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- glycyl bradykinin 甘氨酰缓激肽
- Bradykinin also sparks inflammation that swells blood vessels in the nasal passages, causing congestion. 舒缓激（月太）还会引发炎症，导致鼻腔的血管膨胀，引起鼻塞。
- The kallikrein system, which produces the potent vasodilator bradykinin, is beginning to be studied. 血管舒缓素系统产生强力的血管扩张物质缓激肽，有人正开始这种研究。
- PTD-kallikrein’s functional exertion maybe involved in the mediation of bradykinin B2 receptor. PTD-kallikrein的保护作用可能是通过B2受体的介导而发挥其功效的。
- Objective To study the clinical significance of urine Glycyl proline dipeptidyl aminopeptidase (GPDA) for diagnosis of earlyrenal injury in liver cirrhosis. 目的探讨应用尿甘氨酰脯氨酸二肽氨基肽酶(GPDA)诊断肝硬化早期肾损害的临床价值。
- RESULTS:The ADR induced by benazepril was associated with the aggregation of bradykinin in vivo. 结果:贝那普利所致不良反应主要表现为咳嗽、皮肤过敏反应等,其与体内缓激肽的聚集有关。
- Objective To study the clinical significance of detecting urine glycyl -proline dipeptidyl arninopeptidase (GPDA) at convalescence of acute glomerulonephritis. 目的探讨尿甘氨酰脯氨酸二肽氨基肽酶(GPDA)检测在急性肾炎恢复期的临床应用价值。
- Moreover, the enzyme hydrolyzed bovine low molecular mass kininogen and releasing bradykinin. 另外,此酶能够水解牛低分子量的激肽原,释放舒缓激肽。
- Objective To study the mechanism of selective opening blood tumor barrier by bradykinin in rat cerebral glioma model. 目的研究缓激肽选择性开放脑肿瘤的血肿瘤屏障的机制。
- The outcome is the generation of bradykinin, which increases vascular permeability and inflammation. 缓激肽作为产物，使血管通透性增加，炎症反应增强。
- Bradykinin powerfully stimulates nociceptors, and an antagonist that blocks its receptors would certainly prevent those receptors from activating nociceptors. 那麽，能阻断缓激肽受体的拮抗剂，必定可以防止这些受体造成的痛觉受器活化。
- Background: Angiotensin-converting enzyme (ACE) inhibitors increase the bioavailability of bradykinin, which induces cardiac PC. 背景：血管紧张素抑制剂依那普利能够增加缓激肽的生物利用度，缓激肽具有心肌缺血预适应保护作用。
- Bradykinin B2 receptor antagonist distinctly inhibits the effects of PTD-kallikrein on cerebral ischemia-reperfusion injury. PTD-kallikrein可以高效地通过生物膜及跨越血脑屏障且对细胞无明显毒性作用；
- Carcinoid flush cannot be reproduced by 5-HT infusion though bradykinin injection will produce a tolerably close imitation. HT的输注不能引起类癌性潮红，相反，舒缓激肽的注入则可得到较近似的阳性结果。
- Background: Angiotensin-conerting enzyme (ACE) inhibitors increase the bioaailability of bradykinin, which induces cardiac PC. 背景：血管紧张素抑制剂依那普利能够增加缓激肽的生物利用度，缓激肽具有心肌缺血预适应保护作用。
- Objective To investigate the mechanism of selective opening of blood tumor barrier by bradykinin and to find out its indication factors. 目的探讨缓激肽能选择性地诱发肿瘤血脑屏障开放的机理及影响此种开放的因素。
- The effects of bradykinin (BK) on the carotid baroreflex were examined in 36 anesthetized rats with isolated carotid sinus perfusion. 在36只隔离灌流左侧颈动脉窦区的麻醉大鼠上观察了缓激肽(bradykinin, BK)对颈动脉窦压力感受器反射的影响。
- Methods To determine whether the expression of bradykinin B2 receptor are on the vascular endothelial cell or cerebral glioma cell, double immunohistochemistry was used. 方法通过双重免疫组化染色,确定缓激肽B2受体是存在于血管内皮细胞上还是存在于肿瘤细胞上。
- Mission from the pressure of the wind has been isolated from high concentrations of histamine and bradykinin, but with anti-histamine and anti-drug therapy bradykinin. 从压力性风团中已分离出高浓度组胺和激肽，但用抗组胺和抗激肽药治疗无效。
- Results The results showed that bradykinin B2R did not express on vascular endothelia cell in both normal and tumor tissue, but highly expressed in tumor cell. 结果在正常脑组织和肿瘤的血管内皮细胞上未见缓激肽B2受体的表达,而在肿瘤细胞上发现了高水平表达的缓激肽B2受体。
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