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- Chan ges of bcl-xl protein expression were observed at 24 hours after global brain ischemia, and apoptosis were checke d at 72 hours in hippocampal CA1 subregion. 观察缺血后 2 4h海马CA1区bcl -xl蛋白表达和缺血后 72h细胞凋亡。
- Changes of cytochrome C in hippocamp CA1 subregion were observed 24 hours after global brain ischemia and rat's learning and memory abilities were examined with Y maze and elected voltage 40V at 4 weeks after the model operation. 观察缺血后24h海马CA1区细胞色素C(CytC)变化,缺血后4周利用Y型电迷宫进行大鼠学习和记忆能力测试,选用电压40V。
- Cerebral ischemic control group: Gerbils were established into models of cerebral ischemia by occluding bilateral CCA, and reperfused for 72 hours following 5-minute global brain ischemia (GBI). 脑缺血对照组:夹闭沙土鼠双侧颈总动脉造成脑缺血模型。 全脑缺血5min后再灌注72h。
- Periodic quantity variation of proliferating neuronal progenitors in adult rats after global brain ischemia 成年大鼠全脑缺血性脑损伤后神经前体细胞的增殖周期变化
- Keywords Intervention;Controllable;Carotid artery stenosis;Global brain ischemia（GBI）;Experimental model; 介入;可控性;颈动脉狭窄;全脑缺血;实验模型;
- Global brain ischemia (GBI) 全脑缺血
- Methods Focal brain ischemia animal model was used. 方法应用局限性脑缺血动物模型。
- global brain ischemia 全脑缺血
- The abnormal DA release inhibited by L-NNA during global ischemia may be one of the protective mechanism of this agent on brain ischemia. L-NNA抑制半球缺血时DA的不正常释放可能是其保护缺血大脑的机制之一。
- CDP-choline improved the recoveryof SEPs caused by brain ischemia obviously. CDP-choline可明显促进缺血脑功能的恢复。
- But the detailed mechanisms of G-LOC or G induced brain ischemia as well as protective effect IPC were not clear. 然而不论大G昏迷、G力造成的脑损伤及缺血前制约效应的保护机转都并不清楚。
- It can cause severe complications, such as brain ischemia or cervical hematorrhea incurred by rupture of aneurysm. 它可以引起严重的并发症，比如大脑局部缺血或动脉瘤破裂产生的颈部软组织出血。
- It was suggested that DDPH had some protective effect on acute brain ischemia and ischemia-reperfusion. 结果提示，DDPH对动物脑缺血具有一定的保护作用。
- Global brain refers to a concept relating to shared human knowledge; the sum total of all thoughts, creations, and inventions of the human mind. 全球脑是指这样一个概念，它关于分享人类知识，所有人类智力的思想、创新和发明的总和。
- Objective To observe surviving, migrating and differentiating of mice neural stem cells in global brain ischemical reperfusion injury models of cat. 目的观察小鼠神经干细胞在猫全脑缺血再灌注损伤模型中的存活、迁移及分化。
- Objective To study the improvement effects of GM_1 on brain ischemia reperfusion(BIR) induced memory impairment in mice. 目的探讨脑缺血复灌后连续应用GM1对改善记忆障碍的疗效。
- Disturbances of calcium homeostasis and modification of ca~(2+) pump(Ca~(22+),Mg~(2+)-ATPase)in the rabbit focal brain ischemia. 脑缺血时钙稳态与钙泵(Ca~(2+),Mg~(2+)-ATP酶)活性变化
- Without obvious influence in brain metabolic functions,L1 KO mice is an useful animal model for investigation of the role of L1 in brain ischemia. L1CAM基因缺失对小鼠脑局部代谢功能无明显影响,提示L1 KO小鼠可作为研究L1CAM在脑缺血中作用的动物模型。
- Keywords The kidney injury after brain ischemia reperfusion was correlated with the dysequilibrium between ET and CGRP in which ET was dominant. 脑缺血/中医疗法;益元活血丹/治疗应用;大黄/治疗应用;血栓心脉宁/治疗应用;再灌注损伤;肾疾病/中医药疗法;肾疾病/病理学;肾疾病/超微结构;内皮缩血管肽类/代谢;
- When the collateral circulation is good, the radialis velocity may be normal, Whereas, the risk of brain ischemia would be increase. 反之可能会诱发脑缺血的临床症状。
