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- MIS(myocardial infarct scintigram) 心肌醒塞闪烁图
- myocardial infarct scintigram 心肌梗塞闪烁图
- Results: MDP could reduce the myocardial ischemic limit and myocardial infarct area. 结果:米槁心乐滴丸具有明显减轻心肌缺血程度及缺血范围,减少梗塞区面积。
- Conclusion:The injury of myocardial ultrastructure and myocardial infarct size of PP、DP group may be decreased by PP. 结论：超速起搏预适应(早期及延迟相)均明显减少在体兔缺血再灌注时的心肌细胞超微结构损伤和心梗范围。
- Hindman N B, Schocken D D, Windmann M, et al. Evaluation of a QRS scoring system for estimating myocardial infarct size[J]. Am J Cardiol,1985,55:1485. 中华心血管病杂志编委会.;急性心肌梗死溶栓疗法参考方案[J]
- Methods 100 ACS patients were divided into unstable angina pectoris(UAP) group(n=64),acute myocardial infarct (AMI) group(n=36)and 50 healthy cases as control group. 方法ACS患者100例分为不稳定型心绞痛(UAP)组64例和急性心肌梗死(AMI)组36例,正常对照组50例。
- Methods : By LAD for rat to have myocardidl ischemia,the changes of myocardial infarct size,the serum creatine phosphokinase(CK),actate dehydrogenase(LDH). 方法通过大鼠结扎左冠状动脉前降支(LAD)产生急性心肌梗死模型,观察淫羊藿总苷对实验性心肌梗死的影响。
- Methods: By LAD for rat to have myocardidl ischemia, the changes of myocardial infarct size, the serum creatine phosphokinase (CK), actate dehydrogenase (LDH). 方法通过大鼠结扎左冠状动脉前降支(LAD)产生急性心肌梗死模型，观察淫羊藿总苷对实验性心肌梗死的影响。
- Methods To study the effects of electrocardiograph (ECG), myocardial enzymes and myocardial infarct scope in experimental rats by ligating the coronary arteries. 方法通过大鼠冠脉结扎致心肌缺血模型,观察通心活脉灵对大鼠心电图、心肌酶及梗死面积的影响。
- Objective:Our purpose was to study the influence of KATP channel opener KRN2391(KRN) on cardiac function and myocardial infarct size of isolated rat heart. 目的:探讨ATP敏感性钾通道(KATP通道)开放剂KRN2391(KRN)对离体大鼠心功能及心肌梗死范围的影响。
- Intravascular laser therapy of acute myocardial infarction. 急性心肌梗塞的血管内激光治疗。
- How do you distinguish angina from MI (myocardial infarction)? 心绞痛和心肌梗死有什么区别?
- Myocardial infarction size was determined by TTC stain. 氯化三苯基四氮唑(TTC)染色法评价心肌梗死范围;
- Now we have less angina pectoris or myocardial infarction. 现在我们越来越少人有心绞痛或是心肌梗塞。
- The most likely diagnosis is acute myocardial infarction. 可能性最大的诊断是急性心肌梗死。
- Conclusions Ischemia preadaptation can protect myocardium injury in ischemia reperfusion, the protection is mainly reflected by reduction of myocardium enzyme activity,myocardium infarct area and subcellular structure injury. 结论缺血预适应可以降低心肌酶活性,缩小梗死面积,改善亚细胞结构损害,对心肌缺血再灌注损伤有保护作用。
- Compared with I/R,the myocardial swelling,interstitial hemorrhage and inflammatory cell infiltrate were decreased,the myocardial infarct size was reduced(P<0.01) after CIP and RIP. 与I/R组比较,CIP和RIP均能明显改善心肌损害的形态学改变,降低心肌细胞的肿胀、减少间质出血和炎性细胞的浸润;
- RessultS:The incidence and duration of I-R-induced arrhythmias(VT,Vf) and the severity of myocardial injuries(S-T segment elevation,values of myocardial enzymes) and myocardial infarct size were all obvious lower in group C than in group control,(P<0.01). C组在缺血/再灌注引起的心律失常(VT、Vf)的发生率及持续时间、心肌损伤的程度(ST段抬高值、心肌酶值)及心肌梗死的面积与对照组比较都有明显降低(P<0.;01)。
- Intravenous recombinant streptokinase on acute myocardial infarction. 重组链激酶治疗急性心肌梗塞的疗效与安全性。
- The extent of neutrophilic infiltrates, along with karyolysis and loss of cytoplasmic cross striations, is typical for an acute myocardial infarct of 1 to 4 days in duration from onset. 中性粒细胞广泛浸润，核溶解、胞浆减少，是典型的急性心肌梗死的1-4天的表现。
