Objective In order to study the role of human complement regulatory protein (CRPs) transgene in including DAF, CD59 and MCP in overcoming hyperacute rejection (HAR).

• 目的 研究转人补体调节蛋白 (CRP)DAF、MCP和CD5 9基因对抑制人补体激活从而克服超急性排斥反应的作用。