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- There was no neurological deficit. 没有神经方面的缺陷。
- Neurological deficit was found in 9 cases in group B,while no case in group A. B组有9例患者术后即刻出现神经功能障碍加重,而A组未出现类似病例。
- We report a case of NCM in a patient with previously robust health.No neurological deficit was noted in the patient. 我们报告一例神经皮肤性黑色症,病人过去之健康状况良好且无神经学之异常,于二十一岁时以视力模糊为初始神经学症状表现。
- Bergmann, T.F. &Jongeward, B.V.Manipulative therapy in lower back pain with leg pain and neurological deficit. 李现林.;骶管注射配合整脊疗法及中药熏蒸治疗腰椎间盘突出症
- Brain tissue was taken out to undergo TTC and HE staining and neurological deficit sign scores were performed. 取脑组织作TTC染色和HE染色,并进行神经功能缺陷体征评分。
- Conclusion: Hypoxic preconditioning could decrease the neurological deficit and the neuronal apoptosis of rats with ischemia-reperfusion. 结论:低氧预适应可降低大鼠脑缺血再灌注后的神经功能缺陷和神经元凋亡。
- The values of SOD,MDA and GSH were detected and the neurological deficit scores were observed and recorded in all ACI patients. 结果与对照组相比,治疗组疗效出现时间显著提前,神经功能缺损明显改善,自由基生成明显减少。
- The persistent focal neurological deficit with gross CT-documented infarction could occasionally be found in patients with TTP. 偶尔,我们也可以见到CT证实为较大梗塞的,表现为持续轻偏瘫的TTP病人。
- The prognosis relates with age, level of consciousness, neurological deficit and size, location and the direction of extension of hemotoma showed on CT. 预后取决于年龄、意识水平、神经功能损害程度以及血肿的大小、部位和扩展方向。
- Vascular malformation can be found in various parts of the central nervous system and usually presents with headache, seizures, and focal neurological deficit. 摘要血管畸形可发生于颅内各处,多以癫痫及局部神经学障碍为表现。
- METHODS The FCI - R model with thread embolism of middle cerebral artery occlusion (MCAO) in rats was made. The neurological deficit score(NDS) was assessed. 方法 采用大鼠局灶性大脑中动脉阻断(MCAO)模型,于缺血再灌注(I/R)后第22、70小时分别进行神经功能评分(NDS);
- The effect of Borneol on neurological deficit and the ratio of right/left cerebral hemisphere areas, infarct size in mice with middle cerebral artery occlusion. 结果冰片注射液可延长小鼠结扎双侧颈总动脉及迷走神经、常压耐缺氧后的存活时间,提高MCAO小鼠行为学评分,降低脑梗死面积及脑指数。
- Objective To summarize the experience of treating postoperative cerebral vasospasm and related delayed ischemic neurological deficit (DIND) of intracranial aneurysm. 目的总结动脉瘤手术后血管痉挛及延迟性缺血性神经功能障碍(DIND)的防治经验。
- Neurological deficits occurred postoperativ-ely in 2 patients. 无手术死亡;2例术后发生神经功能缺失.
- Treatment group were perfomed both edaravone and naloxone.The curative effect and neurological deficit scale (NDS) were compared before and 2 weeks after therapy. 对照组予常规治疗,治疗组在此基础上加用依达拉奉和纳洛酮治疗,比较治疗前、治疗后2周临床疗效及神经功能缺损评分(NDS)。
- Group A and B had the highest recurrence of hyperthermia, and the group C and D had the best treatment effect according to comparison of neurological deficit scores (NDS) and activity daily living (ADL) before and after treatment (P<0.05). 体温再发率以A、B两组最高(P <0.;05); 治疗前后神经功能缺损评分(NDS)、日常生活质量评分(ADL)比较;以C、D组效果最佳(P < 0
- The rest was recovery.Conclusion:Early diagnosis, effective haemostasis and reconstruction of carotid artery are the keys to reduce the mortality and decrease the morbidity of neurological deficit. 结论:早期诊断,及时有效的止血,修复血管,尽快恢复颈动脉血流和必要的脑保护措施是降低病死率,减少神经系统并发症的关键。
- With univariate and multivariate regression analysis, prognostic factors significantly associated with survival included intrathecal injection(P=0.005) and local/diffuse neurological deficit(P=0.031). 并发现是否做过鞘内注射(P=0.;005)及累及部位是局限或弥漫(P=0
- Methods Record scores of the Hamilton Rating Scale for Depression (HRSD) for each PSD patient.Assess the degree of neurological deficit by the modified Scandinavian Stroke Scale (MSSS). 方法:220例初发脑卒中住院患者,以汉密尔顿抑郁量表评分评定其抑郁程度,以神经功能缺损程度评分标准评定神经功能缺损程度,分析二者相关性,同时分析卒中部位及病程与抑郁症发病的关系。
- Post-operative temporarily neurological deficits including aphasia,motorial and sensory disturbance presented in8patients,permanent deficit in2. 术后一过性语言、运动或感觉障碍8例,永久性功能障碍2例。