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- Results There are many pseudopodia and microvilli on the glioma cell sur face. 结果胶质瘤细胞表面有大量伪足结构和微绒毛结构。
- Results NECL1 was abrogated or markedly reduced in 6 glioma cell lines. 结果 NECL1在6株人神经胶质瘤细胞系中的表达明显下降或缺失。
- OBJECTIVE: To investigate the influence of CpG ODN107 on the radiosensitivity of human glioma cell line (CHG-5) and its mechanism. 目的探讨CpG ODN107增加人脑胶质瘤细胞(CHG-5)放射敏感性的作用及可能机制。
- Objective To explore the effect of antisense c-myc on human glioma cell U251 after transfection by dint of non-phospholipid liposome (NPL). 目的 确定非磷脂脂质体 (non phospholipidliposome ,NPL)介导反义c myc基因转染人脑胶质瘤 (glioma)的作用。
- Objective To observe the expression of RNA editing deaminase ADAR2(RED1),ADAR3(RED2)mRNA on various degree malignant glioma cell lines. 目的观察不同恶性程度的胶质瘤细胞中RNA编辑酶ADAR2(RED1),ADAR3(RED2)mRNA水平的表达。
- Conclusion Both under hypoxic and aerobic conditions BSO can deplete intracellular GSH and increase the radiation sensitivity of glioma cell. 结论无论是离体还是整体用药,BSO均能降低胶质瘤细胞的GSH含量。BSO对胶质瘤细胞有放射增敏作用。
- Objective To investigate the proliferation kinetics in C6 glioma cell in rat based on establishment of a C6/SD glioma model. 目的建立C6/SD大鼠脑胶质瘤模型,并在其基础上研究C6胶质瘤细胞的增殖动力学。
- Methods To determine whether the expression of bradykinin B2 receptor are on the vascular endothelial cell or cerebral glioma cell, double immunohistochemistry was used. 方法通过双重免疫组化染色,确定缓激肽B2受体是存在于血管内皮细胞上还是存在于肿瘤细胞上。
- Methods Rat C6 glioma cell line was exposed in vitro to adriamycin assisted with X radiation.Cell apoptosis was ascertained by cell morphology and DNA fragment analysis study. 方法:通过光镜、电镜、DNA断裂分析,进行多柔比星X射线联合诱导C6胶质瘤细胞凋亡研究。
- The pseudopodia and microvilli on the glioma cell surface are associated with the invasive ability of the glioma cells. 胶质瘤的微绒毛和伪足结构与其侵袭性相关。
- Objective To study the reinforcement of nimodipine to the therapy of acyclovir(ACV)-mediated HSV-TK gene to induce glioma cell to death and the mechanisms of the bystander effect in the procedure. 目的 :探讨尼莫地平增强阿昔洛韦 (ACV)介导的转HyTK基因治疗诱导脑胶质瘤细胞的死亡方式及旁观者效应机制。
- TETC inhibits the proliferation of cultured rat C6 glioma cells in vitro, the mechanism of which may be involved in cell cycle arrest of the cells. TETC可抑制体外培养大鼠C6胶质瘤细胞增殖,其机制可能与C6细胞周期阻滞在G0/G1期有关。
- OBJECTIVE: To explore the mechanism of killing tumor cells by observing the C6 rat glioma cell apoptosis after cryotherapy. 摘要目的:通过观察大鼠C6脑胶质瘤经冷冻后出现细胞凋亡,探讨冷冻对肿瘤的杀伤机制。
- Methods: After establishing a C6/SD glioma model,by using flow cytometric analysis, immunohistochemistry and HE stain three methods, the proliferation kinetics in C6 glioma cell was measured. 方法:建立C6/SD大鼠胶质瘤模型后,应用流式细胞仪检测、免疫组化、HE染色三种方法,研究C6胶质瘤细胞的增殖动力学指标。
- Methods Rat glioma cells (D74/HveC) were implanted intracranially in immune-competent rats. 越来越多的研究认为肿瘤微环境是肿瘤生长和治疗反应的决定因素。
- Conclusion Triethyltin chloride can inhibit the proliferation of cultured rat C6 glioma cells and induce its apoptosis. 结论氯化三乙基锡可抑制体外培养C6胶质瘤细胞增殖并诱导细胞凋亡。
- Results BSO depleted the GSH content of glioma cells effectively and increased the radiation sensitivity. 结果经BSO作用后胶质瘤细胞的GSH含量显著下降,放射敏感性增加。
- Abstract: Objective To investigate the invasion ability and collagenolytic activity of human glioma cells in vitro. 文章摘要: 目的:观察人胶质瘤细胞的体外侵袭能力及对胶原的溶解作用。
- Objective: To study the effect and mechanism of morphine on proliferation of rat C6 glioma cells. 目的:研究吗啡对培养的大鼠胶质细胞瘤C6的增殖抑制作用及其机制。
- Abstract Objective To explore the establishment for orthotopic implantation model of human glioma cells in nude mice. 摘要 目的 探讨人脑胶质瘤细胞裸鼠原位移植动物模型建立的技术方法。
