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- KGF and KGFR are expressed in Hela cells. Hela细胞中有KGF和KGFR的表达;
- Hela cells were also injected to normal BALB/c and Kunming mice. 同时用Hela细胞作为阳性肿瘤细胞对照,并接种于免疫功能正常的BALB/c小鼠和昆明种小鼠。
- HPV16 E7 was expressed efficiently in HEK-293 and HeLa cells. 荧光显微镜下,转染后HEK-293细胞和HeLa细胞表达绿色荧光蛋白。
- Either the cell growth inhibitory rate or the colony inhibitory rate of the HeLa cells was 100%(P<0.01) after being exposed to 12 Gy irradiation, meanwhile, the value of 2,3-DPG was nearly unchanged(P>0.05). 12 Gy的辐射剂量能够完全抑制肿瘤细胞的增生(细胞生长抑制率及克隆形成抑制率均为100%25;P<0.;01);且不影响红细胞的携氧功能(不辐射与辐射后的2;3 DPG值无显著性差异;P>0
- Methods: (1)To detect toxic effect of PAP to Hela cell by MTT assay. 方法:(1)用MTT法检测PAP对Hela细胞的毒性作用;
- RT-PCR showed that E1A gene had a stable transfection in Hela cells and obviously down-regulated Her-2 expression. RT-PCR结果显示:E1A基因在Hela细胞中能稳定表达,E1A基因的转染明显降低了Her-2基因在Hela细胞中的表达水平;
- Objective: To study the fate of LAK cell and rescuing effect of Astragalus polysaccharide (APS) during and following assault on Hela cells. 目的:进一步探讨LAK细胞攻击Hela细胞后的归宿及黄芪多糖(APS)的影响。
- Low dose cisplatin can enhance the sensitivity of radiotherapy through change cell cycle of Hela cell lines. 小剂量顺铂可通过改变培养的人Hela细胞周期 ,诱导细胞G2 /M期延迟从而提高放疗敏感性 ,可能与其内在的 p5 3基因表达改变有关。
- At 12 h, the H_2O_2 level in HeLa cells and AsPC-1 cells were similar to that of the control group. 结论:As2O3诱导HeLa细胞和AsPC-1细胞凋亡与细胞内的H2O2水平改变有关,而与HPV的感染无明显相关性。
- The s-lap/GFP fusion protein can express in Hela cells and locate in entire cells, but express highly in the nucleus. s- lap/ GFP融合蛋白可在人 Hela细胞中表达 ,并主要定位于细胞核。
- Flow cytometry (FCM) was used to examine the effects of 4-HPR with or without DDP on cell cycle and apoptosis of HeLa cells. 采用流式细胞仪测定4-HPR、DDP、4-HPR+DDP对HeLa肿瘤细胞系细胞增殖周期和细胞凋亡的变化。
- The CPE inhibition assay was used to test the antiviral activity of GD4 on RSV in Hela cells. 采用细胞病变抑制实验,观察GD4在Hela细胞中对RSV的抑制作用。
- G418-resistant clones of Hela cells were obtained and confirmed as positive by RT-PCR. 重组质粒转染Hela细胞后,经G418筛选成功获得阳性克隆。
- Aim To evaluate the manner and molecular mechanism of HeLa cell death induced by Coxsackie virus B3(CVB3). 目的评价柯萨奇病毒B3CVB3致HeLa细胞死亡的方式及分子机制。
- Gpd-Tp92 fusion gene constructed in pcDNA3.1 (+) vector could express a fusion protein in Hela cells. pcDNA3.;1(+)/Gpd-Tp92在HeLa细胞中能有效表达。
- Both genes were cloned into GFP expression vector pIRES2 EGFP and transfected into HeLa cells. 将上述基因克隆入绿色荧光蛋白(GFP)表达载体pIRES2-EGFP中,转染人HeLa细胞,在荧光显微镜和电镜下观察转染细胞的形态和结构。
- The KGF and KGFR genes were expressed in the Hela cells. The KGF and KGFR proteins were expressed in the Hela cells. 从基因水平和蛋白水平证实KGF及KGFR在Hela细胞中有表达。
- The augment of G 1 ratio in the proliferation cycle of Hela cells presents a concentration-dependent manner. 细胞周期分析显示槲皮素组 G 1 期细胞比例明显增多,呈量-效关系。
- Role of Mitochondria in HeLa Cell Apoptosis Induced by Hematoporphyrin monomethyl ether- PDT[J]. 引用该论文 丁新民;刘凡光;顾瑛;曾晶;戴维德;李晓松.
- Stably transfected and control Hela cell lines were injected subcutaneously into female Balb/c/nu mice. 稳定转染Ang-2的细胞注射Balb/c裸鼠皮下。