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- hypercapnia hypoxia 低氧高碳酸血症
- The hypoxia and hypercapnia ameliorated after thyroid hormone therapy. 甲状腺激素替代治疗后可改善低氧和高碳酸血症。
- Using this model, the responses of circulatory and respiratory systems under hypoxia and hypercapnia are discussed. 利用此仿真模型，仿真研究在低氧状态、高碳酸状态下，人体血液循环系统和呼吸系统的反应；
- An increase in ventilation during hypoxia and hypercapnia is a useful clinical sign and also a homeostatic response. 低氧和高碳酸血证时增加通气量是临床上有用的信号也是内环境稳态的反应。
- It was shown that during the acute stage of the disease hypoxia was usually present, but hypercapnia was only found in severe cases. 同时动态地观察了血液气体变化与临床心力衰竭及中毒性脑病的关系。
- In terms of anesthetic management, this means the avoidance of hypoxia, hypercapnia, endogenous catecholamine release, or Valsalva maneuvers (1). 从麻醉管理的角度，这意味着可避免出现低氧、高碳酸血症、内源性儿茶酚胺释放或出现瓦尔萨尔瓦动作[1]。
- Severe hypoxemia, hypercapnia, and metabolic acidosis with symptoms of target organ or tissue hypoxia may ensue if prompt therapy is not instituted. 如不及时开始治疗，便可发生严重的低氧血症、碱中毒、代谢性酸毒症伴靶器官或组织缺氧症状。
- Finally, it is important to recognize that the ventilatory response to hypercapnia and hypoxia is blunted in the elderly patient. 因此，认识到老年病人对高碳酸血症和低氧的通气反应迟钝是重要的。
- In terms of anesthetic management, this means the aoidance of hypoxia, hypercapnia, endogenous catecholamine release, or alsala maneuers (1). 从麻醉管理的角度，这意味着可避免出现低氧、高碳酸血症、内源性儿茶酚胺释放或出现瓦尔萨尔瓦动作[1]。
- It can prolong mouse s survival time in hypoxia. 醒神液能延长小鼠耐缺氧存活时间。
- Shock, sepsis, acidosis, hypoxia, and so forth, are treated. 对休克，感染，酸中毒，缺氧等应加以治疗。
- Hypoxemia and hypercapnia were connected with high coagulation. 低氧血症、高碳酸血症与高凝状态的形成密切相关。
- A pathological deficiency of oxygen, especially hypoxia. 组织缺氧病理性缺氧，尤指组织缺氧
- Locating the Impairment of Human Cognitive Function during Hypoxia. 急性缺氧条件下脑认知功能发生障碍的时段。
- Hypoxia was the main causeof swelling of mltochondria. 缺氧可导致肝线粒体肿胀。
- The results were as follows: (1) During hypercapnia (5% CO2) or hyper-capnia combined with hypoxia (10% O2), CBF increased in the control group (p<0.05, p<0.01) but was not altered in adapted group. 结果表明;高CO_2和低O_2高CO_2时;适应组CBF改变不明显;对照组CBF明显增加(p<0.;01)。
- The cells have reduced in size or been lost from hypoxia. 由于缺氧细胞已萎缩或消失。
- In contrast, the ECE-2 mRNA level was not affected by hypoxia. 缺氧前后ECE - 2的mRNA水平无明显差异。
- Mothed: the hypoxia bearing was observed in experimental mouse. 方法:对实验小鼠进行耐缺氧等实验观察。
- Objective: To predict the fetus intrauterine hypoxia. 摘要目的：预测胎儿宫内缺氧。
