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- experimental ischemic brain 实验性脑缺血
- The study summarized the recent advances of acupuncture on the treatment of experimental ischemic cerebral in the changes of brain morphosis, blood rheology, brain microcirculation and so on. 本文从针刺对实验性脑缺血在脑组织形态学改变、血液流变学、脑微循环等方面的研究进展作一综述。
- Conclusion FG can reduce ischemic brain damage and protect cerebral tissue. 结论甘草总黄酮能减轻缺血性脑损害,保护脑组织。
- AIM: To investigate the mechanism for electroacupuncture in improving the plasticity of synapse after ischemic brain injury. 目的:探讨针刺对缺血性脑损伤后的突触可塑性促进作用的机制。
- Cerebral ischemia may induce upregulation of ANG and GFAP expression in the ischemic brain areas. 脑缺血可诱导缺血脑区ANG和GFAP的表达上调。
- To study the effects of glutathione on hypoxic ischemic brain damage in neonatal rats hippocampal. 研究谷胱甘肽对缺氧缺血脑损伤引起的神经细胞凋亡的作用。
- Gabryel B,Trzeciak HI.Role of astrocytes in pathogenesis of ischemic brain injury[J].Neurotox Res,2001 ;3(2):205-21. 周琴;廖维靖;杨万同;等.;阿魏酸钠促进局灶性脑缺血再灌注后神经功能恢复和血管生成作用的研究[J]
- Cerebral ischemia activates endogenous angiogenic factors,and increase microvessel density and form new capillaries in ischemic brain tissues. 脑缺血能激活内源性血管生成因子,使缺血脑组织血管密度增高,新生毛细血管形成。
- On the other hand, cycloheximide could reduce the expression of P 53 and P 21 proteins in ischemic brain tissue also protect CA1 region from DND. 另外,放线菌酮可抑制脑组织P53、P21蛋白的表达,并对DND具有一定的保护作用。
- Objective To study the effect and possible mechanism of He Ne laser acupoint irradiation treatment on hypoxic ischemic brain damage (HIBD). 目的探讨氦氖激光穴位照射对新生鼠缺血缺氧性脑损伤的作用及其可能机制。
- The mechanism of ischemic brain injury has not been elucidated thoroughly now,and there is no effective therapy in clinic. 缺血性脑损伤的发生机制目前尚未明确,临床缺乏有效的治疗手段。
- Decrease of NO in serum and increase of that in brain tissue may lead to cerebral vasospasm and ischemic brain damage respectively after SAH. 血清NO减少、脑组织NO增加分别在脑血管痉挛发生及加重脑缺血损害中起重要作用。
- Hypothermia is important to our healthy and disease.Such as: hypothermia lessens ischemic brain injury whereas hyperthermia exacerbates it. 低体温与人体疾并健康密切相关,如在许多缺血性疾病时,降低体温可降低机体代谢从而减少缺血性的损害,反之,升高体温使病情恶化。
- Aim: To observe the effects of lidocaine and thiopental on the neuronal injury induced by the experimental ischemia in hippocampus slice cultures obtained from postnatal 22 days SD rats. 目的:观察利多卡因和硫喷妥钠对生后22d大鼠培养海马脑片的实验型缺血后神经元损伤的影响。
- Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。
- Micro thrombosis and cerebral hemorrhage could be found after hypoxic ischemic brain damage.A series of neuron changes such as neuron edema,neuron loss,and gliosis happen... 结果缺氧缺血后即刻脑皮质、髓质、海马微血管数增多,微血管通透性增强,微血管内血栓形成,脑内多部位出血,同时神经元出现水肿、丢失、胶质细胞增生等一系列缺血缺氧改变。
- The mechanisms contributed to ischemic brain injury involve excitotoxicity,calcium overload,free radical reaction,depolarization surrounding infarct,inflammation and apoptosis. 兴奋毒性、钙超载、自由基反应、梗死周围除极、炎症和凋亡是造成缺血性脑损伤的中心环节。
- Objective: To study the changes of cortes distribution of calcium and somatostain content in dementia rat after iterative ischemia brain damage. 目的 :探讨反复脑缺血后痴呆大鼠皮质内钙分布及生长抑素含量的变化。
- Abstract: Objective: To study the changes of cortes distribution of calcium and somatostain content in dementia rat after iterative ischemia brain damage. 摘 要: 目的:探讨反复脑缺血后痴呆大鼠皮质内钙分布及生长抑素含量的变化。