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- In rats dependence to flurazepam, hippocampal nNOS increased but without statistical significance. 而對氟西泮抗癇依賴組則與對照組差別無統計學意義。
- Conclusion NO may play a role in rats tolerance and dependence to the anticonvulsant action of flurazepam. 結論NO可能是介導氟西泮抗癇耐受性和依賴性的因素之一。
- Method Animal models of tolerance and dependence to flurazepam were established. 方法建立大鼠對氟西泮抗癇耐受性和依賴性的模型。
- Total NOS activity increased dramatically both in flurazepam tolerane and dependene rats. 兩組NOS活性均顯著高於各自的對照組。
- Aim To study the role of nitric oxide by measuring the expression and activity of nitric oxide synthase in hippocampus of rats tolerance and dependence to the anticonvulsant action of flurazepam. 目的通過測定大鼠對氟西泮抗癇耐受性和依賴性時海馬中一氧化氮合酶(NOS)蛋白表達及活性的變化,探討一氧化氮(NO)在此過程中可能的作用。方法建立大鼠對氟西泮抗癇耐受性和依賴性的模型。
- Keywords FLZ;Ginkgo biloba extract;oxidized low density lipoprotein;macrophage;endothelial cell;apoptosis; 銀杏葉提取物;氧化低密度脂蛋白;巨噬細胞;內皮細胞;凋亡;
- Keywords flurazepam;tolerance;dependence;nitric oxide(NO);nitric oxide synthase(NOS); 氟西泮;耐受性;依賴性;一氧化氮(NO);一氧化氮合酶(NOS);
- 5. Conclusion NO may play a role in rats tolerance and dependence to the anticonvulsant action of flurazepam. 結論NO可能是介導氟西泮抗癇耐受性和依賴性的因素之一。
- Molecular mechanism of the tolerance to flurazepam and reversal of tolerance with flumazenil in genetically epilepsy-prone rats 氟馬西尼逆轉驚厥易感大鼠氟西泮耐受性的分子機制
- Changes of nitric oxide synthase in hippocampus of rats tolerant to and dependent on the anticonvulsant action of flurazepam 大鼠對氟西泮抗癇耐受和依賴時海馬中一氧化氮合酶的變化
- Modification of GABAA receptor subunit mRNA content in audiogenic seizure rat cortex and hippocampus following tolerance to flurazepam 氟西泮耐受聽源性驚厥大鼠腦皮質運動區和海馬區GABAA受體亞單位mRNA含量的變化
- FLZ (=flurazepam) 氟胺安定
- Poisoning by flurazepam 氟胺安定中毒
- Flurazepam Dihydrochloride 鹽酸氟胺安定
- flurazepam hydrochloride 鹽酸氟西泮, 鹽酸氟安定, 鹽酸氟胺安定
- Flurazepam measurement 氟胺安定測量
- Flurazepam Monohydrochloride 鹽酸氟胺安定
- flurazepamn. [葯]氟胺安定;氟二乙氨乙基安定(安眠藥)