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- PGD synthase 前列腺素D2合成酶
- Synthase polymorphism has become a focus of study. 合酶基因多态性已成为研究的热点。
- Adds profile data from one or more. Pgc files to the. Pgd file. 将配置文件数据从一个或多个.;pgc文件添加到
- One or more. Pgc files whose profile data you want to merge into the. Pgd file. 一个或多个.;pgc文件;需要将其配置文件数据合并到
- The. Pgd file into which you are merging data from the. Pgc file or files. 将来自.;pgc文件的数据合并到其中的
- A technique already in use is called preimplantation genetic diagnosis (PGD). 目前已经应用的一项技术叫做植入前基因诊断(PGD)。
- Effect of iodine on the activity of nitric oxide synthase in rat hippocampus. 高碘对大鼠海马一氧化氮合酶蛋白表达的影响。
- The science of PGD has steadily expanded its scope, often in contentious ways. PGD的研究正逐步拓展其领域,通常是以令人非议的方法。
- Chitin synthase is the important enzyme that converts UDP-GlcNAc to chitin. 摘要几丁质合成酶是生物合成几丁质的关键物质。
- ATP is produced by a complex in the mitochondrial membrane called ATP synthase. 三磷酸腺苷是由一个复杂的线粒体膜称为ATP合酶。
- It is useful for discovery of novel fungal polyketide synthase gene clusters. 本文综述了近五年来建立的几种分离真菌聚酮合酶基因的方法。
- The gradient this creates produces ATP by a similar ATP synthase complex. 这造成的梯度产生ATP的一个类似的ATP合酶复杂。
- Allele oxide synthase (AOS) was a key enzyme in JA biosynthesis pathway. 丙二烯氧化物合成酶基因(allele oxide sysnthase,AOS)是JA生物合成的关键酶基因,也是影响JA信号途径的调控基因。
- Chitin synthase is the important enzyme that convert UDP-GlcNAc to chitin. 几丁质合成酶是生物合成几丁质的关键物质。
- The AfSS gene of sucrose synthase in the Medicago falcata L was cloned. 并克隆与黄花苜蓿 (Medicago falcata L.;) 耐冷性相关的蔗糖合酶基因(MfSS)。
- A model used PGA, PGV and PGD for calibrating seismic response spectrum was introduced. 介绍了用地面峰值加速度(PGA)、峰值速度(PGV)和峰值位移(PGD)三个参数标定设计地震反应谱的模型。
- The citrate synthase of mammalian tissues have been shown to be inhibited by ATP. 有人已经证实哺乳动物组织的柠檬酸合成酶受ATP的抑制。
- In the following example, the profile data in myapp1. Pgc was added to the. Pgd file 3 times. 在下面的示例中;将myapp1.;pgc中的配置文件数据添加到
- If you do not specify any. Pgc files, pgomgr will merge all. Pgc files whose filenames are the same as the. Pgd file. 如果不指定任何.;pgc文件;pgomgr将合并与
- Results also indicate that metallothionein 3 was over expressed in lungs that didn't develop PGD. 这些结果同时还提示肺内金属硫蛋白3过表达并不会发展为PGD。