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- The myocardial cell is enclosed by the sarcolemma. 心肌细胞由肌膜包裹。
- Fig.9 Myocardium cell of Procambarus clarkii.Showing striation of myofibril. 图9螯虾心肌细胞示肌原纤维的横纹。
- To speculate early if there is myocardial cell damagein infant pneumonia, on which therapeutics and prognostic base. 目的早期预测婴幼儿肺炎是否有心肌细胞损害,为治疗及预后的判断提供依据。
- The changes are consistent with those of myocardial cell AP of Se-Iow, rats in situ. 这一结果与低硒大鼠在体心肌细胞动作电位的改变完全一致。
- Conclusion Both HSP70 and JNK inhibitor can delay apoptosis induced by H2O2 and protect myocardial cell. 结果热休克预处理后HSP70在细胞浆内大量表达。
- For patients who are suspected of myocardial cell damage, how can we make a quick diagnose in order to give them an immediate and effective treatment? 对怀疑有心肌损伤的病人如何尽快确诊,以便快速有效地治疗?
- Objective The protective effects of shenfu injection on myocardial cell damagewere observed in patients undergoing open heart surgery. 目的观察参附注射液对体外循环下心内直视手术患者心肌损伤标志物的影响。
- Fig.2 Anoxia reperfusion group without the cerium ions:Myocardial cell appears edema, mitochondria swell, cristae vague. No cerium deposits are observed in the cell. 图2缺氧再灌注无铈组：心肌细胞水肿，线粒体肿胀，嵴模糊。细胞内未见铈沉积颗粒。
- Every protect rate of the myocardial cell of every medicine serum dissimilarity to infect with CVB_(3m) for the medicine time by MTT method. 应用MTT法检测各含药血清不同给药时间对CVB_(3m)感染的心肌细胞的保护率。
- Conclusion:Acupuncturing pericardium channel points can effectively protect myocardial cell by regulating the contenits of ATP,ADP and AMP. 结论:针刺手厥阴心包经穴可明显改变ATP、ADP和AMP含量,保护心肌的有氧代谢,降低氧自由基的产生,从而有效地保护心肌细胞。
- Fig.2 Anoxia reperfusion group without the cerium ions:Myocardial cell appears edema, mitochondria swell, cristae vague. 图2缺氧再灌注无铈组：心肌细胞水肿，线粒体肿胀，嵴模糊。
- This paper had summarized the intracellular change of myocardial cell, extra cellular matrix, neurotransmittor and activation of endothelium. 该文就心肌细胞内在改变、细胞外基质和神经递质及内皮细胞功能的激活等方面,进行较系统地综述。
- Conclusions:The myocardial cell apoptosis can be induced by ischemiareperfusion and be inhibited by Astragalus Membranaceus. 结论：缺血再灌注可引起心肌细胞的凋亡，黄芪确实可抑制凋亡的发生。
- It can activate sodium channel,inrease sodium current.it can inrease liber calcium concentration in the myocardial cell. 实验证明：As可激活钙通道，增加钙通道电流 As增加细胞内游离的钙离子浓度 As可激活钠通道，增加钠电流 关键词：龙牙葱木皂苷 培养心肌细胞 L-型钙电流 钠通道 膜片钳 正性肌力作用 Aralosides(As)are omni-glycosides extracted from roots;petiols and skins of Aralia (Miq) seem.
- Methods Cultured myocardial cell from neonate rats were divided in hypoxia group A （hypoxla-reoxygenation） and groups B（EPO）. 方法 培养新生大鼠心室肌细胞分成缺氧组和促红细胞生成素（EPO）组。
- Objective To observe the protection effect and its mechanisms of Salviae Miltiorrhizae Injection(SMT) on myocardium cell injury induced by adriamycin(ADR). 目的观察丹参注射液(Salviae Miltiorrhizae injections SMI)对阿霉素(Adriamycin,ADR)诱导大鼠心肌细胞损伤的保护作用并探讨其作用机制。
- The rate of apoptosis, the expression of fas and caspase-3 genes in the myocardial cell in group B were significantly higher than that in group A(P<0.01). 过度训练组大鼠心肌细胞凋亡率、fas和caspase-3基因表达水平均显著高于正常对照组(P<0.;01)。
- There were no significantly difference in the rate of apoptosis, the expression of fas and caspase-3 genes in myocardial cell in group C as compared with that in group A(P>0.05). 而过度训练+牛磺酸组心肌细胞凋亡率、fas和cas-pase-3基因表达与正常对照组无显著差异(P>0.;05)。
- Conclusions Ultrasound-mediated microbubble destruction could enhance the permeability of myocardial cell membrane.This might be one mechanism for gene delivery with microbubbles. 结论超声破坏微泡可提高心肌细胞膜通透性，可能是超声微泡造影剂增强组织中基因转染的机制之一。
- The ultrastructure of myocardium cell was slightly destroyed and the infarct size was significantly smaller in the cap-feed group than the normally feed rabbits and CF rabbits. 卡托普利加胆固醇组心肌细胞超微结构结构破坏及心肌梗死面积明显小于对照组和胆固醇组。
