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- glucuronyl epimerase 葡糖醛酸差向异构酶
- The mechanism of chloramphenicol toxicity is apparently failure of the newborn to conjugate chloramphenicol with glucuronic acid due to inadequate activity of hepatic glucuronyl transferase. 氯霉素中毒的机制显然是由于新生儿肝脏的葡萄糖醛酸转移酶活性不足,不能使氯霉素与葡萄糖醛酸结合。
- hereditary deficiency of glucuronyl transferase 遗传性葡萄糖醛酰基转移酶缺乏症
- UDP glucuronyl transferase deficiency UDP葡糖醛酸转移酶缺乏
- 3-hydroxyacyl coenzyme A epimerase 3-羟酰辅酶A表异构酶
- uridine diphosphate glucose epimerase 二磷酸尿苷葡糖表异构酶
- Uridine diphosphate galactose-4 epimerase deficiency 尿苷二磷酸半乳糖-4-表异构酶缺乏
- glucuronyl transferase 葡糖醛酸基转移酶
- glucuronyl 葡糖醛酸基转移酶
- glucuronyl group 葡糖醛酸基
- 3-Hydroxybutyryl-CoA epimerase 3-羟丁酰-CoA表异构酶
- 7.The mechanism of chloramphenicol toxicity is apparently failure of the newborn to conjugate chloramphenicol with glucuronic acid due to inadequate activity of hepatic glucuronyl transferase. 氯霉素中毒的机制显然是由于新生儿肝脏的葡萄糖醛酸转移酶活性不足,不能使氯霉素与葡萄糖醛酸结合。
- 4-Hydroxyproline epimerase 4-羟脯氨酸表异构酶
- epimerasen. [生化]表异构酶;差向(异构)酶
- 9.The mechanism of chloramphenicol toxicity is apparently failure of the newborn to conjugate chloramphenicol with glucuronic acid due to inadequate activity of hepatic glucuronyl transferase. 氯霉素中毒的机制显然是由于新生儿肝脏的葡萄糖醛酸转移酶活性不足,不能使氯霉素与葡萄糖醛酸结合。
- 6.The mechanism of chloramphenicol toxicity is apparently failure of the newborn to conjugate chloramphenicol with glucuronic acid due to inadequate activity of hepatic glucuronyl transferase. 氯霉素中毒的机制显然是由于新生儿肝脏的葡萄糖醛酸转移酶活性不足,不能使氯霉素与葡萄糖醛酸结合。
- epimerase deficiency 表异构酶缺乏
- ribulose phosphate epimerase 磷酸核酮糖差向异构酶
- acylglucosamine epimerase 酰基葡萄糖胺表异构酶
- pentose epimerase 戊糖表异构酶