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- Results Spinal cord, blood vessels, membrana pleuralis or lung tissue injury wasn't found.The direction and length of the screws were satisfied in the postoperative X-films. 结果患者术后均未出现脊髓神经、血管、胸膜或肺组织损伤等并发症。
- Influence of ambroxol on paraquat-induced lung tissue injury and change of pulmonary surfactant-associated protein A in the experimental rats 氨溴索对百草枯中毒大鼠肺组织损伤及肺表面活性蛋白-A的影响
- thoracic surgery itself is a kind of healthy lung tissue injury, lung resection I am prone to pulmonary lung Atelectasis appeared significantly increased risk of lung infections. 开胸手术本身对健康肺组织就是一种损伤，肺切除术后余肺很容易出现肺不张，出现肺部感染的机率明显增加。
- lung tissue injury 肺损伤
- To observe the pathological changes of lung tissue. 观察肺组织病理变化。
- During One-lung ventilation,detelectasis/dilation of lung tissue,ischemic reperfusion injury and hyoxemia can result in or accelerate lung injury. 在单肺通气过程中,肺组织的萎陷/复张、缺血再灌注损伤和低氧血症均可导致并加重肺损伤。
- METHODS: Models of rabbits I/R injury were made.The effects of astragalius membranaceus on PS, LPO in blood plasma and lung tissue were tested. 方法：复制家兔肠损伤模型，测定血浆和肺组织LPO及肺表面活性物质含量变化，同时计算肺系数及进行肺病理学检查，并观察黄芪对LPO及肺表面活性物质含量的影响。
- The two lung abscesses seen here are examples of liquefactive necrosis in which there is a liquid center in an area of tissue injury. 暗红甚至是灰色的梗死肠段与下部的粉红色正常肠段有着明显的区别。
- Objective To explore the change rule of G protein coupled receptor kinase 2(GRK2) in the lung tissue of rats with acute lung injury(ALI) induced by lipopolysaccharide (LPS). 目的探讨脂多糖(LPS)诱导急性肺损伤(ALI)大鼠肺组织G蛋白偶联受体激酶2(GRK2)的变化规律。
- Lung tissue atrophies and is not as efficient with age. 衰老的肺组织萎缩，效率降低。
- These waes reduce the lung tissue which has been thickened by asthma attacks. 无线电波可减轻哮喘发作引起的肺组织增厚。
- Kinins are proinflammatory peptides that are released during tissue injury. 激肽是在组织损伤时释放的一种促炎症反应的肽类物质。
- Conclusion The soluble IC of HFRS can cause direct tissue injury in the HFRS. 结论在HFRS的发病中，HFRS的可溶性IC可直接引起组织损伤。
- The expression of CLIP-170 in lung tissue was increased significantly(P<0.01) after injury,then reduced indistinctly(P>0.05) with the development of the repair process and increased again in the whole fibrosis process(P<0.01). 与对照组比较;CLIP-170的表达在气道上皮急性损伤后有明显上升趋势(P<0.;01);随修复进程其表达有所下调;但差异无显著性意义(P>0
- After 90 minutes ischemia and 120 minutes reperfusion,W/D,MDA,LPI, MPO activity and LTD degree were significantly higher in I/R group than those of the control group (all P < 0.01, respectively ), and the morphology injury of lung tissue was obvious. 经 90 min缺血、12 0 m in再灌注后 ;缺血再灌注组的 W/D、L PI、MDA、MPO活性及 L TD程度较对照组均显著升高 (P均 <0 .;0 1);肺组织病理损伤明显。
- These waves reduce the lung tissue which has been thickened by asthma attacks. 无线电波可减轻哮喘发作引起的肺组织增厚。
- Inflammation of lung tissue. 局部急性肺炎肺部组织的炎症
- Too many neutrophils in the lung tissue for too many years can cause serious injury to the lungs, resulting in time in emphysema. 肺组织里中性白细胞过多，积聚的年月太长，就会造成肺严重损伤，最终导致肺气肿。
- Experimental study of acute soft tissue injury treated with Crocus sativus cream. 西红花膏治疗急性软组织损伤的实验研究。
- Protects skin, eyes, liver and lung tissues against free radical damage. 保护皮肤、眼睛、肝脏及肺组织不受游离基破坏。
