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- METHODS: The diabetic rat model was made with alloxan. 方法利用四氧嘧啶复制糖尿病大鼠模型。
- Objective To establish a typical rat model with syndrome X. 目的建立一种典型的X综合症动物模型。
- Old rat model is more similar with human being of perimenopause. 针对某一症状的模型中,去卵巢大、小鼠的复合模型运用较多。
- Method:Anti\|tumor in a rat model and flow cytometry(FCM). 方法:体内抑瘤实验、流式细胞仪检测及电镜观察。
- This rat model of endosteum ossification was successful. 该动物模型是成功的。
- Rat model of chronic renal failure induced by subtotal nephrectomy. 大鼠肾大部切除诱发慢性肾衰模型的建立。
- Methods Rat model of PIHF was established by left coronary artery ligation. 方法以大鼠左冠状动脉结扎术造成心肌梗死后心衰模型。
- Methods Male SD rats were induced to diabetic rat model with streptozotocin. 大鼠断头处死，分离肾小球，提取纯化胞浆及胞膜蛋白。
- Method: Evoking the DM rats model is by abdominal cavity Injection with STZ. 方法：用链脲佐菌素(TZ)腔注射一次性诱发糖尿病大鼠模型。
- Methods of making rat model of small for size liver transplantation. 大鼠小体积原位肝移植模型的建立。
- Method: The SD rat model with HF was produced by constricting abdominal aorta. 方法:采用缩窄腹主动脉方法复制大鼠心衰模型。
- Methods To establish the rat model with constriction of the abdominal aorta. 方法建立腹主动脉狭窄大鼠模型。
- Methods: A stable rat model of HPH was established by normobaric hy-poxia. 方法：应用常压缺氧方法建立HPH大鼠模型；
- The results indicate that rat model of pulmonary fibrosis replicated by BLM is similar to that of mankind. 结果表明：博莱霉素肺纤维化动物模型的肺病理组织学与病理生理学改变与人肺纤维化相似。
- The AD rat model was established by injecting IBO into rats brain to damage bilateral nbM. 采用大鼠脑组织立体定位微量注射技术,用鹅膏蕈氨酸(IBO)损毁大鼠双侧迈纳特基底核(nbM)制作AD模型。
- Method The rat model of ulcerative colitis was induced with2,4,6 trinitrobenzene sulfonic acid. 方法采用三硝基苯磺酸法制备大鼠实验性溃疡性结肠炎模型。
- Objective: To research the rat model with colpitis and cervicitis by phenol slurry. 目的:研究苯酚胶浆致大鼠阴道及宫颈炎的动物模型。
- These results demonstrated that rat models with DGR was successful . 结果:DGR模型大鼠胃液pH及TBA明显升高(P<0.;01);证明DGR模型成功。
- To establish reliable copolymer-1(COP-1)-immunized chronic EIOP rats models. 建立COP-1免疫的慢性高眼压大鼠模型。
- The number of Clara cells and the secretion of CC16 reduce in a rat model of COPD, which may be a mechanism for COPD. COPD大鼠的气道炎症可导致Clara细胞超微结构改变,Clara细胞数量及CC16的合成及分泌量减少,这可能与COPD的发生发展有一定的关系。
