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- obese gene receptor 瘦素受体
- DATA SYNTHESIS: The leptin was codes by the obese gene (ob), the fatty tissue secreted the protein, the known its main function was the regulation eats food, the energy consumption and the body mass. 资料综合:瘦素(Leptin)是由肥胖基因(ob)编码,脂肪组织分泌的蛋白质,已知其主要功能是调控进食、能量消耗及体质量。
- Leptin is one endogenous excitatory autacoid, encoded by obese gene and secreted by adipose cells. 瘦素(leptin)是肥胖基因(ob基因)编码，脂肪细胞分泌的一种内源性激素，主要调控体重和脂肪分布。
- Zhang YY, Proenca R, Maffei M, et al. Positional cloning of the mouse obese gene and it's human homdogue[J]. Nature,1994, 372,425-432. 汪丽惠，许广润，张树基，等．现代内科诊疗手册[M]．北京医科大学、中国协和医科大学联合出版社，1994．507
- Isse N,Ogawa Y,Tamura N,et al.Structural organization and chromosomal assignment of the human obese gene[J].J Biol chem,1995,270:2778. 徐淑静;徐明彤;傅祖植;等.;肥胖基因和瘦素[J]
- Obese Gene and Its Receptor in the Regulation of Fat Deposition 肥胖基因及其受体与脂肪沉积调控
- Plasma obese gene(OG) product [OP-(57-92)] level was measured with ra-dioimmunoassay in 21 normal subjects ,24 adult obese patients and 20 patients with type I diabetes (DM I ). 采用放射免疫分析法测定了21例非肥胖正常人,24例肥胖患者及20例糖尿病患者血浆肥胖蛋白[OP-(57-92)]水平。
- Mouse obese gene was amplified by PCR and subcloned into prokaryotic expression vector pET-28a(+) to construct the recombinant pET-28a-lep. Transforming pET-28a-lep into E. 本研究以克隆有小鼠肥胖基因的质粒pMET mouse leptin为模板，PCR扩增小鼠的ob基因，定向克隆至原核表达载体pET-28a(+)，构建重组表达质粒pET-28a-lep。
- In this article,we introduced the discovery,Configuration,funetion and orientation of obese gene and orexin gene ,It also introduced the pathogenesis of adiposity. 本文介绍了肥胖基因和增食欲素基因的发现、结构、功能、定位及其肥胖发病的机制。
- The scientific theory improved that obese gene can help our ancestor get through staving, while in this age of plentiful supply of food, obese gene is now getting us into troubles. 科学理论揭示，肥胖基因可帮助我们的祖先熬过饥荒，然而在现在这个食物充裕的时代，却正在给我们的生活带来很多麻烦。
- Obese Gene Research and its New Progress 肥胖基因的研究及其新进展
- Cloning of Human Obese Gene and its Expression 人类肥胖基因克隆和表达
- Expression of human obese gene in E. Coli 人肥胖基因在大肠杆菌中的表达
- The discovery of the obesity gene in humans half a decade ago offered evidence that chronic weight gain is the consequence of a mismatch between nature and nurture. 5年前人类肥胖基因的发现提供了证据，从生物学的角度，证明了慢性体重增长是营养和身体本能不协调的结果。
- Obese patients are advised to change their diet. 建议肥胖病人改变饮食。
- Obese people find it difficult to stop overeating. 胖人觉得一吃就过量，难以控制。
- The case for FTO's inolement is strengthened by the fact that other obesity gene hunters are finding the FTO polymorphism as well. FTO的发现事实证实了其他肥胖基因寻求者也探求FTO的同质异形结构。
- The obesogenous reasons were discussed in three factors, Which is obesity gene, insulin and fat cell, and the factors were affected by aerobic exercise. 肥胖症;;胰岛素;;脂肪组织;;脂细胞;;基因;;氧;;体育运动
- OB: How did you get into classical conducting? 你是如何开始古典音乐的指挥的?
- The gene is called GRPR, for gastrin-releasing (peptide) receptor. 该基因称GRPR，即胃泌素释放汰受体。
