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- Postischemia edapatinn 缺血后适应
- Conclusions: We conclude that postischemia mild hyperglycemia do not aggravate cerebral ischemia reperfusion injury. 结论:缺血后轻度高血糖对缺血再灌注损伤无明显影响。
- Conclusion:NGF has a protective effect on neuronal injury induced by reperfusion postischemia. 结论:NGF对缺血再灌注所致的神经细胞损伤可能具有一定的保护作用。
- Objective: To explore whether postischemia mild hyperglycemia can effect the brain damage in rats. 目的:探讨脑缺血后轻度高血糖对大鼠局灶性脑缺血再灌注损伤的影响。
- OX6 positive ramified cells and round cells appeared in ischemic p enumbra (ramified) and core (round) at 3 day postischemia, and increased and pea ked in number at 1 week postischemia, and then declined gradually. OX6阳性细胞在半暗带区呈分支状 ,而在脑缺血中央区为圆形的阿米巴样细胞。 阳性细胞在脑缺血后 3d出现 ,脑缺血 1周时 ,其形态和数量变化最显著 ,并持续到脑缺血后 2周。
- ATP, ADP, AMP, CP in immature heart preischemia and postischemia 先天性心脏病患儿心肌缺血前后CP,ATP,ADP及AMP的变化
- Keywords Postischemia adapation;Myocardium;Ischeminareperfusion injury; 缺血后适应;心肌;再灌注损伤;
- Postischemia Mild Hyperglycemia Failed to Enlarge Focal Cerebra Infarct Size in Rats 大鼠脑局部缺血后高血糖对再灌注损伤的影响
- Moore EE,Moore FA,Francise RJ,et al. The postischemia gut serves as a priming bed for circulating neutrophils that provoke multiple organ failure.Trauma 1994:37:881-7 曹卫红;胡森;孙丹.;肠道部分缺血再灌流损伤诱发多器官功能障碍综合症[J]
- postischemia 缺血后