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- CONCLUSION DK can protect myocardium against ischemia reperfusion injury. 结论dk对大鼠心肌缺血再灌注损伤有保护作用。
- NGF could ease the delayed neuron death (DND) after ischemia reperfusion. NGF可以减轻海马迟发性神经细胞死亡 (DND)性损伤。
- Conclusion: Ischemia reperfusion could induce the apoptosis of the cell. 说明缺血再灌注损伤能诱发细胞凋亡。
- Egb761 may reduce the myocardial ischemic reperfusion injury of the patients undergoing open heart surgery. Egb761能减轻心脏直视手术后心肌缺血再灌注损伤。
- Methods: Ischemic reperfusion model of the hindlimb was set up,and 30 rats were randomly divided into three groups. 方法: 制作右后肢缺血再灌注模型,30只大鼠随机分为3组,假手术组;
- Aim: To study the protective effects of Bergenin on cerebral ischemic reperfusion in mice. 摘要目的:研究岩白菜素对小鼠脑组织缺血再灌注的保护作用。
- Conclusion BDE may have protect effect on cerebral ischemic reperfusion damage by decreasing Nos activity. 结论BDE抑制神经元NOS活性,对脑缺血再灌流损伤具有脑保护作用。
- Results The HSP70 expression of group using Flunarizine was higher than that of ischemic reperfusion group (P<0.05). 结果氟桂利嗪组HSP70表达高于缺血再灌注组(P<0.;05)。
- Conclusion:Ligustrazine could preserve the renal function of ischemic reperfusion ... 结论:川芎嗪对大鼠缺血再灌注损伤肾组织具有保护作用。
- Spinal Cord Ischemic Reperfusion Injury(SCII) is a serious nervous system injury. 脊髓缺血-再灌注损伤(SCII)是一种严重的神经系统损伤。
- During One-lung ventilation,detelectasis/dilation of lung tissue,ischemic reperfusion injury and hyoxemia can result in or accelerate lung injury. 在单肺通气过程中,肺组织的萎陷/复张、缺血再灌注损伤和低氧血症均可导致并加重肺损伤。
- Objective To study the expression of HSP70 and the effects of tetramethylpyrazine (TMP) on cerebral ischemic reperfusion injury (CIRI) in rats. 目的研究大鼠脑缺血?再灌注(CIRI)时热休克蛋白(HSP)70表达及川芎嗪对HSP70表达的影响。
- AIM: To study the effect of ischemic postconditioning on myocyte apoptosis in rats with myocardial ischemia reperfusion (MIR). 目的:观察缺血后处理对大鼠缺血再灌注心肌细胞凋亡的影响。
- The BWC and EB of the brain increased after cerebral ischemic reperfusion and peaked at reperfusion 2d,and decreased after applying phycocyanin. 脑缺血再灌注6h BWC和EB含量增加,至2d达高峰,应用藻蓝蛋白后均明显减少。
- Methods 60 Wister rats were divided randomly into the following groups: normal control group, sham operative group and ischemic reperfusion group. 方法将 60只 Wistar大鼠随机分为正常对照组、假手术组、缺血再灌注组。
- Methods Seventy two Wister rats were randomly divided into ischemia reperfusion (I/R) group and ischemic pre condioning (IPC) group. 方法 实验选用大鼠 72只 ,分为缺血再灌注 (I R)组和IPC组。
- Ischemic postconditioning can relieve the ischemic reperfusion injury, the mechanism of which may be related with the decreased myocardial apoptosis. 缺血后处理可减轻缺血再灌注损伤、其机制可能与减少心肌细胞凋亡有关。
- Myocardium of rats in both ischemic reperfusion(IR) and IPC groups were subjected to 30min of ischemia followed by 120min of reperfusion. IPC组行3次缺血5min,再灌注10min的预处理,缺血再灌注(IR)组与IPC组行30min缺血及120min再灌注;
- Preservative injury and degree of ischemic reperfusion injury are common causes of influencing renal function and occurrence of reject reaction. 热缺血和冷缺血时间及缺血再灌注损伤程度是影响术后肾功能恢复和急、慢性排斥反应发生的重要因素。
- Currently ischemic preconditioning is one of the most efficacious ways to treat ischemia reperfusion via triggering endogenously protective system. 缺血预适应是目前治疗缺血/再灌注损伤最为有效方法之一,其机制是通过调动机体内源性保护体系而发挥作用。