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- Conclusion Captopril is able to preserve ischemic myocardium. 结论卡托普利对缺血心肌具有保护作用。
- Objection: To assess ischemic myocardium area by tissue velocity imaging(TVI). 目的:探讨组织速度显像技术(TVI)对心肌缺血面积的估测作用。
- Objective: To study the effect of adenine sulphate preconditioning(ASPC) on apoptosis of rat s ischemic myocardium. 目的:探讨硫酸腺嘌呤预处理对大鼠缺血心肌细胞凋亡的保护。
- PF significantly enhanced DTP and VFT of ischemic myocardium and increased ERP (all P <0.01). PF显著提高缺血心肌的 DTP和 VFT(均 P<0 .;0 1);延长 ERP(P<0
- Results:Thrombolysis therapy of urokinase can significantly decrease MIS,saving ischemic myocardium. 结果:用尿激酶溶栓治疗可显著减少(MIS),挽救缺血濒死的心肌;
- Abstract： Reperfusion of ischemic myocardium is necessar y to salvage tissue from ultimate death. 摘 要： 心肌缺血后的再灌注对于挽救心肌细胞死亡是非常必要的。
- Objective To observe the morphological change of nucleus of ischemic myocardium. 目的观察缺血心肌细胞核的形态改变。
- Reperfusion of ischemic myocardium is necessar y to salvage tissue from ultimate death. 心肌缺血后的再灌注对于挽救心肌细胞死亡是非常必要的。
- In the meantime, the fibroatrophy of the host ischemic myocardium had been effectively inhibited. 卫星细胞在缺血心肌中可分化成心肌细胞样细胞；
- Objective To study the features of abnormal left ventricular diastolic function in ischemic myocardium by DTI (Doppler Tissue Imaging). 目的 利用DTI技术研究缺血心肌左室舒张功能损伤特征。
- The sensitivity and specificity of SRI with DSE for detecting ischemic myocardium were (90.0)% and (91.7)%, respectively. 应变率检测缺血节段的敏感性和特异性分别为90.;0%25和91
- DNA ladder pattern of ischemic myocardium was revealed by agrose gel electrophoresis in MIR group while it was not found in RIP+MIR group. 凝胶电泳显示 ,MIR组的缺血组织DNA呈云梯状 ,RIP +MIR组则无 ;
- DTI DSE can quantificatively detect the changes of wall motion of ischemic myocardium,and it's promised to detect ischemic myocardium noninvasivly. DTI技术与DSE结合可定量评价缺血心肌室壁运动异常 ,对冠心病缺血心肌的无创性诊断具有应用价值
- Murry CE, Jennings RB, Reimer KA. Preconditioning with ischemia:a delay of lethal cell injury in ischemic myocardium[J]. Circulation,1986,74(5): 1124. 李庆林;陈志武;马传庚.;黄蜀葵花总黄酮对心肌损伤的保护作用及其机制[J]
- Objective To evaluate the pharmaceutical effect of angiotensin-converting enzyme inhibitor(ACEI) captopril on ischemic myocardium. 目的评价血管紧张素转换酶抑制剂(ACEI)卡托普利对缺血心肌的药理作用。
- Murry CE, Jennings RB, Reimer KA. Preconditioning with ischemia: a delay of lethal injury in ischemic myocardium [J].Circulation, 1986,74:1124. 鲁巍峰;夏强蛋白激酶C与缺血预处理心肌保护作用[J].;生理科学进展;1999;30(1):74
- Preclinical studies hae indicated that human CD34+ stem cells induce neoascularization in ischemic myocardium, which enhances perfusion and function. 长期的研究已经表明在缺血心肌用人CD34+干细胞移植诱导新血管再生能增加心肌灌流和心功能。
- Exogenous MSCs labeled with bromodeoxyuridine (BrdU) were injected into the border and central area of the ischemic myocardium. 采用局部注射法，将5溴脱氧尿核苷(BrdU)标记的MSCs移植到兔梗死心肌边缘带和中心区。
- Murry CE,Jennings RB,reamer KA.Preconditioning with isehemia:a delay of lethal cell injury in ischemic myocardium[J].Circulation,1986,74(8):1124. 易受乡;林亚平;严洁;等.;针刺内关与缺血预处理对心肌缺血再灌注损伤保护作用的比较[J]
- This paper aims at how the correlation between RAS and NO affecting the ischemic myocardium and the mechanism of VEGF expression. 本文就这二者之间的联系对于缺血心肌的影响及血管内皮生长因子表达的机制作一综述。
