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- The level of androgen had no relation with endometrium hyperplasia. 雄激素水平的高低与子宫内膜增生无关。
- Objective: To evaluate the effects of levonorgestrol-releasing intrauterine system (LNG-IUS) for treatment of endometrium hyperplasia. 子宫内膜病理示子宫内膜增生过长改变全部消失,内膜呈轻度分泌现象及。
- The expression of FHIT in adenocarcinoma endometrium was lower than in endometrial hyperplasia(P<0.01). 在子宫内膜各种增生性病变阳性表达高于内膜腺癌(P<0.;01)。
- Objective: To investigate the expression and clinical significance of FHIT in endometrial hyperplasia and adenocarcinoma of endometrium. 摘要目的:探讨FHIT在子宫内膜增生性病变及子宫内膜腺癌中表达的临床意义。
- C. Etiology. Exposure of the endometrium to continuous estrogen unopposed by progesterone can lead to endometrial hyperplasia. 病因学:子宫内膜长期暴露于雌激素的作用下而没有孕激素的拮抗从而导致内膜增生过长。
- Endometrial hyperplasia is defined as proliferation of endometrial glands resulting in a greater gland-to-stroma ratio than normal endometrium. 子宫内膜增生过长是由于子宫内膜腺体增生,导致腺体与间质的比例比正常内膜增加引起的疾病。
- Objective Explore the function of Shaofu Zhuyu Tang on endometrial hyperplasia. 目的探讨少腹逐瘀汤治疗子宫内膜增殖症的作用。
- Objective To study the relationship between endometrial hyperplasia and the expression of proliferative cell antigen in carcinoma of endometrium and its characteristic. 目的探讨子宫内膜增生与内膜癌增殖细胞核抗原表达计数相关性,及其对判断生物学行为及预后的意义。
- Methods: The expression of FHIT was studied by using Envision immunohistochemisty in 60 cases of endometrial hyperplasia and 60 cases adenocarcinoma of endometrium. 方法:采用免疫组化方法检测子宫内膜增生性病变60例及子宫内膜腺癌60例中FHIT的表达情况。
- Results: Of 196 cases,endometrial hyperplasia was found in 100,irregular shedding of endometrium in 8,decidual tissue in 16,organic lesion of endometrium. 结果:196例病人中,子宫内膜增生症100例,子宫内膜不规则脱卸8例,蜕膜样组织16例,子宫内膜器质性病变10例。
- Methods The expression of aromatase cytochrome P450 in normal endometrium,endometrial hyperplasia and endometrial cancer were detected by immunohistochemical analysis. 方法应用免疫组织化学方法检测正常子宫内膜、子宫内膜增生症及子宫内膜癌中细胞色素芳香化酶P450的表达。
- Results The microvascular density(MVD) in tumour increased gradually from normal endometrium to endometrial hyperplasia and to endometrial carcinoma(P<0.01). 结果从正常子宫内膜、增生过长到内膜癌组织内微血管密度(MVD)逐渐增多(P<0.;01)。
- Conclusion MYD in endometrial hyperplasia increases.MVD can be used as a prognostic factor. 结论子宫内膜增生过长组织中血管生成增多,MVD与子宫内膜癌临床预后有关。
- Endometrial hyperplasia usually results from chronic estrogen stimulation unopposed by progesterone. 子宫内膜增生过长经常是由于长期无孕激素拮抗的雌激素作用所引起。
- Exogenous estrogen. Continuous exposure to unopposed estrogen results in endometrial hyperplasia. 外源性雌激素:长期暴露与无拮抗的雌激素环境下可导致内膜增生过长。
- Method: Analysis on 60 cases with endometrial hyperplasia symptom through transvaginal ultrasonography inspection. 方法:对60例经阴道超声检查发现子宫内膜增生的病例进行分析。
- Endometrial hyperplasia with atypia is a premalignant condition, preferably treated with hysterectomy. 内膜增生过长伴有不典型增生属于癌前病变,行子宫切除术更为适合。
- Conclusion It has good curative effect to treat endometrial hyperplasia with Shaofu Zhuyu Tang. 结论少腹逐瘀汤治疗子宫内膜增殖症有较好疗效。
- Objective: To evaluate the diagnostic value of transvaginal ultrasonography in the detection of endometrial hyperplasia. 目的:探讨经阴道超声检测子宫内膜增生的诊断价值。
- Baseline biopsies revealed endometrial hyperplasia in 21 women who had taken sequential HRT and in one who had taken unopposed estrogen. 21名连续HRT治疗的妇女和1名采用非对抗性雌激素疗法的妇女活组织检查显示有子宫内膜增生。