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- Abstract: Brain ischemic preconditioning is that transient ischemia in sublethal dose will result in tolerance to the following ischemia in lethal dose for long time. 摘要: 脑缺血预处理即给予短暂亚致死量缺血可对随后的长时间致死性缺血损伤产生耐受。
- The mechanism of brain ischemic tolerance (BIT) induced by cerebral ischemic preconditioning (CIP) involves cascades of events including release of neurotransmitter, activation of receptors and gene expression. 脑缺血预处理(cerebral ischemic preconditioning,CIP)诱导的脑缺血耐受(brain ischemic tolerance,BIT)的产生涉及神经介质、受体及基因的表达等一系列过程。
- Brain ischemic preconditioning 脑缺血预处理
- Methods Focal brain ischemia animal model was used. 方法应用局限性脑缺血动物模型。
- It has similar protective effects on myocardium to ischemic preconditioning. 它具有同缺血预处理相似的心肌保护效应。
- Purpose To investigate the influence of ischemic preconditioning(IP) on permeability of blood brain barrier(BBB)and expression of matrix metalloproteinase-9 (MMP?9)in subsequent ischemic hemisphere. 目的 研究缺血预处理 (IP)对再缺血脑组织血脑屏障通透性和MMP 9表达的影响。
- ATP-Sensitive Potassium Current Alterations during Ischemic Preconditioning in Guinea Pig Ventricular Myocytes. 心肌缺血预适应引起的ATP敏感性钾电流变化。
- Objective: To discuss the role of uncoupling protein 2(UCP2) in myocardium with ischemic preconditioning(IPC). 目的:探讨解偶联蛋白-2(UCP2)在心肌缺血预适应(IPC)心肌保护中的作用。
- Objective To investigate the effect of myocardial ischemic preconditioning( IP) on myocardium in hyperlipemia rabbits. 目的研究缺血预适应(p)高血脂动脉硬化兔心肌的影响。
- CDP-choline improved the recoveryof SEPs caused by brain ischemia obviously. CDP-choline可明显促进缺血脑功能的恢复。
- AIM To compare the protective effects of norepinephrine preconditioning(NEPC)and ischemic preconditioning(IPC)on myocardial ischemic/reperfused injury in rats in vivo. 目的观察比较外源性微量去甲肾上腺素预处理及经典缺血预处理对在体大鼠缺血/再灌注心脏损伤的保护效应。
- Aim To observe the effect of limb atraumatic ischemic preconditioning (RIP) on the myocardial ischemia-reperfusion (I/R) injury in rats. 摘要目的研究无创性肢体缺血预适应(RIP)对大鼠缺血/再灌后心脏损伤的影响。
- These data support the concept that the cardioprotective effects of DPR of ischemic preconditioning can be streng thened by DETA. DETA诱导的DPR,在心脏的获取-低温保存-移植后再灌注过程中,对心肌细胞有显著的保护作用。
- No DND was found in the hippocampus of the rats subjected to sham operation and CIP, in which 3 min cerebral ischemic preconditioning was performed. Sham组和3min CIP组海马未见DND。
- Objective To explore involvement of the Nogo-A/NgR pathway in neuroprotective effect of ischemic preconditioning (IPC) on focal ischemia in rats. 目的研究Nogo-A/NgR通路参与缺血预处理(IPC)对局灶性脑缺血的神经保护作用。
- Both Brain ischemic preprocessing and postprocessing can result in neuroprotective effect by inducing brain ischemic tolerance. 脑缺血预处理及后处理均可以诱导脑缺血耐受形成而产生神经保护作用。
- UCP2 is overexpressed following ischemic preconditioning, and it may be involved in the mechanism of the cardioprotection induced by IPC. IPC可减轻心肌超微结构损伤程度和减少细胞凋亡,IPC可诱导UCP2表达,提示UCP2可能参与了IPC的心肌保护作用。
- DWI is sensitive to early ischemic changes,which can find the changes of brain ischemia earlier than that of routine T2WI. DWI对早期缺血改变非常敏感,DWI较常规T2加权图像能更早地发现脑缺血的改变。
- Objective To investigate the protection of Shenyuan dan capsule on pharmaco ischemic preconditioning (PIP) in Wistar rats. 摘要目的探讨参元丹胶囊对心肌缺血大鼠的心肌保护作用。
- Currently ischemic preconditioning is one of the most efficacious ways to treat ischemia reperfusion via triggering endogenously protective system. 缺血预适应是目前治疗缺血/再灌注损伤最为有效方法之一,其机制是通过调动机体内源性保护体系而发挥作用。