您要查找的是不是：
- CT of brain edema 脑水肿的CT表现
- The deggree of peritumoral brain edema (PTBE)was estimated on the basis of CT of head and MRI . 根据头颅CT或MRI估计瘤周水肿(peritumoral brain edema,PTBE)的程度。
- The size of brain edema was measured with CT on day 0, 1, 3, 7, 14, 21 respectively after SHI. The size of brain edema at the trauma cite and ICP as well as the clinical outcome were compared and analysed. 每例病人伤后0、1、3、7、14、21 d各行一次头颅CT检查，分析比较两组颅内压、伤灶脑水肿体积变化和预后状况。
- The treatment of brain edema type is using hyperosmotic dehydration as core. 脑水肿型以高渗脱水剂为主。
- Results CT of brain stem hemorrhage showed high density hemorrhage and hematoma image. 结果脑干出血CT表现为脑干高密度出血及血肿影。
- Change of brain edema was analyzed by Evans blue extravasation and brain water content. 通过观察Evans蓝扩散距离、脑组织含水量的不同两项指标来观察脑水肿程度。
- Results: Patients treated with NBCA glue embolism had significantly lower rates of brain edema an... 结论:术中NBCA胶栓塞可以显著提高复杂脑海绵状血管瘤的治疗效果。
- Objective To study the clinical features, liver function, the electroencephalogram(EEG),the electrocardiogram(ECG),and the changes of Brain CT of alcoholism mental disorders. 目的为了解慢性酒中毒性精神障碍的临床特点,肝功能、ECG、EEG及头颅CT改变情况。方法分析了慢性酒中毒性精神障碍70例的临床资料。
- The release and activation of -2/9 around hematoma in CH is one of the factors of brain edema forming, and can result in the form of VBE. 大鼠脑出血后MMP-2/9的释放与激活,是脑出血后早中期脑水肿形成的主要因素,导致BBB通透性增加而形成血管源性脑水肿。
- Mechanism of brain edema has close relationship with damage to blood-brain barrier (BBB) and disfunction of brain cell metabolism. 脑水肿的发病机制与血脑屏障(Blood-Brain Barrier，BBB)功能损害和脑细胞代谢紊乱关系最为密切。
- Objective To study the relations between the site of head injury and the position of brain edema induced tentorial notch herniation (TNH). 目的探讨头部受力后导致脑肿胀并发TNH的相互关系。方法采用福尔马林固定的91例大脑标本,根据脑的损伤特点分三组分别作冠、矢及水平状切面,肉眼观察。
- Objective To study the pathophysiological mechanisms of brain edema and pathological changes in both hemispheres after one MCA-OR in rats. 目的探讨鼠一侧大脑中动脉栓塞/再灌流(MCA/OR)后大脑半球脑水肿形成及病理变化的病生机制。
- Objective To investigate the relation between the degree of brain edema and semiconductor laser output power and acting time during heat therapy of brain malignant tumor. 目的 研究脑肿瘤热疗中脑水肿与半导体激光输出功率和作用时间的关系。
- Conclusions AVP level is closely associated with the prognosis after acute brain injury.AVP may play a vital role in pathogenesis of brain edema after acute brain injury. 结论急性颅脑损伤后血浆中AVP的动态变化与急性颅脑损伤预后密切相关，AVP参与了颅脑损伤后继发性脑水肿形成的病理生理过程。
- The article expounds the pathophysiological mechanisms of brain edema formation after acute stroke and the common treatments for brain edema at present. 文章阐述了急性卒中后脑水肿形成的病理生理学机制以及目前治疗脑水肿的常用方法。
- Conclusion MMP-2 and MMP-9 take part in the damage of BBB,the formation of brain edema and the inflammatory reaction and may play important role in the recovery of TBI. 结论 MMP-2和MMP-9参与了创伤性脑损伤后血脑屏障破坏、脑水肿形成、炎性反应等过程，并在损伤修复中可能有重要作用。
- Results There were three types of brain edema induced TNH after cerebral injury: 65 cases with anterior herniation (71.4%), 5 cases with posterior herniation (5.5%), and 21 c... 结论头部受力后易在额颞部发生前位疝。TNH虽与受力部位、受力机制有一定关系,但仍与受力强度、伤后存活时间关系密切。
- A cortical injury in rats was made by the impact method described by Dennis. Animals were sacrificed at 6 hours, 24 hours and 48 hours, respectively after injured. Water content in brain was measured as an indicator of brain edema. 分别于伤后6小时、24’J’时、48小时处死动物，应用干湿重法测定大脑含水量，应用组织学方法观察脑组织病理改变与神经细胞超微结构的差别，同时测定大鼠脑皮层组织 Na＋K＋ATP酶、SOD活性和 MDA水平，评价大黄多糖对脑外伤大鼠的脑保护作用。
- Ct of your E-mail. The resume must be an attachment in Email. 简历请必须以附件。
- And its mechanism might be the effect of AW on AW receptor mediated by cAMP, cGMP, and that in turn inhibited the Na+ -K+ ATPase activity of brain cell membrane, then exaggerated the formation of ischemic brain edema. 其作用机理可能是通过cAMP、cGMP介导的AVP受体的作用,产生抑制脑细胞膜Na~+-K~+ATP酶活力的效应,从而促进缺血性脑水肿的形成。
