您要查找的是不是：
- Objective:To study the relation between severe oligospermia, azoospermia and abnormal chromosome karyotypes. 目的:探讨男性严重少精子症和无精子症与染色体异常的关系。
- FSH and LH in oligospermia were higher than in normal semen, but the differences were not significant (P>0.05). 少精症组FSH、LH虽均较正常精液组高，但无显著性意义（P>0.;05）。
- Conclusion Mutation of Exon A in AR gene plays an important part in infertile men with oligospermia. 结论雄性激素受体基因外显子A即基因转录激活区的突变是造成少精不育的重要原因。
- Objective: To study the meaning of the karyotype analysis in oligospermia and azoospermia. 目的探讨染色体核型分析在少精子及无精子者中存在的意义。
- The patients with oligospermia and asthnospermia, Group A was higher than group B in normal sperm morphology percent. 在少精组及弱精组中A组精子正常形态率高于B组，差异有统计学意义。
- Conclusion: chromosomal abnormalities play an important role in oligospermia and azoospermia. 结论染色体异常是导致少、弱精子及无精子症的重要因素之一。
- CONCLUSION: Both of abnormal chromosome karyotype and Y-chromosome microdeletion are important to cause azoospermia and severe oligospermia. 结论：染色体核型异常和Y染色体微缺失均与无精子症和严重少精子症的发生有关。
- Objective To investigate the clinical value of the determinations of serum sex hormone in the patients with oligospermia and azoospermia. 目的探讨少精、无精症患者血清性激素检测的临床应用价值。
- Objective:To investigate the effects of Shengjingbao on spermatogenesis in the mouse model of oligospermia and its action mechanisms. 目的:研究"生精宝"对小鼠少精子症模型生精功能的作用并探讨其机制。
- Objective: To study the imprinting status of H19 in oligospermia and asthenospermia and to investigate the correlation between H19 and spermatogenesiss disturbance. 目的:通过研究印迹基因H19在少弱精子症中的印迹状态,探讨H19与精子发生障碍的相关性。
- Objective In order to efficiently evaluate the result to treat oligospermia or asthenozoospermia with combination of Chinese Traditional and Western medicine. 摘要目的采用新标准更科学地评估中西医结合治疗少弱精子性不育症的疗效。
- Results FSH and LH in azoospermia and severe oligospermia were all significantly higher than in normal semen (P<0.01), and FSH was raised more highly than LH each other. 结果无精症组及重度少精症组FSH、LH均显著高于正常精液组（P<0.;01），且FSH升高幅度均大于LH。
- BACKGRONUD &AIM: To investigate the relationship between spermatogenesis disorder and genetic defects of patients with azoospermia or severe oligospermia. 摘要背景与目的：探讨无精子症和严重少精子症患者的遗传缺陷与精子生成障碍的关系。
- Adverse effects were bilateral nosocomial pneumonia in 2 patients, late endocrine dysfunction in 3 patients, and oligospermia in 9 patients.There were no deaths. 不良效果是2例患者双侧医院性肺炎，3例患者晚内分泌功能失调，9例患者少精液症，但是没有患者死亡。
- Methods: The periphery blood lymphocyte was cultured and the G-banding chromosome was analyzed on 394 male patients with the severe oligospermia and azoospermia. 方法:对364例严重少精子症和无精子症患者进行外周血淋巴细胞培养,G显带染色体核型分析。
- BACKGRONUD & AIM: To investigate the relationship between spermatogenesis disorder and genetic defects of patients with azoospermia or severe oligospermia. 背景与目的：探讨无精子症和严重少精子症患者的遗传缺陷与精子生成障碍的关系。
- Compared with the normal controls.PCNA proliferation index (PI) was lower (P<0.001) and apoptosis index (AI) higher (P<0.01) in testes of oligospermia patients. 结果与正常睾丸相比，治疗前患者PCNA增殖指数(PI)偏低(P<0.;001)，凋亡指数(AI)较高(P<0
- Results: The proportion of autosomal structure abnormalities was 5.1% (23/454) among the patients with idiopathic oligospermia and azoospermia, while that of chromosomal polymorphism was 8.8%(40/454). 结果在454例特发性生精异常患者中有23(5.;1%25)例涉及常染色体结构异常;40(8
- Conclusions Tamoxifen significantly decreases the incidence of germ cell apoptosis and improve the spermatogenic function.Tamoxifen is recommended to advocate in cases of idiopathic oligospermia. 结论少精子症精子减少的主要原因之一是由于生精细胞增殖能力减低及凋亡增加，它莫昔芬可纠正生精细胞增殖与凋亡的失衡状态，改善患者生精功能及生育能力。
- It is mainly indicated for male loss of libido, short and small penis, dysfunction of erection and premature ejaculation, no activity of spermatozoa, oligospermia and other conditions. 藏药与其它药的区别在于藏药多为主药材及金诃为药引，不加别的成份，而中药往往以淀粉、红糖等作为辅料。
