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- chronic hypoxic hypercapnia 慢性低O2高CO2
- Effects of L-arginine on pulmonary circulation andcerebral blood flow in acute and chronic hypoxic rats. L－精氨酸对急慢性缺氧大鼠肺循环和脑血流的影响
- Abstract AIM： The influence of cilazapril and taurine on apopto sis of chronic hypoxic lung tissues was observed. 摘 要 目的： 初步观察西拉普利和牛磺酸对慢性缺氧肺 组织细胞凋亡的影响。
- Both acute or chronic hypoxia can cause vasoconstriction and vasoconstriction increase . 急性或慢性的低氧都能使肺血管收缩，血流阻力增大。
- Objective: To explore the effect of chronic hypoxia on the proliferation and dedifferentiation of LLC-PK1 cells. 目的：研究低氧对近端肾小管细胞株(LLC-PK1)细胞增殖和去分化的作用。
- A gloss of chronic hypoxia in normal and diseased individuals at high altitude Zubieta-Castillo G. Zubieta-Calleja G.R. Zubieta-Calleja L. 试论高原地区正常及患病个体的慢性缺氧。
- Objective To observe the effects of adrenocorticotropin(ACTH) and testosterone(T) on erythropoiesis in mice with chronic hypoxia. 目的 观察促肾上腺皮质激素 (ACTH)和睾酮 (T)对低氧小鼠红细胞生成的影响。
- The ratio of ATP to total adenylate pool in acute and chronic hypoxia groups was respectively 41 5% and 42 5% of that in the normoxic control group. ATP在总腺苷酸库中所占比例在急、慢性缺氧时分别是对照的 41 5%25和 42 5%25;
- This article reviews the progress on the relationship between chronic hypoxia with cerebral hypoperfusion and presenile dementia mechanism. 本文就慢性缺氧/大脑低灌注与早老性痴呆的发病机制相关研究的进展作一综述。
- Conclusion: Chronic hypoxia induces both proliferation and dedifferentiation of LLC-PK1 cells mediated, in part, by the sustained activation of PKC. 结论：慢性低氧可诱导LLC-PK1细胞的增殖和去分化，这些作用部分由PKC持续激活介导。
- But in chronic hypoxic exposure, the inhibition of both RNA synthesis and protein synthesis was alleviated, being 72% and 76% of the normoxic control, respectively. 慢性缺氧暴露后线粒体RNA和蛋白质合成活性分别为对照的72%25和76%25;
- Methods: 40 chronic hypoxia mice models wers established and devided randomly into 3 groups: puerarin group, anoxia group and the control group. 方法建立小鼠慢性缺氧模型30只，随机分为葛根素治疗组、缺氧组各15只，另设正常对照组10只。
- Aim: To study the effect of chimonin on chronic hypoxia and hypercapnic pulmonary hypertention and to explore its mechanism. 目的:研究知母宁对慢性低氧高二氧化碳性肺动脉高压的抑制作用及其作用机制。
- For example, during chronic hypoxia the bone marrow produces more erythrocytes to carry and release more oxygen to the tissues. 例如，在慢性缺氧过程中，骨髓产生更多的红细胞以携带和释放更多的氧气给组织。
- As regards the pathogenesis, the auther believes that myocardial damage dueto chronic hypoxia should be stressed. Early diagnosis and treatment are important. 因此,慢性缺氧对心肌的损害值得重视,临床早诊断早治疗尤为重要。
- Besides, some studies also showed that the NHE-1 specific inhibitors suppressed the PASMCs proliferation and reduced pulmonary vascular remodeling induced by chronic hypoxia. NHE-1特异性抑制剂可显著抑制PASMCs增殖，减轻肺血管结构重建。
- This study suggests that plasma NSE activity may reflect the degree of brain damage by chronic hypoxia and remain of CO2 in the patients with chronic pulmonary heart disease. 提示血清NSE活性可以反映慢性肺心病患者缺氧、二氧化碳潴留引起的脑损害程度。
- These results suggest that PDGF BB polypaptide may stimulate PDGF mRNA expression and promote the secretion in PASMC, and PDGF may have an important role in the proliferation of PASMC seen in the chronic hypoxic pulmonary hypertension. 本研究结果提示，PDGF?BB多肽能刺激肺动脉平滑肌细胞PDGFmRNA表达，促进其分泌，在慢性低氧性肺动脉高压血管平滑肌细胞增殖中具有重要作用
- The results were as follows:1.The mean pulmonary arte-rial pressure(P pa)and pulmonary vascular resistance(PVR)were signiflcantly decreased by in-haled NO in chronic hypoxic rats(P<0.01),but these changes were not observed in normoxiccontrol rats. 结果表明：（1）常氧吸入NO时能明显降低慢性缺氧大鼠肺动脉平均压（Ppa）和肺血管阻力（PVR），但对正常大鼠的Ppa和PVR无明显影响；
- Acute and chronic hypoxia might lead to different biology within the tumour and this might have a direct effect on the design of new therapies for the treatment of hypoxic tumours. 急性和慢性乏氧可能在肿瘤中导致不同的生物学效应，并且其可能对于乏氧肿瘤治疗的新疗法设计直接产生影响。
