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- Both Brain ischemic preprocessing and postprocessing can result in neuroprotective effect by inducing brain ischemic tolerance. 脑缺血预处理及后处理均可以诱导脑缺血耐受形成而产生神经保护作用。
- The mechanism of brain ischemic tolerance (BIT) induced by cerebral ischemic preconditioning (CIP) involves cascades of events including release of neurotransmitter, activation of receptors and gene expression. 脑缺血预处理(cerebral ischemic preconditioning,CIP)诱导的脑缺血耐受(brain ischemic tolerance,BIT)的产生涉及神经介质、受体及基因的表达等一系列过程。
- Brain Ischemic Tolerance Induced by Spreading Depression in Rats in Vivo 扩散性抑制诱导大鼠皮层缺血耐受的在体研究
- brain ischemic tolerance 脑缺血耐受
- IPC induces delay ischemic tolerance possibly via a partial involvement of the Nogo-A/NgR pathway in the brain. Nogo-A/NgR通路可能参与IPC诱导的脑缺血耐受,保护脑缺血性损伤。
- Methods Focal brain ischemia animal model was used. 方法应用局限性脑缺血动物模型。
- Abstract: Brain ischemic preconditioning is that transient ischemia in sublethal dose will result in tolerance to the following ischemia in lethal dose for long time. 摘要: 脑缺血预处理即给予短暂亚致死量缺血可对随后的长时间致死性缺血损伤产生耐受。
- In contrast,no apparent changes in BK channel open probability were observed in ischemic tolerance group. 而对 CA区锥体神经元具有明显保护作用的缺血耐受组 BK通道的开放概率与正常对照组无明显变化。
- CDP-choline improved the recoveryof SEPs caused by brain ischemia obviously. CDP-choline可明显促进缺血脑功能的恢复。
- AIM:To observe the relation between astroglial activation state and ischemic tolerance in the gerbil hippocampus. 目的:观察海马区星形胶质细胞的活化状态与缺血耐受性的关系。
- Objective: To observe the cerebral ischemic tolerance induced by PDS preconditioning and its effect on expression of GFAP. 目的:观察三七二醇皂甙(PDS)预处理诱导脑缺血耐受及对胶质纤维酸性蛋白(GFAP)表达水平的影响。
- Induction of Cerebral Ischemic Tolerance by Erythromycin Preconditioning Reprograms the Transcriptional Response to Ischemia and Suppresses Inflammation. 红霉素预处理诱导的脑缺血耐受重编对缺血转录反应并抑制炎症反应。
- DWI is sensitive to early ischemic changes,which can find the changes of brain ischemia earlier than that of routine T2WI. DWI对早期缺血改变非常敏感,DWI较常规T2加权图像能更早地发现脑缺血的改变。
- Based on rabbit MCAO focal brain ischemic model, we studied the effect of NO on ischemic brain by using L-Arginine, a precursor of NO and NOS inhibitor L-NNA. 本文在兔MCAO局灶脑缺血模型基础上,利用外源性一氧化氮(NO)前体物质L-精氨酸和NOS抑制剂L-NNA研究NO对脑缺血后脑水肿和脑梗塞的影响。
- But the detailed mechanisms of G-LOC or G induced brain ischemia as well as protective effect IPC were not clear. 然而不论大G昏迷、G力造成的脑损伤及缺血前制约效应的保护机转都并不清楚。
- It can cause severe complications, such as brain ischemia or cervical hematorrhea incurred by rupture of aneurysm. 它可以引起严重的并发症,比如大脑局部缺血或动脉瘤破裂产生的颈部软组织出血。
- It has been reported that ischemic tolerance (IT) might be related to excitatory amino acid(EAA), adenosine, immediate early gene(IEG), heat shock protein(HSP) and oncogene B cell lymphoma-2(Bcl-2), etc. 目前脑IT的确切机制尚不清楚,认为可能与兴奋性氨基酸(exicitatory amino acid,EAA)、腺苷、即早基因(immediate early gene,IEG)、热休克蛋白(heat shock protein,HSP)及原癌基因B细胞淋巴瘤-2(B cell lymphoma-2,Bc1-2)等因素有关。
- It was suggested that DDPH had some protective effect on acute brain ischemia and ischemia-reperfusion. 结果提示,DDPH对动物脑缺血具有一定的保护作用。
- Objective To study the improvement effects of GM_1 on brain ischemia reperfusion(BIR) induced memory impairment in mice. 目的探讨脑缺血复灌后连续应用GM1对改善记忆障碍的疗效。
- Disturbances of calcium homeostasis and modification of ca~(2+) pump(Ca~(22+),Mg~(2+)-ATPase)in the rabbit focal brain ischemia. 脑缺血时钙稳态与钙泵(Ca~(2+),Mg~(2+)-ATP酶)活性变化