Objective The current study was designed to investigate the effect and mechanism of apoptosis of HL-60 cells induced by 131I-GMCSF, and to estimate the value of targeted radiopharmaceutic therapy in patients with acute myeloblastic leukemia (AML).

• 目的 观察131I-GMCSF诱导HL-60细胞凋亡及其机制，探讨其在急性髓性白血病放射导向治疗中的作用。