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- Methods Focal brain ischemia animal model was used. 方法应用局限性脑缺血动物模型。
- CDP-choline improved the recoveryof SEPs caused by brain ischemia obviously. CDP-choline可明显促进缺血脑功能的恢复。
- Both Brain ischemic preprocessing and postprocessing can result in neuroprotective effect by inducing brain ischemic tolerance. 脑缺血预处理及后处理均可以诱导脑缺血耐受形成而产生神经保护作用。
- DWI is sensitive to early ischemic changes,which can find the changes of brain ischemia earlier than that of routine T2WI. DWI对早期缺血改变非常敏感,DWI较常规T2加权图像能更早地发现脑缺血的改变。
- Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。
- But the detailed mechanisms of G-LOC or G induced brain ischemia as well as protective effect IPC were not clear. 然而不论大G昏迷、G力造成的脑损伤及缺血前制约效应的保护机转都并不清楚。
- It can cause severe complications, such as brain ischemia or cervical hematorrhea incurred by rupture of aneurysm. 它可以引起严重的并发症，比如大脑局部缺血或动脉瘤破裂产生的颈部软组织出血。
- It was suggested that DDPH had some protective effect on acute brain ischemia and ischemia-reperfusion. 结果提示，DDPH对动物脑缺血具有一定的保护作用。
- Abstract: Brain ischemic preconditioning is that transient ischemia in sublethal dose will result in tolerance to the following ischemia in lethal dose for long time. 摘要： 脑缺血预处理即给予短暂亚致死量缺血可对随后的长时间致死性缺血损伤产生耐受。
- Objective To study the improvement effects of GM_1 on brain ischemia reperfusion(BIR) induced memory impairment in mice. 目的探讨脑缺血复灌后连续应用GM1对改善记忆障碍的疗效。
- Disturbances of calcium homeostasis and modification of ca~(2+) pump(Ca~(22+),Mg~(2+)-ATPase)in the rabbit focal brain ischemia. 脑缺血时钙稳态与钙泵(Ca~(2+),Mg~(2+)-ATP酶)活性变化
- Without obvious influence in brain metabolic functions,L1 KO mice is an useful animal model for investigation of the role of L1 in brain ischemia. L1CAM基因缺失对小鼠脑局部代谢功能无明显影响,提示L1 KO小鼠可作为研究L1CAM在脑缺血中作用的动物模型。
- The abnormal DA release inhibited by L-NNA during global ischemia may be one of the protective mechanism of this agent on brain ischemia. L-NNA抑制半球缺血时DA的不正常释放可能是其保护缺血大脑的机制之一。
- Keywords The kidney injury after brain ischemia reperfusion was correlated with the dysequilibrium between ET and CGRP in which ET was dominant. 脑缺血/中医疗法;益元活血丹/治疗应用;大黄/治疗应用;血栓心脉宁/治疗应用;再灌注损伤;肾疾病/中医药疗法;肾疾病/病理学;肾疾病/超微结构;内皮缩血管肽类/代谢;
- When the collateral circulation is good, the radialis velocity may be normal, Whereas, the risk of brain ischemia would be increase. 反之可能会诱发脑缺血的临床症状。
- Chan ges of bcl-xl protein expression were observed at 24 hours after global brain ischemia, and apoptosis were checke d at 72 hours in hippocampal CA1 subregion. 观察缺血后 2 4h海马CA1区bcl -xl蛋白表达和缺血后 72h细胞凋亡。
- GU WP, YANG QD, XIE GJ.Experimental delayed neure death present apoptosis after brain ischemia refilling [J].China Journal of Modern Medicine, 2001, 11(3): 30-31. [2]谷文萍;杨期东;谢光洁.;实验性脑缺血再灌后迟发性神经元死亡表现细胞凋亡[J]
- Cerebral ischemic control group: Gerbils were established into models of cerebral ischemia by occluding bilateral CCA, and reperfused for 72 hours following 5-minute global brain ischemia (GBI). 脑缺血对照组:夹闭沙土鼠双侧颈总动脉造成脑缺血模型。 全脑缺血5min后再灌注72h。
- The mechanism of brain ischemic tolerance (BIT) induced by cerebral ischemic preconditioning (CIP) involves cascades of events including release of neurotransmitter, activation of receptors and gene expression. 脑缺血预处理（cerebral ischemic preconditioning，CIP）诱导的脑缺血耐受（brain ischemic tolerance，BIT）的产生涉及神经介质、受体及基因的表达等一系列过程。
- Some advantages in the field were described in detail includ-ing the basie principle of MR perfusion- weighted imaging, application of imaging diagnosis in acute brain ischemia and its future and prospect. 本文重点介绍了磁共振脑血流灌注加权成像的基本原理,在急性脑缺血疾病诊断中的应用,以及磁共振脑血流灌注加权成像的应用前景和展望。
