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- Objective:To know about the present research on skeletal Muscular atrophy with denervation,and to explore its development. 目的:了解失神经支配骨骼肌萎缩的研究现状并对未来研究方向进行探讨。
- CONCLUSION: The transplantation of MSCs into denervated muscles can significantly retard denervated skeletal muscular atrophy, and this effect is only observed within 8 weeks after denervation. 结论：骨髓间充质干细胞移植到失神经骨骼肌内可明显延缓失神经支配骨骼肌的萎缩，并且这一作用只在细胞移植8周以内比较明显。
- Biological and histological changes of denervated skeletal muscular atrophy 失神经支配骨骼肌萎缩的生物学和组织学变化
- Skeletal muscular atrophy 骨骼肌萎缩
- Muscular atrophy of a person affected with paralysis. 因麻痹而导致人体肌肉萎缩
- Individual local myasthenia gravis patients, muscular atrophy. 个别患者出现局部肌无力、肌萎缩。
- The skeletal muscular system consists of the 206 bones and hundreds of muscles that protect and support the internal organs and help the body move. 骨骼肌肉系统由206块骨骼和数百块肌肉组成，它们保护、支持内部器官，帮助身体运动。
- Belonging to the forkhead/winged helix transcription factors family,FOXC2 is expressed in both adipose and skeletal muscular tissue. FOXC2属翼状螺旋／叉头转录因子家族,在脂肪组织及骨骼肌中均有表达。
- Calcium ion exists extensively in cells as the second messenger,and calcium channel blocker (CCB) is widely used to treat cardiac, skeletal muscular diseases. 钙离子作为细胞内第二信使,广泛存在于体细胞内,因而钙通道拮抗剂(CCB)被广泛应用于心脏、骨骼肌和神经元等研究及心血管疾病治疗。
- A gene implicated in Spinal Muscular Atrophy showed an association in three populations, but not in Yoruba from Ibadan, Nigeria. 一个涉及脊髓性肌萎缩病的基因显示出与三个人群有关联但却没有在伊巴丹、尼日利亚、的优鲁巴人身上发现。
- Clinical, electromyography (EMG) and nerve conduction velocity (NCV) features of 32 cases with peroneal muscular atrophy (PMA) are reported. 本文报告32例腓骨肌萎缩症(PMA)患者的临床及肌电图(EMG)和神经传导速度(NCV)的改变。
- With toe (HSI) A slow growth, thickening deformation, dry skin thinning, Ganmao exfoliated and muscular atrophy. 伴趾(指)甲生长缓慢，增厚变形，皮肤干燥变薄，汗毛脱落和肌肉萎缩。
- Two cases of shoulder petoneal muscular atrophy (SPMA),a rare type of charcot-Marie- Tooth disease,are reported. 本文报告两例进行性神经性肌萎缩症(Charcot-Marie-Tooth)的少见特殊类型-肩胛腓骨萎缩型,亦属慢性遗传性进行性周围神经病的变异型之一。
- Objective To study survival motor neuron (SMN) deletion in adult-onset patients with spinal muscular atrophy (SMA). 目的探讨成年起病的脊肌萎缩症(SMA)患者的运动神经元存活基因SMN的缺失情况。
- Objective To establish a gene diagnosis assay for spinal muscular atrophy(SMA) in children. 目的探讨儿童脊髓性肌萎缩症(SMA)的特异性基因诊断方法。
- Objective: To establish a genetic diagnosis assay for spinal muscular atrophy (SMA) in children. 摘要目的：建立儿童型脊髓性肌萎缩症(SMA)的特异性基因诊断平台。
- The double lower limb cannot walk, has not seen the ischemic muscular atrophy and the myodynamia is not normal. 双下肢不能行走，未见肌肉萎缩和肌力不正常。
- Moreover, when the E6 DRG cells co cultured with skeletal muscular tissues for 12 dyas, some of the DRG cells were immunostained by GABA antiserum, and the terminal fibers of sensory neurons were observed connecting with muscular cells. 然而，当同样胚龄的DRG细胞与骨骼肌细胞共同培养12d后，有部分DRG细胞呈GABA阳性反应，其神经突起与骨骼肌细胞相连。
- Inspect the patient's hands and wrists, noting any swelling, redness, nodules, deformities, or muscular atrophy. 仔细观察病人的手和手腕，注意有没有肿块，红点，小瘤，畸形，肌肉萎缩等症状
- ObjectiveTo establish spinal muscular atrophy (SMA) cell model by blocking the expression of SMN1 gene with shRNA. 目的：应用RNA干扰沉默SMN1基因的表达建立脊髓性肌萎缩症（SMA）的细胞模型。
