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- CVF can significantly inhibit the expression of complement C1qand C3d, reduce brain infarct volume, alleviate brain water content, depress the permeability of BBB, lighten the damage of in pathology and ultra-structure of brain tissue. 眼镜蛇毒因子能显著抑制补体C1q和C3d的表达、减少脑梗死体积、减轻脑组织含水量、降低血脑屏障的通透性，减轻脑组织病理和超微结构的损害。
- Brain infarct volume 脑梗死体积
- Results: ASA can reduce I / R rat infarct volume and neurological function score, the increase in brain tissue SOD activity, the lower the content of MDA, elevated blood PGI/TXA2 ratio. 结果:ASA能够降低I/R大鼠脑梗死体积和神经功能评分,增加脑组织中SOD的活性,降低MDA的含量,升高血PGI/TXA2的比值。
- MAIN OUTCOME MEASURES: Incidence of animal model and infarct volume. 主要观察指标:动物模型出现率;梗死体积。
- NGNHFT can reduce the infarction volume of brain of MCAO rats. 4. 3， 脑功能恢复汤可以减小缺血再灌损伤后动物的脑梗死体积。
- Brain infarction volume 脑缺血再灌注
- Objective: To observe the therapeutic effect of cinepazide maleate on brain infarct. 目的:观察桂哌齐特对脑梗死的治疗效果。
- Objective To observe the effect of Zhongfengkang on the infarct volume of focal cerebral ischemia rats. 目的探讨中风康对局灶性脑缺血大鼠脑梗死体积变化的影响。
- Reduce blood fat and blood pressure, prevent arteriosclerosis and brain infarct; restrain and kill the bacteria, kill the virus and suppression tumor. 降血脂、降血压,预防动脉硬化和脑梗塞;抑菌、杀菌、杀病毒;抑制肿瘤。
- The cerebral infarct area was analysised and the cerebral infarct volume was calculated by computer image processing in the infarct volume group. 脑梗塞体积组,计算机图像分析脑梗塞区面积,计算脑梗塞体积。
- In the future VEGF could be gene therapy to decrease the infarct volume and improve the blood flow of the infarct penumbra area. 对神经元也有保护作用，也向我们提供未来一种新的治疗方法，是否可以利用VEGF的基因治疗，减少脑梗塞的面积，改善缺血周围区的血液供应等。
- Infarct volumes were evaluated by image analysis software. 采用图像分析软件测量梗死体积。
- Conclusion:Tong Nao Ling has marked effect on brain infarction. 通脑灵治疗脑梗塞有较好疗效。
- In infarct volume,group B performed significant difference with group C and group D at 2 d,3 d,and 7 d post pMCAO,but there was no difference between group C and group D. 梗死体积测定;在2、3、7d;B组与C组、D组比较差异有统计学意义(P?0.;05); C组与D组比较差异无统计学意义(P?0
- When USW treatment was started at 18 h after reperfusion, decreased infarct volume and water content were observed, but RM treatment did not show this relationship. 脑缺血再灌注后18h,超短波治疗能减少脑含水量,减小梗死体积;
- Behavior disorders were observed at 3, 6, 12h after the operation. Willis circle, infarct volume and histopathologic changes were examined 12 hours after operation. 于手术后 3、6、12h观察行为障碍并评分 ,手术后 12h取脑观察Willis环、脑梗死体积及病理改变。
- Objestive:To determine relation among therapeutic time windows,infarct volume and electroencephalography (EEG) in renovascular hypertensive stroke-prone rats(RHRSP). 目的：研究不同时间窗脑血栓溶解治疗后脑电变化与梗死体积的关系。
- It has been reported that MMP-2 and MMP-9 are activated after onset of stroke in clinical practice as well as in experimental brain ischemia and inhibiting the activityof MMP-9 can reduce brain infarct. 临床上已有报道MMP-2及MMP-9的活性在脑中风发作后明显升高。 动物实验也显示局部脑缺血后MMP-9在早期就被激活，抑制MMP-9的活性可以减小脑梗塞面积。
- Methods We made infarct models of rats with thread embolism and observed the changes of the infarct volume in rats which had re perfused 4 hours after 2 hours embolism. 方法采用线栓法制作大鼠局灶性脑缺血再灌流模型,大脑中动脉阻塞2小时,再灌流4小时后观察梗死体积的变化。
- For example, in clinical trials, tirilazad therapy for ischemic stroke increases the risk of death and dependency, whereas in animal studies, it reduces infarct volume and seems to improve neurobehavioral scores. 例如，在临床试验中，替拉扎特治疗缺血性中风增加了其死亡的危险和依赖，尽管在动物实验中他减少了梗塞面积，提高了神经功能评分。
