检测了各种不同细胞亚株的HLA-1、HLA-2、B7-1、B7-2的表达。 结果：耐药亚株COC1/DDP、SKOV_3/Adr和亲本株COC1、SKOV_3的N-GSLs表达有差异，COC1/DDP、SKOV_3/Adr的CMH表达较COC1、SKOV_3强。
