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- MAIN OUTCOME MEASURES: Incidence of animal model and infarct volume. 主要观察指标:动物模型出现率;梗死体积。
- Objective To observe the effect of Zhongfengkang on the infarct volume of focal cerebral ischemia rats. 目的探讨中风康对局灶性脑缺血大鼠脑梗死体积变化的影响。
- The cerebral infarct area was analysised and the cerebral infarct volume was calculated by computer image processing in the infarct volume group. 脑梗塞体积组,计算机图像分析脑梗塞区面积,计算脑梗塞体积。
- In the future VEGF could be gene therapy to decrease the infarct volume and improve the blood flow of the infarct penumbra area. 对神经元也有保护作用,也向我们提供未来一种新的治疗方法,是否可以利用VEGF的基因治疗,减少脑梗塞的面积,改善缺血周围区的血液供应等。
- In infarct volume,group B performed significant difference with group C and group D at 2 d,3 d,and 7 d post pMCAO,but there was no difference between group C and group D. 梗死体积测定;在2、3、7d;B组与C组、D组比较差异有统计学意义(P?0.;05); C组与D组比较差异无统计学意义(P?0
- When USW treatment was started at 18 h after reperfusion, decreased infarct volume and water content were observed, but RM treatment did not show this relationship. 脑缺血再灌注后18h,超短波治疗能减少脑含水量,减小梗死体积;
- Behavior disorders were observed at 3, 6, 12h after the operation. Willis circle, infarct volume and histopathologic changes were examined 12 hours after operation. 于手术后 3、6、12h观察行为障碍并评分 ,手术后 12h取脑观察Willis环、脑梗死体积及病理改变。
- Infarct volumes were evaluated by image analysis software. 采用图像分析软件测量梗死体积。
- Objestive:To determine relation among therapeutic time windows,infarct volume and electroencephalography (EEG) in renovascular hypertensive stroke-prone rats(RHRSP). 目的:研究不同时间窗脑血栓溶解治疗后脑电变化与梗死体积的关系。
- Methods We made infarct models of rats with thread embolism and observed the changes of the infarct volume in rats which had re perfused 4 hours after 2 hours embolism. 方法采用线栓法制作大鼠局灶性脑缺血再灌流模型,大脑中动脉阻塞2小时,再灌流4小时后观察梗死体积的变化。
- NGNHFT can reduce the infarction volume of brain of MCAO rats. 4. 3, 脑功能恢复汤可以减小缺血再灌损伤后动物的脑梗死体积。
- CVF can significantly inhibit the expression of complement C1qand C3d, reduce brain infarct volume, alleviate brain water content, depress the permeability of BBB, lighten the damage of in pathology and ultra-structure of brain tissue. 眼镜蛇毒因子能显著抑制补体C1q和C3d的表达、减少脑梗死体积、减轻脑组织含水量、降低血脑屏障的通透性,减轻脑组织病理和超微结构的损害。
- For example, in clinical trials, tirilazad therapy for ischemic stroke increases the risk of death and dependency, whereas in animal studies, it reduces infarct volume and seems to improve neurobehavioral scores. 例如,在临床试验中,替拉扎特治疗缺血性中风增加了其死亡的危险和依赖,尽管在动物实验中他减少了梗塞面积,提高了神经功能评分。
- The infarct volume ratios of the rats of group A was tended to be smaller and more inconsistent than group B (coefficient of variation (CV) of the group A is 1.08, CV of the group B is 0.12). B组大鼠脑梗死体积比明显较A组稳定;变异系数(coefficient of variation; CV )分别为CVB =0.;12;CVA=1
- Results: ASA can reduce I / R rat infarct volume and neurological function score, the increase in brain tissue SOD activity, the lower the content of MDA, elevated blood PGI/TXA2 ratio. 结果:ASA能够降低I/R大鼠脑梗死体积和神经功能评分,增加脑组织中SOD的活性,降低MDA的含量,升高血PGI/TXA2的比值。
- The model of reversible middle cerebral artery occlusion(MCAO) was used to study the effect of electroacupuncture(EA) on somatosensory evoked potential (SEP) and infarct volume after MCAO in rats. 本实验在大脑中动脉阻塞(MCAO)及再灌注动物模型上,观察缺血后电针对大鼠体感诱发电位(SEP)和脑梗塞体积的影响。
- Compared the occlusion of tanshinone preconditioning group and the ischemic preconditioning group with the occlusion of MCAO group, the former two groups had smaller infarct volume. 结果丹参酮预处理组和预缺血组梗死体积较MCAO组明显减少;
- RESULTS: Animals treated by decompressive craniectomy resulted in significantly smaller infarct volumes than those without craniectomy (P<0.01). 结果:手术组梗塞范围小于对照组(P<0.;01)。
- AbstractA repreducible medel of rat cortical infarction was developod in our study. Vve also explored the quantitative analysis method for infarct volume. 本文报告了大鼠皮层脑梗塞模型的制作和梗塞体积定量检测的方法。
- scintigraphic measurement of infarct volume (心肌)梗塞体积的闪烁照相测量