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- INFLUENCE THE FACTORS FOR LDL?ACM COMPLEX TAKE IN LEUKEMIA CELL LINE K??562? 影响白血病细胞株 K_(562) 对 LDL-ACM 复合物摄取因素的研究
- Objective To explore the effect of Cepharanthine (CEP)on human leukemic cell line K562 and its multidrug-resistant counterpart K562/ADR. 目的 研究盐酸千金藤素(CEP)对K5 62 /ADR细胞多药耐药性的逆转作用及逆转机制。
- Abstract Objective:To explore the suppression of curcumin on leukemia cell line HL60/DAR. 目的:研究姜黄素对白血病耐药细胞HL60/ADR的抑制作用。
- Objective To explore the cytotoxicity of 2-chlorodeoxyadenosine (2-CDA) to sensitive or multidrug-resistant (MDR) K562 leukemia cell line. 目的 探讨 2 -氯脱氧腺苷 (2 - CDA)对多药耐药白血病细胞 (K5 6 2 / A0 2 )的毒性作用。
- HHV 6 DNA sequence was also present in two leukemia cell lines. 两株白血病细胞株也扩增出HHV?6DNA序列。
- Objective: To study the anti-leukemia effect of hydroxyapatite(HAP) nanoparticles on human leukemia cell line K562 in vitro. 目的:探讨羟基磷灰石(HAP)纳米粒子体外抑制人白血病细胞K562的生长增殖作用。
- Objective: To investigate the differentiation of multidrug resistant human leukemia cell line K562/ADM induced by cyclic adenosine monophosphate (cyclic AMP, cAMP). 目的:研究环腺苷酸(cyclicadenosinemonophosphate,cAMP)对人白血病多药耐药K562/ADM细胞的诱导分化作用。
- Objective: To investigate the expression of Fas, Fas ligand (FasL) and CD80 on the cell surface of mouse acute myelomonocytic leukemia cell line WEHI-3 and the function of FasL. 目的研究小鼠急性粒-单核白血病WEHI-3细胞系表面Fas、Fas配体(FasL)、CD80分子表达以及FasL的功能。
- Objective: To explore whether gancyclovir (GCV) can inhibit the proliferation and induce the erythro differentiation of the K562 human myeloid leukemia cell line. 目的:探讨更昔洛韦(GCV)对人白血病细胞的抗增殖和诱导分化作用。
- Methods: Under ATP-BLA, multilabel counter was used to determine the luminescent value of ATP and leukemic cell line K562 which had different cellular concentration, cultured time and drug-exposed conentration. 方法:采用ATP鄄BLA法,用液体闪烁记数仪检测ATP标准品以及白血病细胞株K562在不同细胞浓度、药物浓度、培养时间下的荧光发光值。
- Methods:The effects of cyclosporin A (CsA) on drug sensitivity,intracellular drug accumulation and drug efflux in multidrug resistant human leukemic cell line K562/DOX were studied by MTT colorimetric assay and spectrofluorimetry. 方法:以环孢霉素A(CsA)作为耐药逆转剂,用MTT法体外药物敏感试验,观察CsA对多药耐药白血病细胞株K562/DOX的药物敏感性、细胞内药物的积聚和外排的影响。
- BACKGROUND & AIM:To examine the inhibitory effects of Phellinus linteus intracellular polysaccharide(PLIP) on proliferation of leukemic cell line K562,and to explore its preliminary mechanisms. 背景与目的:观察桑黄胞内多糖(PLIP)对体外培养的人白血病细胞K562的增殖抑制作用,并初步探讨其作用机制。
- Methods:APAF-1 cDNA types in leukemic cell lines K562 and CEM/VLB100 were studied by cDNA cloning strategy. 方法:应用RT-PCR结合测序方法研究白血病细胞系K562和CEM/VLB100APAF-1cDNA类型;
- DBA/2 mice i.p.transplanted with lymphocytic leukemic cells(L1210 cell line)were employed to study the effects of three extractions from Tephroseris kirilowii Turcz. 利用淋巴性白血病L1210细胞荷瘤DBA/2小鼠为模型,研究了狗舌草的3种提取物对供试小鼠生存期的影响。
- DBA/2 mice i.p.transplanted with lymphocytic leukemic cells (L1210 cell line) were employed to study the effects of three extractions from Tephroseris kirilowii Turcz. 摘要利用淋巴性白血病L1210细胞荷瘤DBA/2小鼠为模型,研究了狗舌草的3种提取物对供试小鼠生存期的影响。
- Abstract BACKGROUND & AIM: To test the feasibility to use leukemic cell lines in the screening process for anticancerous drugs. 摘要 背景与目的: 探讨以人白血病细胞系筛选抗癌药物的可行性。
- Methods Apoptosis induced by Ara c in human myeloid leukemia cell line HL 60 was investigated by applying light microscopy, electron microscopy combined with DNA electrophoresis and flow cytometry analysis techniques. 方法应用光镜、电镜,结合DNA电泳及流式细胞仪分析技术,观察阿糖胞苷(Ara-c)诱导的急性髓系白血病细胞系HL-60凋亡模式。
- Objective: To study the effect of Rhizoma typhonii extract (RTE) on T Lymphocytic leukemia Cell Lines CEM. 目的:探讨附子提取物对T淋巴细胞白血病细胞株CEM的作用。
- Using Immunohistochemistry to detect Fas\Caspase-3 expression in 18 leukemia patients and 2 leukemia cell lines. 应用免疫组织化学SABC法检测临床白血病患者化疗前和细胞株凋亡前后Fas、Caspase-3的表达。
- Preliminary study on the characteristics and functions of MAGE-H1/Apr-1At the initial stage of this research, the total RNA was isolated from the apoptotic human leukemia cell line HL-60 that was induced byall-trans-retinoic acid (ATRA). 提取了维甲酸诱导凋亡后的HL-60细胞的总RNA,根据MAGE-H1/Apr-1的cDNA序列设计引物,运用RT-PCR技术扩增出MAGE-H1/Apr-1的开放读框序列并测序。