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- ECCM and ET 1 have mitogenic effects on SMC. ECCM和 ET- 1可促进 SMC的增殖作用 .
- ET 1 conld play a role in the fibrogenic process of OSF. 由此提示，ET－1可能参与了OSF的纤维化过程
- Endotoxin could increase the synthesis and release of ET 1 and NO. 内毒素使ET?1 和NO 含量增加；
- It plays the role partly by reducing the level of NO and ET 1 in the body. 可能部分通过减少NO和ET?1生成而起作用
- ET 1 and CGRP may play an important role in the development of active SLE. 提示ET?1与CGRP失衡可能是SLE活动期发生、发展的重要因素。
- NO and PGI 2 may antagonize the biological action of ET 1 during endotoxemia. NO及PGI2可部分拮抗ET?1的生物学效应。
- There were negative correlations between the plasma levels of ET 1 and CGRP. 直线相关分析表明，血浆ET?1与CGPR呈显著负相关。
- ET 1 and the ETB agonist, sarafatoxin S6c increased BP to a greater extent in D 2+/+ than in D 2-/- mice. ET 1和ETB激动剂sarafatoxinS6c升高D2 +/ +鼠血压强于D2 - / -鼠。
- ET 1 mRNA was mainly presented in free side of Clara cell and NEB of rat bronchus. 慢性缺氧高二氧化碳大鼠支气管中ET 1mRNA主要定位于Clara细胞和神经上皮小体的游离面胞浆。
- ET 1,ETA and ETB mRNA expressions were all evidently increased after 60 minutes of ischemia. 结果发现：再灌流1小时，ET?1、ETA、ETBmRNA均明显升高； 再灌流24小时仍维持较高水平。
- Endogenous CO released from VSMCs has an inhibitive effect on VSMCs proliferation induced by endothelin 1 (ET 1). VSMCs产生的CO对ET?1刺激的平滑肌细胞的增殖有抑制作用。
- L NMA and Phos inhibited the increases of nitrate or ET 1 respectively, and reduced their baseline levels. L?NMA 和Phos 分别抑制血浆NO－3 或ET?1浓度异常升高及基础分泌，短暂改善血流动力学指标。
- Coccygeal blood pressure, blood viscosity, AT 1,ET 1, ROS in kidney and apoptosis in kidney were observed. 埋泵前后小鼠尾测高血压,检测血液黏度、血管紧张素1(AT-1)、内皮素1(ET-1),肾组织中活性氧化物(ROS)、肾组织细胞凋亡情况。
- Results: (1)Microinjection of 2 pmol ET 1 into rVLM could not induce ventricular arrhythmias(VA) in normal heart. 结果：在正常心脏，rVLM内微量注射ET?1（2pmol）不引起心脏节律的紊乱；
- In DCM groups, EDD is negatively correlated with the levels of both plasma vWF and ET 1 concentrations. DCM患者EDD与血浆vWF及ET 1浓度均呈显著负相关。
- Our firm is likely to reap a big profit this year. 今年我们公司很可能获得巨额利润。
- Expressions of that ET 1,ETA,and ETB mRNA expression are all evidently increased at 1 hour postburn. 烧伤后1小时，ET－1、ETA、ETB、mRNA均明显升高；
- The result showed that ET 1 decreased hepatic blood flow and lasted for longer time. 结果显示：ET?1能使肝血流量迅速降低，并且持续时间较长；
- The results showed that inhibition of production of ET 1 and PGI 2 was induced by dengue virus infection in HUVECs. 登革病毒感染可使HUVEC分泌ET?1 及PGI2 的能力受到明显抑制。
- Thrombin stimulates HMC to produce ET 1 and ET 1 mediates the HMC proliferation. 凝血酶刺激HMC产生ET?1，ET?1介导了HMC增生。
