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- CD11b was expressed in 2 cases of chronic phase CML, but not in 2 cases in blastic phase. CD11b在2例慢性期CML患者中表达 ,而在2例急变期患者中不表达。
- In group STE,SD/chord was not significantly improved both in acute and chronic phase. ST段抬高组 ,SD/chord在急性期和慢性期未见明显改善。
- Out of 9 cases of chronic phase of CML, 8 showed positive expression of bcr/abl gene which accounted for 88. 9% . 9例CML患者中;8例bcr/abl基因阳性;占88.;9%25。
- The WT1 expression level of chronic myeloid leukemia(CML) in blast crisis was higher than that of CML in chronic phase. 慢性髓性白血病(CML)急变期患者WT1基因表达水平显著高于慢性期。
- The transplantation effective rate was 88.8%as counting by ANLL CR1 and CGL chronic phase. 按急性白血病CR1和慢粒白血病慢性期计算总有效率88.;8%25(8/9例)。
- Most patients being treated in the chronic phase of CML can go about their day-to-day activities. 在慢性期治疗过程中，大多数病人仍可以进行他们日常的活动。
- Methods : We have chosen 37 CML cases, including 27 chronic phase (CP), 5 accelerated phase (AP) and 5 blast crisis (BC) cases . 方法:选取37例CML病人骨髓及口腔黏膜细胞的DNA标本,其中慢性期(chronic phase,CP)27例,加速期(accelerated phase,AP)5例,急变期(blast crisis,BC)5例。
- The pre-BMT situations were, 91 cases in first chronic phase (CP1), 19 in accelerating phase (AP), 2 in blast crisis (BC) and 6 in advanced CP (>CP2). 118例患者中慢性期91例，加速期19例，急变期2例，第2次以上慢性期6例。
- Methods:Bone marrow mononuclear cells(MNCs) from 10 newly diagnosed Ph chromosome positive CML patients in chronic phase were separated and cultured in vitro for 10 days. 方法 :分离 10例初诊的慢性期Ph+ CML患者骨髓单个核细胞 (MNC) ,体外培养 10天 ,利用染色体分析法检测培养前后Ph+ 中期细胞比例 ;
- The WT1 positive rate in 15 CML patients was 46.7%, the positive rate in 6 patients at blast crisis and 1 patient at accelerated phase were 100%, 0 in 8 patients at chronic phase. 15例CML的WT1基因表达阳性率为46.;7%25，其中6例急变期和1例加速期阳性率为100%25，8例慢性期阳性率为0。
- GFAP was observed only in PAG at subacute and chronic phases of inflammation. 星形胶质细胞标记物GFAP在急性期和慢性期有显著增加；
- CML typically starts with an indolent chronic phase followed by an aggressive myeloid or lymphoid blast crisis phase, which is dependent on the constitutive kinase activity of the BCR-ABL oncoprotein. 依赖于BCR-ABL融和基因表达的激酶活性，CML患者通常有一个稳定的慢性期随后进入髓变或淋变的加速期。
- WT1 mRNA expression was revealed in K562,HL?60 and TF?1 cell lines, 21 of 25 (84%) AML patients,8 of 11 (73%) ALL patients,2 CML patients in blast crisis and 1 of 7 CML patients in accelerated and chronic phase. K562、HL－60及TF－1三株白血病细胞系均高度表达WT1，而U937细胞系未测到WT1。 急性髓系白血病（AML）25例中21例、急性淋巴细胞白血病（ALL）11例中8例、慢性粒细胞白血病（CML）急性变者2例、慢性及加速期CML患者7例中1例WT1表达阳性。
- In 39 CML patients, WT1 mRNA was detectable in 8 of 21 (38.1%) Chronic Phase and 17 of 17 (100%) Acceleration Phase and Blast Crisis, WT1 mRNA was undetected in 1 post-Allo-BMT patient. 39例慢性粒细胞白血病(CML)中慢性期21例有8例阳性(38.;1%25)，加速期和急变期17例均为阳性(100%25)，1例异基因骨髓移植后WT1 mRNA持续为阴性。
- METHODS: Polymerase chain reaction (PCR) was used to detect FLT3 gen e expression, and FLT3/ITD gene mutation in 53 CML patients of chronic phase, an d 34 CML patients of accelerated phase or blast crisis. 方法:采用聚合酶链反应(polymerasechainreaction,PCR)检测53例CML慢性期和34例CML加速期或急变期患者DNA水平FLT3基因及FLT3/ITD基因突变。
- The expression rates of CD 34 and HLA DR were 3.6% and 31% in the chronic phase, 36% and 73% in acceleration phase, 62% and 100% in blastic phase, respectively. Both CD 34 and HLA DR were significantly higher in blastic phase than chronic phase (P<0.05). CML慢性期、加速期、急变期 CD34 及 HL A- DR抗原表达阳性率分别为 3.;6%25和 31%25;36%25和 73%25;6 2%25和 10 0%25;慢性期与急变期相比差异有统计学意义 ( P <0
- Background and objective:The natural disease history of chronic myeloid leukemia (CML) typicallyevolves 3 distinct clinical stages: chronic phase(CP), accelerated phase and blast crisis. 慢性粒细胞白血病(Chronic myeloid leukemia，CML)的自然病程可分为慢性期(Chronic phase，CP)，加速期(Accelerated phase，CP)和急变期(Blastcrisis phase，BP)。
- Methods An 18 years old patient (body weight 75 kg) with chronic myelogenous leukemia in first chronic phase received HLA identical sibling CBT after conditioning with modified busulfan/cyclophosphamide (Bu/CTX) regimen. 方法 1例 18岁体重 75kg的慢性髓系白血病慢性期 (CML CP)患者在用改良的马利兰、环磷酰胺 (Bu CTX)方案预处理后给予HLA相合同胞CBT。
- Methods Five CML patients,2 in chronic phase(CP),1 in accelerated phase(AP) and 2 in blast crisis(BC), all comfirmed the presence of b3a2 bcr/abl mRNA by RT PCR(reverse transcriptase polymerase chain reaction). 方法 5例CML中慢性期 2例 ,加速期 1例 ,急变期 2例 ,均具b3a2型bcr/ablmRNA。
- Methods Polymerase chain reaction(PCR)was used to detect FLT3 gene expression in 103 patients of AML cases, 63 ALL, 53 CML in chronic phase, 31 CML in blast crisis, 11 CLL, 36 MDS, 9 MM and 13 NHL with marrow infiltration. 方法采用多聚酶链反应(PCR)方法检测103例初治AML、76例急性淋巴细胞白血病(ALL)、53例慢性粒细胞白血病(CML)慢性期、31例CML急变期、11例慢性淋巴细胞白血病(CLL)、36骨髓增生异常综合征(MDS)患者、9例多发性骨髓瘤(MM)及13例伴有骨髓侵犯非霍奇金淋巴瘤(NHL)患者FLT3基因表达情况。
