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- Methods Focal ischemic models of middle cerebral artery occlusion (MCAO) were established by inserting nylon monofilament through the right common carotid artery into the anterior cerebral artery. 方法经右侧颈总动脉将尼龙单丝线栓至大脑中动脉造成永久性缺血模型。
- focal ischemic model 局灶性脑缺血模型
- Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。
- Permanent focal cerebral ischemia model was established and the HR and MABP of rats were recorded. 假手术组除不向颈内动脉插入尼龙线外其余步骤与缺血组均相同。
- Methods: In different time, the concentrations of CINC and the activities ofmyeloperioxidase (MPO)were observed in rats with focal cerebral ischemic model rats made by thread embolismmethod. 方法：用线栓法制备大鼠局灶性脑缺血模型，观察不同脑缺血时程血浆（sera）和脑组织内CINC浓度与髓质过氧化物酶（MPO）活性。
- Focal ischemia also could raise the neurogenesis in hippocampus dentate gyrus. 局灶性脑缺血后亦可增加海马齿状回内神经发生。
- Objective:To study the influence of reperfusion after ischemia for different time on cerebral infarction volume and neurological function of the focal ischemia reperfusion model in rats. 目的:研究不同缺血时间对大鼠局灶性脑缺血再灌注模型脑梗塞灶体积及神经功能缺损的影响。
- The focal cerebral ischemia model was produced by occluding middle cerebral artery(MCA) for 2h and reperfusing 24h. Cell apoptosis was detected by Tunel,the expression of bcl-2 and bax were analyzed by immunohistochemical. 线栓法制备大鼠大脑中动脉阻塞模型，缺血2h再灌注24h后观察PAMD对脑缺血后细胞凋亡及相关基因bcl-2和bax表达的影响，Tunel法测细胞凋亡，免疫组化法观察bcl-2和bax的表达。
- Objective: To investigate the effect of insulin on c fos gene expression in focal ischemic rat brain. 目的：观察胰岛素对局灶缺血脑组织c?fos基因表达的影响。
- The abnormal cells of hippocampal Cornu Ammon second area (CA2) could be observed 5d after ischemia and markedly increase at 8d, these meant that the transient focal cerebral ischemia model was established successfully. 在5d组，海马CA2区开始有细胞异常，8d后，异常细胞明显增多。 表明短暂性局灶性脑缺血模型制作成功。
- Objective To explore involvement of the Nogo-A/NgR pathway in neuroprotective effect of ischemic preconditioning (IPC) on focal ischemia in rats. 目的研究Nogo-A/NgR通路参与缺血预处理(IPC)对局灶性脑缺血的神经保护作用。
- Methods: with the acute cardiac ischemia model induced by pituitrin in rats. 方法:采用垂体后叶素诱发大鼠急性心肌缺血模型观察药物的作用。
- The left hindlimb ischemia model was made in non-treatment and treatment group. 非治疗组不做负压治疗。
- Non-Invasive Assessment of Vasoconstrictive Effects of Hyperoxygenation in Focal Ischemia. 局部缺血中氧合过度血管收缩效果中的非侵入性评价。
- Objective To study the regularity of expression of Caspase activated DNase (CAD) gene in ischemic penumbra after rat focal ischemic middle cerebral artery occlusion/reperfusion. 目的 探讨大鼠局灶性脑缺血再灌注后缺血半暗带Caspase 3激活的DNA酶 (CAD)基因的表达变化规律。
- Endogenous VEGF Expression Related to Collateral Vessels Formation in Rabbit Ischemic Model. 兔缺血后肢内源性VEGF蛋白表达与侧支形成相关性研究。
- Betz AL, Coester H C. Effect of Steroids on Edema and Sodium Uptake of the Brain During Focal Ischemia in Rats[J].Stroke,1990,21:1199. 卞留贯;张天锡;赵卫同;等.;脑缺血早期脑水肿与脑微血管及突触膜钠钾三磷酸腺苷酶活性变化[J]
- NDI can revive the neurologic impairment which was induced by the total cerebral ischemic model. 因此得出,NDI对全脑缺血再灌注模型所引起的神经功能缺损的恢复具有较好的疗效。
- Thus, overexpression of the glucose transporter gene via the introduction of a herpes simplex viral vector into the nervous system protected neurons from focal ischemic injury (64). 这样，葡萄糖载体基因的过度表达通过疱疹单一腺病毒载体进入到神经系统，保护神经元不受局部缺血损伤（64）。
- A cerebral ischemic model of SD mice developed by ferric chloride was applied to test the thrombolysis effect of non-ephedrine alkaloids part. 结果非麻黄碱部分具有与麻黄碱部分及盐酸麻黄碱对照品溶液相当的溶栓作用。
