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- BackgroundCerebral ischemia reperfusion damage is a kind of delayed neuronal death ( DND). A lot of investigations have showed that DND is closely related to apoptosis. 脑缺血再灌注损伤是一种迟发性神经元死亡(delayed neuronal death,DND)，大量研究表明DND与细胞凋亡(cell apoptosis,CA)密切相关。
- Cerebral ischemia reperfusion damage 脑缺血再灌注损伤
- Conclusion BDE may have protect effect on cerebral ischemic reperfusion damage by decreasing Nos activity. 结论BDE抑制神经元NOS活性，对脑缺血再灌流损伤具有脑保护作用。
- Objective To observe the effects of Buyanghuanwu Decoction Extract (BDE) on nitric oxide synthase (NOS) activity in cerebral ischemic reperfusion damage. 目的观察补阳还五汤提取物(BDE)对脑缺血再灌流后一氧化氮合酶(NOS)活性影响。
- Mao XP, Chen XF, Zhang L, et al.Influences of Buyanghuanwu decoction extract on nitric oxide synthase activeity in cerebral ischemic reperfusion damage in rats[J].Stroke Nerv Dis, 2001, 8(2): 103-4. [2]毛小平;陈秀芳;张玲;等.;补阳还五汤提取物对脑缺血再灌流后一氧化氮合酶活性的影响[J]
- Keywords cerebral hypoxia;cerebral ischemia reperfusion damage;memantine;milkvetch;neonatal rats;pathology; 脑缺氧;脑缺血;再灌注损伤;黄芪;美金胺;大鼠;新生;病理;
- Ischemia reperfusion damage 缺血再灌注损伤
- OBJECTIVE To investigate the protective mechanism of adenosine in renal damage of ischemia reperfusion . 探讨腺苷对肾脏缺血再灌注损伤的保护作用机制。
- CONCLUSION DK can protect myocardium against ischemia reperfusion injury. 结论dk对大鼠心肌缺血再灌注损伤有保护作用。
- Experimental study on changes of hippocampus EAA, MCa, CaM contents on rat global cerebral ischemic reperfusion damage 全脑缺血再灌注兴奋性氨基酸、线粒体钙、钙调素含量的变化
- NGF could ease the delayed neuron death (DND) after ischemia reperfusion. NGF可以减轻海马迟发性神经细胞死亡 (DND)性损伤。
- Conclusion: Ischemia reperfusion could induce the apoptosis of the cell. 说明缺血再灌注损伤能诱发细胞凋亡。
- Conclusion: The rise of CINC was an effect of activiting WBC after the ischemia or ischemia reperfusion and indicated that the change was involving in the damage of ischemia reperfusion. CINC水平增高始于再灌注1h;再灌注3h时达高峰;为0.;65±0
- Objective To investigate the protective mechanism of flunarizine in gerbil brain after ischemia reperfusion. 目的进一步探讨盐酸氟桂嗪对缺血再灌注后神经细胞保护作用的机制。
- Study on the Effect of Hepatic Glycogen Content on Liver Ischemia Reperfusion Injury and Its Mechanism in Rabbits. 肝细胞糖原拮抗肝脏热缺血再灌注损伤相关机理的研究。
- Objective:Study the effect of acusector on serum CK and LDH of cerebral ischemia reperfusion injury model in rats. 目的:研究电针对实验性脑缺血再灌注损伤大鼠血清中肌酸激酶(CK)和乳酸脱氢酶(LDH)的影响。
- The results suggested that CGS has a protective effect on cerebral ischemia reperfusion injury in rats. 表明CGS对大鼠脑缺血再灌注损伤具有保护作用。
- The retina is very easy to be subjected to ischemia reperfusion injury (IRI) because of its special vascular supply. 视网膜血供特点十分特殊，易致缺血再灌注损伤(ischemia-reperfusion injury,IRI)。
- Background: Hepatic ischemia reperfusion (HIR) injury is a common acute disease of surgery. 肝脏缺血再灌注损伤(Hepatic ischemia reperfusion injury，HIR)是指肝脏缺血后重获血流灌注或氧供后，肝脏损伤会在缺血缺氧损伤的基础上进一步加重。
- Ischemia reperfusion injury (IRI) is a common clinical pathological process, and a key step in acute organ failure. 缺血再灌注损伤(ischemia reperfusion injury，IRI)是临床常见的病理过程，为急性器官功能衰竭的重要发病环节。
